Anatomy of a Penis- A few questions.

Well I’ve read through a lot of threads dealing with the anatomy of a penis, and i’m still confused. If anyone could help answering some of these questions that would be appreciated. I know this is a lot of information presented, so bear with me.

1. I’m a little confused about the corpus spongiosum, the role of the BC, and the corpus cavernosum. When you flex your BC muscle, it pushes blood into the corpus spongiosum, which, then pushes blood into the glans and into the larger chambers, the corpus cavernosum. Concurrently, blood (via stimuli) is pushed through your corpus cavernosum, and also into the glans. The large amount of blood in your penis, is absorbed in your tissues, expanding gradually. This suffocates the penile arteries and hinders the escape of blood, resulting in an erection. Further, when an erection is achieved, and the penile chambers are sufficiently filled with blood, and a “kegel” is performed (or a flex of the BC muscle), it “forces” more blood into the chambers/tissues, and “pushes” the less-oxygenated, old blood, out (while having the new blood, trapped inside).

What is the role of the penile veins on top of your penis, along with the dorsal vein? Is this where the blood initially comes to, and THEN is sent to the corpus cavernosum, then out the penile arteries? Or is it that the blood goes into the penile/dorsal veins, WHILE the corpus cavernosum is being filled; then the blood in the penile/dorsal veins goes into the corpus cavernosum to fill it up further?

2. Turtling- I took an adderall today and notice my unit turtle to about an 1 inch and half (EDIT: probably about 2 inches…nonetheless it is still extreme turtling). While I know amphetamines hurt my EQ, and I don’t completely understand the relationship of amphetamines to my penis, the point i’m looking at is with turtling in general. When you turtle, is the penis retracted into yourself as a whole? Or does the corpus cavernosum/spongiousm deplete itself, all the while it is being retracted? Does it shrink in size, thus affecting the tunica, making it much, much, smaller (no blood in penile tissue to expand it). How counter productive is this to “general PE or penile enlargement (literally)” especially AFTER a penis workout?

3. I’ve been reading more and more about the role of cold/heat, in the ligaments. I’ve read about elastic and plastic deformation. This is where it gets interesting.

Scenario A. Suppose I was to heat my penis to the optimal heat, for stretching. I would finish heating, then I would stretch. I would stretch for a while, and let my penis cool down gradually (while stretching). When my penis is room temperature, I am finished. Would this be the optimal scenario to stretch?

Scenario B. I perform my heating, then I stretch. But I keep heating, and keep stretching. When I am finished stretching (I was heating all the while), I am finished all together. No cool down.

Scenario C. I heat my penis, then stretch. I am finished stretching, and, in a elongated state, cool my penis using a cold object, e.g., cold towel, ice, cold water.

In scenario A, which seems to be what most people here at Thunders do, is this the best scenario to achieve plastic deformation? Is scenario B the scenario most likely to achieve elastic deformation? Although the result of length gain would obviously be dependent on the force exerted, the elastic deformation would then become plastic, but in a much greater time? Elastic, then plastic? In scenario C, the heat would be conducive to elastic/plastic deformation. Then, while temporary elongated, if one were to apply a cold object, would this help “solidify” gains? What is the science behind this? Obviously we know that heat helps the ligs become elastic, but what about cold?

4. When an erection is obtained, the penile arteries become suffocated. If one were to add a cold wrap around the penis, would the cold constrict the penile arteries further? Thus aiding in blood retention in the penis? If so, one could kegel more and more blood into the penis, expanding the amount of blood that the penis could usually hold, to a greater amount. But if the cold doesn’t have this blood retaining effect, would the cold harm the erection quality?

In your penis, and everywhere else in your body, arteries bring oxygenated blood into your tissues (gradually dispersed through small vessels like arterioles and finally capillaries), while veins take the more de-oxygenated blood back to the heart. Arteries are thick walled and high pressure. Veins are thinner walled because they are under much lower pressure. The arteries are usually deep while veins are superficial. In the case of the penis the Dorsal arteries run along side the larger dorsal vein. The veins on the surface of your penis, and the dorsal vein, are for the transport of blood out of the penis.

Pressure in the penis assists in the constriction of veins to prevent outflow. I suppose that is what you mean by artery suffocation, but it it not arteries that control outflow, and there is no suffocation.

For your 3 scenarios, you need to understand that plastic and elastic deformation are mechanical terms for materials and maybe don’t apply exactly to PE. If you stretch a rubber band or balloon, and it goes back to its original shape, it was deformed elastically. If it does not return to it’s original shape, then it has crossed into plastic deformation. For a penis, if you achieve plastic deformation, it means that you have permanently changed your penis size and shape immediately as a result of the intensity of the stretch. I doubt that happens in real life. The tunica is not very elastic, and after reaching it’s elastic limit, it would probably tear rather than plastically deform. Picture plastic deformation as the balloon that is baggier after its initial inflation. With a penis, the growth probably occurs from healing of damage caused by stretching. The heating and cooling options you describe are all used with success, and the best one is a matter of opinion and probably depends on the person.

