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Correct me if I’m wrong but anti-aromatase drugs only stop the aromatase enzyme that coverts test to estrogen. They will not make your body produce more test just stop it being converted to estrogen. Also there won’t be much aromatase enzyme in you unless your test levels get really high I.e you are injecting the stuff! So don’t take some expecting it to make a difference unless you are on steroids.
Estrogen level is a main feedback mechanism of the testosterone level. Your body regulates testosterone production based in large part on how much estrogen it perceives. If you either fool the sensor using a SERM or actually lower estrogen with an AI, more GnRH is secreted, which results in more LH, causing your nuts to make more testosterone. When testosterone is higher, more of it aromatizes into estrogen.
The body is pretty good at adjusting things to keep levels where it thinks they should be. For example, SHBG increases when E goes up. Makes sense. Higher E indicates higher T, so less testosterone needs to be free.
If you knock E way down with a non-steroidal AI, your body will try to adjust, IIRC by producing more aromatase. When you then stop taking the AI, estrogen rebounds even higher than before. Another negative is with very little estrogen present, estrogen receptors will upregulate. Not good.
SERMs such as clomiphene (Clomid) and tamoxifen (Nolvadex) are estrogen antagonists in some tissues and estrogen agonists in others. Here is a recent article about tamoxifen: http://www.meso morphosis.com/a … id-nolvadex.htm
Clomiphene and tamoxifen both have some side effects, especially in high doses. I think it’s not wise to risk screwing up your vision, increase the risk of blood clots, etc. for no reason other than to experiment with a prescription drug. Don’t take them unless you need them. Newer SERMs are supposed to be less toxic and more tissue specific.