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The big penis and mens' sexual health source, increasing penis size around the world.

Gaining volume with Kyrpa

Hey Kyrpa,

interesting thread.
Just did my first US session thanks to your findings.
The sensations turned out really different to what I initially expected.

I took L-Citrulline Malate ( 6 gram) right before the session and put my extender on low tension at about 1000 gram tension for about 20 Min while using the US device.
The first 5 Min were really disappointing.I bought a one time used device for 20 euro on ebay and I thought the device was defect.
But after 5 Mins I began to feel it. Afterwards I followed up with a short mid-high intensity pulse pump session in the 7 inHG range under IR light in a narrow fit cylinder.

Really interesting results in the steelcord and in volume because of more elasticity..
Never seen my veins working in such a manner too. Subtle but it is there. Free flow so to say.
Tunica is softened up and my blood chambers expanded well while pulse pumping.
Warm sensations in (!) the shafts hours later.

I am sold so far on US, but I don’t know yet what it really does.

Did the exact same session 8 days ago and did not experience these effects at all.
Question is if US will influence gains.
We will see.

Gonna do a experimental run with pumping in a IPR template and watch the effects on tunica and recovery rate.

Thanks for posting your findings. PE never gets boring ;)


" PE is a helluva drug. "

Originally Posted by pe_pe
Hey Kyrpa,

interesting thread.
Just did my first US session thanks to your findings.
The sensations turned out really different to what I initially expected.

I took L-Citrulline Malate ( 6 gram) right before the session and put my extender on low tension at about 1000 gram tension for about 20 Min while using the US device.
The first 5 Min were really disappointing.I bought a one time used device for 20 euro on ebay and I thought the device was defect.
But after 5 Mins I began to feel it. Afterwards I followed up with a short mid-high intensity pulse pump session in the 7 inHG range under IR light in a narrow fit cylinder.

Really interesting results in the steelcord and in volume because of more elasticity..
Never seen my veins working in such a manner too. Subtle but it is there. Free flow so to say.
Tunica is softened up and my blood chambers expanded well while pulse pumping.
Warm sensations in (!) the shafts hours later.

I am sold so far on US, but I don’t know yet what it really does.

Did the exact same session 8 days ago and did not experience these effects at all.
Question is if US will influence gains.
We will see.

Gonna do a experimental run with pumping in a IPR template and watch the effects on tunica and recovery rate.

Thanks for posting your findings. PE never gets boring ;)

Thank you pe_pe for your words. Great to hear someone finding inspiration in my notes.

I am convinced using US is not just a way to warm up for exercises, it is rather an active tool for enhancing penile tissues.

Keep on experimenting and posting your findings if you will.


START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)

Little catch up on things

If there is some one out there wanting to hear about Period 3 ,unfortunately I have to disappoint you.
Never went for it as it should be executed. I had two cycles done when I tuned it down before really got into US and intensive stretching. Been just enjoying myself and having sex and being lazy between my daily work.
Though having backed off from program, I have still be doing some easily performed maintenance with some results as well. Before turning into maintenance mode I could reach 23.4 cm BPFSL post-exercise.
I have had morning pumping for a 30mins ,combined with vac stretching for 60-120min with a fitness rubber bands.
For the next day I have had pumping combined with 3 x10 min sets with my newly purchased cable clamps.
Working out Two to three days with similar rest periods. No heat whatsoever no manual stretching at all.

I can confirm my previous thoughts on narrowing the gap between BPFSL and BPEL with this kind of procedures. BPEL is going upwards and no enhancement on BPFSL.

Now measuring 21.4 cm BPEL/ 22.8 BPFSL no mentionable difference on girth.

Not done with it , I have an urge to go forward and you will be first to know when I am on it again.

Keep on hammering


START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)

Kyrpa, I’d be interested in your thoughts or experience with pulsed vs constant tension while under US heat. At least a couple of studies suggest that pulsing the tension results in greater residual length. For example, once target heat has been achieved, slowly increasing up to a max weight of 5lbs then back down to 1lbs. Do this in 5 min cycles for a total of 60 minutes. This is supposed to be more effective than a constant heated stretch at 5lbs for 60 min.

Originally Posted by Tutt
Kyrpa, I’d be interested in your thoughts or experience with pulsed vs constant tension while under US heat. At least a couple of studies suggest that pulsing the tension results in greater residual length. For example, once target heat has been achieved, slowly increasing up to a max weight of 5lbs then back down to 1lbs. Do this in 5 min cycles for a total of 60 minutes. This is supposed to be more effective than a constant heated stretch at 5lbs for 60 min.

Hello Tutt,

No experience on this. Yes there are studies showing results you suggested. The pulse frequency should be the key. Every study I have looked used frequencies which would need some kind of pulsating apparatus. You need to build one first.

Maybe I look deeper on it the day my gains start to show signs of diminishing permanently.

Which studies are you referring?


START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)

Originally Posted by Kyrpa
Been just enjoying myself and having sex….

Well done and great log Kyrpa!


Once upon a time (2015): 6.40” x 4.50”

Today: 7.25” x 5.00”, Thunder Cocks Unite!

I think we can...Little Engine’s Climb

Originally Posted by LittleEngine
Well done and great log Kyrpa!

Thank you Sir.


START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)

Originally Posted by Kyrpa
Hello Tutt,

No experience on this. Yes there are studies showing results you suggested. The pulse frequency should be the key. Every study I have looked used frequencies which would need some kind of pulsating apparatus. You need to build one first.

Maybe I look deeper on it the day my gains start to show signs of diminishing permanently.

Which studies are you referring?

Unfortunately, I don’t have the full study citations handy. I know there were 1 or 2 rat tail studies testing cyclic straining and residual tendon elongation without heat. One of them might have been;

Elongation of rat tail tendon: effect of load and temperature.
Warren CG, et al. Arch Phys Med Rehabil. 1971.

I don’t have the full text study available right now. I seem to recall an assertion that a cyclical repeat of strain and relaxation produced the greatest residual elongation without rupture. This is also indicated in the study by Rigby et al 1959. Looking at figure 6, there is a dramatic decay in the load necessary to produce significantly greater extensions, ultimately reaching an impressive 35%.

Gustav et al 1951 show that the first strain curve (if less than 1%/min) can be taken to as much as 20%, but requires a constant or increased load depending on time, albeit the load increase required to produce an incremental increase in strain decreases as both time and extention increase. This, combined with the study above and others, suggest that it is the cyclical nature of the strain that allows the extreme plasticity without reaching the point of failure.

Keeping in mind that these are in vitro studies and that in vivo there would undoubtedly be severe pain at slightly beyond the elastic limit. But the evidence also suggests that cyclic strain changes the actual arrangement of collagen tissue, resulting in less damage and consequently less discomfort and arguably reduced healing times.

There are also additional considerations in vivo as it pertains to PE. The ‘I’ phase of IPR cannot be overemphasized at the expense of ‘P’ and ‘R’. Specifically, several studies suggest that internally produced Hyaluronic Acid, critical protein kinases, and Nitric Oxide each play a key role in cell proliferation and avoidance of “wound” type collagen formation (scarring and adhesions) in favor of repair via normal tissue growth. Additionally, they determine the type, shape, and orientation of collagen production. I’ve come across a couple specific references to these factors reaching peak production and concentration at 30min of strain and was dependent on the magnitude of the strain (I.e. Higher strain equals higher production). Obviously there were limits, as at a certain point the strain exceeded the limits of cell viability. One of the studies went on to suggest that strains in excess of 60 minutes resulted in a significant increase in cell apoptosis over time. Also, the Ribgy study asserts that strains in excess of 60 minutes are “deleterious” to the tissue. Although, in the case of PE this deleterious result might be desirable within moderation.

Some here focus very heavily on how to get the maximum residual extension of the collagen structures, while it is probably more advisable long term to find the optimal balance between residual extension and healthy cell proliferation. The latter would necessitate a commitment to retaining the viability of existing tissue and simultaneously triggering peak production of “scarless” healing agents.

Consequently, the most optimal regime would seem to include a very controlled strain cycle with the following characteristics. I will use the term baseline length to denote the transition point at the toe of the load strain curve. Roughly, this would correspond to the completely flaccid penis in a fully relaxed state and a light load (about 1lbs) over a period of several minutes. The term ‘elastic limit’ will refer to what we normally probably consider BPFSL under a modest load (about 3-5lbs depending on the conditioning).

STEP 1.. No heat if not at least partially loaded and within elastic zone, as anything above 37C results in collagen tissue contraction when not under load.

STEP 2.. Within elastic zone heat collagen to about 41-42C. Below 40C is repeatedly shown to leave the tissue unaltered, while above 43C risks tissue necrosis over prolonged periods. Keep in mind for next steps that studies experienced repeated tendon rupture at around 4% strain when temps were increased above 40C.

STEP 3.. Set strain rate of about 0.5-1.0%/minute, to avoid tissue damage/pain.

STEP 4.. Start 1st strain to about 2.5% beyond baseline length (should be no discomfort as we will still be within elastic range), followed by a 5 minute ambient cool down at static strain (notice I said static strain not constant load) and another 5 minutes relaxation under zero load. This is theoretically within the full elastic recovery safe zone but will get the tissue ready for a stretch.

STEP 5.. Under light load reheat to 41-42C then strain at 0.5%/min to 3% beyond baseline (should feel a stretch but not pain). 5 min ambient cool down under static strain and 5 min additional rest at no load. This should be just barely past the recovery zone and fundamentally weaken the collagen secondary bonds triggering a healing response. My assumption is that this step corresponds to about 1mm beyond the elastic limit referred to above.

STEP 6.. Under light load reheat to 41-42C and strain at 0.5%/min to about 4% beyond baseline (stop if any pain). Hold at this static strain under constant heat for 30min. 10-15min ambient cool down at this static strain, assuming no pain during cooling. This should be about 2.5mm past the elastic limit.

STEP 7.. Reduce load to near zero while maintaining new baseline length for 3-6 hours. The previous steps should’ve resulted in a stress-relaxation already taking the load back near zero, so this sort of ADS is mostly preventing the muscles from getting involved or contribution from other tissues that didn’t reach the plastic flow region.

What to do after that is very debatable. The half life of the HA and NO in collagen tissue is 1-3 weeks which follows more of a macro IPR timeline. Arguably, this procedure could be followed once every 3-4 days with some minor daily manuals or periodic light ADS (in both cases not exceeding the new elastic limit) to maintain the tissue memory of the extended state during initial cell proliferation. My assumption would be an ADS routine of 60min light stretch 4 times a day. After about 14 days switching into a macro P phase for 2-3 weeks with continued periodic ADS use but no more plastic strain, and then a Macro R phase for about 2-3 months with nothing like more than Kegels and light jelqs to massage the tissue.

I should probably clarify that the regime from the previous post would require a level of precision that would necessitate a purpose built device unlike anything that exists currently. Elastic bands, springs, and weights would be replaced with micrometer actuators on a linear translation stage.

I could easily envision a precision mod to a PMP with vacuum attachment that would allow for the proper rate of extension while leaving open access for US heat.

Originally Posted by Tutt
Unfortunately, I don’t have the full study citations handy. I know there were 1 or 2 rat tail studies testing cyclic straining and residual tendon elongation without heat. One of them might have been;

Elongation of rat tail tendon: effect of load and temperature.
Warren CG, et al. Arch Phys Med Rehabil. 1971.

I don’t have the full text study available right now. I seem to recall an assertion that a cyclical repeat of strain and relaxation produced the greatest residual elongation without rupture. This is also indicated in the study by Rigby et al 1959. Looking at figure 6, there is a dramatic decay in the load necessary to produce significantly greater extensions, ultimately reaching an impressive 35%.

Gustav et al 1951 show that the first strain curve (if less than 1%/min) can be taken to as much as 20%, but requires a constant or increased load depending on time, albeit the load increase required to produce an incremental increase in strain decreases as both time and extention increase. This, combined with the study above and others, suggest that it is the cyclical nature of the strain that allows the extreme plasticity without reaching the point of failure.

Keeping in mind that these are in vitro studies and that in vivo there would undoubtedly be severe pain at slightly beyond the elastic limit. But the evidence also suggests that cyclic strain changes the actual arrangement of collagen tissue, resulting in less damage and consequently less discomfort and arguably reduced healing times.

There are also additional considerations in vivo as it pertains to PE. The ‘I’ phase of IPR cannot be overemphasized at the expense of ‘P’ and ‘R’. Specifically, several studies suggest that internally produced Hyaluronic Acid, critical protein kinases, and Nitric Oxide each play a key role in cell proliferation and avoidance of “wound” type collagen formation (scarring and adhesions) in favor of repair via normal tissue growth. Additionally, they determine the type, shape, and orientation of collagen production. I’ve come across a couple specific references to these factors reaching peak production and concentration at 30min of strain and was dependent on the magnitude of the strain (I.e. Higher strain equals higher production). Obviously there were limits, as at a certain point the strain exceeded the limits of cell viability. One of the studies went on to suggest that strains in excess of 60 minutes resulted in a significant increase in cell apoptosis over time. Also, the Ribgy study asserts that strains in excess of 60 minutes are “deleterious” to the tissue. Although, in the case of PE this deleterious result might be desirable within moderation.

Some here focus very heavily on how to get the maximum residual extension of the collagen structures, while it is probably more advisable long term to find the optimal balance between residual extension and healthy cell proliferation. The latter would necessitate a commitment to retaining the viability of existing tissue and simultaneously triggering peak production of “scarless” healing agents.

Consequently, the most optimal regime would seem to include a very controlled strain cycle with the following characteristics. I will use the term baseline length to denote the transition point at the toe of the load strain curve. Roughly, this would correspond to the completely flaccid penis in a fully relaxed state and a light load (about 1lbs) over a period of several minutes. The term ‘elastic limit’ will refer to what we normally probably consider BPFSL under a modest load (about 3-5lbs depending on the conditioning).

STEP 1.. No heat if not at least partially loaded and within elastic zone, as anything above 37C results in collagen tissue contraction when not under load.

STEP 2.. Within elastic zone heat collagen to about 41-42C. Below 40C is repeatedly shown to leave the tissue unaltered, while above 43C risks tissue necrosis over prolonged periods. Keep in mind for next steps that studies experienced repeated tendon rupture at around 4% strain when temps were increased above 40C.

STEP 3.. Set strain rate of about 0.5-1.0%/minute, to avoid tissue damage/pain.

STEP 4.. Start 1st strain to about 2.5% beyond baseline length (should be no discomfort as we will still be within elastic range), followed by a 5 minute ambient cool down at static strain (notice I said static strain not constant load) and another 5 minutes relaxation under zero load. This is theoretically within the full elastic recovery safe zone but will get the tissue ready for a stretch.

STEP 5.. Under light load reheat to 41-42C then strain at 0.5%/min to 3% beyond baseline (should feel a stretch but not pain). 5 min ambient cool down under static strain and 5 min additional rest at no load. This should be just barely past the recovery zone and fundamentally weaken the collagen secondary bonds triggering a healing response. My assumption is that this step corresponds to about 1mm beyond the elastic limit referred to above.

STEP 6.. Under light load reheat to 41-42C and strain at 0.5%/min to about 4% beyond baseline (stop if any pain). Hold at this static strain under constant heat for 30min. 10-15min ambient cool down at this static strain, assuming no pain during cooling. This should be about 2.5mm past the elastic limit.

STEP 7.. Reduce load to near zero while maintaining new baseline length for 3-6 hours. The previous steps should’ve resulted in a stress-relaxation already taking the load back near zero, so this sort of ADS is mostly preventing the muscles from getting involved or contribution from other tissues that didn’t reach the plastic flow region.

What to do after that is very debatable. The half life of the HA and NO in collagen tissue is 1-3 weeks which follows more of a macro IPR timeline. Arguably, this procedure could be followed once every 3-4 days with some minor daily manuals or periodic light ADS (in both cases not exceeding the new elastic limit) to maintain the tissue memory of the extended state during initial cell proliferation. My assumption would be an ADS routine of 60min light stretch 4 times a day. After about 14 days switching into a macro P phase for 2-3 weeks with continued periodic ADS use but no more plastic strain, and then a Macro R phase for about 2-3 months with nothing like more than Kegels and light jelqs to massage the tissue.

There is a lot to comment above but I like have a take on few things first, and maybe get in to it deeper later on.
We have to decide If we are talking about pulsed or cyclic strain? There would be huge difference in practical applications and outcomes as well.
You asked if I have opinion or experiences with pulsed stretch and I do not have. But now the focus seems to be turned to cyclic stretch which I consider for being quite different concept in practice.

Having used 30 second lasting manual stretches as my main workout targeting the plastic region, I could argue that I have practical experiences in cyclic stretching.
As you haven´t pointed any cyclic frequency in your example I assume manual stretching in a such manner would fit in to your procedure.

You would need to modulate the percentages you are using to fit this penile environment.
4% strain would be excessive. There a several studies pointing out that micro failures start to form in collagen tissues at 2,5 -3% already.

I would like to point out that we have already focused in practical applications measuring actual percentages of strain by manko007 and me.
I have reached maximum of 3,6% strain measured post exercise with US heated manual stretches.
I have attached a datasheet where you can track percentages and practices used.
Loads are not presented but for vac stretching 1 - 4 kg, manual stretching are tested to be in the range of 8- 10kg. Average strain is 2,5%.
Manko007 have attached few datasheets on his log, and you can find the percentages to be similar.
Similar results as well even with a different approach with the practices

I agree on temperatures confirming 40 - 43C should be safe, but disagree with the need of ADS use.

I would like to see you using your protocol in practice.
These rat tail studies have been discussed here for decades and theories have been thrown all over.

I have no interests in theories in vivo that much, I have past that and taken the bull by his horns already.
What I would suggest is to get your hands dirty and turn it on applications.

Table 4.webp
(137.1 KB, 350 views)

START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)


Last edited by Kyrpa : 07-04-2019 at .

Apologies about using the term pulsed before. I was really referring to cyclic strain.

We might not be too far apart in our estimation of strain limits. I might just be specifying a slightly different starting point. Are you saying that you achieve a heated max strain about 2-3% beyond pre-PE BPFSL? If so, then we are saying the same thing.

Anyway, sorry for hijacking your thread. I appreciate your insights as my interest in PE is admittedly more based on curiosity and research than personal development.

Originally Posted by Tutt
Apologies about using the term pulsed before. I was really referring to cyclic strain.

We might not be too far apart in our estimation of strain limits. I might just be specifying a slightly different starting point. Are you saying that you achieve a heated max strain about 2-3% beyond pre-PE BPFSL? If so, then we are saying the same thing.

Anyway, sorry for hijacking your thread. I appreciate your insights as my interest in PE is admittedly more based on curiosity and research than personal development.

No worries just wanted to ensure we are talking the same language.

Percentages presented are from pre-exercise BPFSL measured as you described. That would be the initial elastic limit.

One thing we have to take into consideration is the fact that the actual elastic limit is way further that.
Heat and the mechanical stress mold the collagen tissue to lenghten even more before reaching the plastic region.

I have tried to cover this on my approach with maximizing the stress relaxation effect before going for the plastic enhancement with manual stretches.
I would claim my elastic limit is pretty much at 2,2% strain from the pre -exercise BPFSL measure. After this strain we are approaching the target region.

For the practical solution with the precision you were describing, there are some obstacles to win.
Measuring while on vac attachment is not accurate as the slippage with the cup would hinder the measurement giving at least +- 2mm differences easily.
Also reaching above 4% strains would be hard to reach because the difficulties with the grip and high loads needed for this strain.

If you have real life medical background on these matters, I would like you to be tuned in and observe what is happening here and give your insights as well if you will.


START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)

“I would like to see you using your protocol in practice.

X2


Big cock, tight abs, fit body, strong mind.

Originally Posted by Buckfever
“I would like to see you using your protocol in practice.

X2

I’m working on the device that would allow for the required precision and control. In a perfect world, I would combine my programmable tensiometer with a device resembling a modified penimaster pro. I would also use hands-free radio frequency for heat. Obviously, a more crude prototype makes more sense at this time. I do have a tensiometer, a PMP, a strong US machine, and a precision mill for the mods. Just a bit more design and I’ll see what I can figure out.

Kyrpa, I was curious what is the typical amount of time you use US? I have started experimenting with a 3MHz machine, and am very encourged by the deep tissue heating ability. I am unsure if there is any danger in using it for a 40 minute vacuum stretching routine, as long as temperature is kept below 43 Celsius? I am going to start a 5 week routine using your 3 on 2 off schedule. I will be rotating exercises daily using the US on day 1, and day 3 of each cycle while vacuum stretching.

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