Thanks for the response, and the edit, Mravg. Well maybe I didn’t use plastic/elastic deformation, in the right context, in accordance to PE that is. But you doubt plastic deformation? Isn’t that what we are striving to do, for stretching? Prolonged elastic deformation must surely result in plastic deformation, for the penis. I mean, in plastic deformation, before it becomes deformed permanently, it goes through elastic deformation, then plastic deformation (obviously with more added force). You stretch elastically, and gradually it gets longer, longer, until is much longer than the first moment you started stretching (over time). I mean, if we aren’t striving for plastic deformation, what exactly are we looking to do by tugging on our dicks? For something to be defined as “plastic deformation” must it be deformed, “immediately?” (as you stated).

Isn’t plastic deformation what we want? We gradually stretch the penis, until eventually, it is longer than before, permanently. Surely this new, lengtheir penis is “plastically deformed?” You say that after reaching the tunica’s elastic limit, it would probably tear than plastically deform. Who is to saying we are stretching to the tunica’s elastic limit, or ultimate load?

From thread, Connective tissue- FIRST “THREAD OF THE YEAR”

In order to deform, and then reform a ligament into a more desirable length and form, the applied “constant” load must reach over 40% of that particular ligament’s “ultimate load”. A ligament’s ultimate load is defined as “the final load reached by a structure before failure”. (5,6) So, the force needed to create new ligamentous form is well below the safety limit for loading that ligament, but must be high enough to stretch the ligament such that it only undergoes a partial rebound towards its original length and shape.

Yea, I agree, once the tunica’s elastic limit is reached, then massive tears and things you don’t want to happen, happen. But by stretching to far under the tunica’s elastic limit, we are promoting growth.

…Continued from above.

Yea, I agree, once the tunica’s elastic limit is reached, then massive tears and things you don’t want to happen, happen. But by stretching to far under the tunica’s elastic limit, we are promoting growth. Growth? Tissue damage caused by stretching, causes mitosis of the fibroblasts, which make up part of the collagen found in the tunica, among other tissue repair. Isn’t this a “new” penis? All this caused by stretching? If you have any links to the matter that would help me better understand this, that would be very helpful! Peace!

And I was just about to go to bed… (:) )

My responses are in blue.

1. I’m a little confused about the corpus spongiosum, the role of the BC, and the corpus cavernosum. When you flex your BC muscle, it pushes blood into the corpus spongiosum, which, then pushes blood into the glans [You’re right up to here.] and into the larger chambers, the corpus cavernosum. [No. There is no connection between the glans and the corpora cavernosa.] Concurrently, blood (via stimuli) is pushed through your corpus cavernosum, [Not pushed, rather allowed in via your normal blood pressure by the relaxation of arteries.] and also into the glans. [No, there is no connection between the CC and the glans.] The large amount of blood in your penis, is absorbed in your tissues, expanding gradually. [The CC are expandable chambers. They don’t absorb blood, they fill like a balloon.] This suffocates the penile arteries and hinders the escape of blood, resulting in an erection. [Small veins drain the CC when flaccid. A small amount of blood is usually passing through the corpora cavernosa and it drains via these small veins. At erection the CC compress these veins between the tunica (the veins pass through it) and Buck’s fascia. When these small veins are compressed, blood builds up in the CC causing the erection.] Further, when an erection is achieved, and the penile chambers are sufficiently filled with blood, and a “kegel” is performed (or a flex of the BC muscle), it “forces” more blood into the chambers/tissues, and “pushes” the less-oxygenated, old blood, out (while having the new blood, trapped inside). [A kegel contracts the bulbocavernosis (BC) muscle and forces blood into the corpora cavernosa and glans. Other muscles, the ischiocaverosus, compress the ends of the twin corpora cavernosa and increase the pressure within them. That’s all the kegel does, it temporarily increases pressure making the erection harder. There is always some blood leaving and new blood entering the penis via the blood pressure, even during an erection. You do not have to force the fresh blood in and deoxygenated blood out, it will happen automatically.]

What is the role of the penile veins on top of your penis, along with the dorsal vein? [As mravg said, veins drain blood from the body and send it back to the heart. The veins you can see are where the tiny corpora cavernosa veins drain into.] Is this where the blood initially comes to, and THEN is sent to the corpus cavernosum, then out the penile arteries? Or is it that the blood goes into the penile/dorsal veins, WHILE the corpus cavernosum is being filled; then the blood in the penile/dorsal veins goes into the corpus cavernosum to fill it up further? [No, you’re backward. The blood comes into the penis via arteries deep in the pelvis at the inner ends of the CC and CS. Blood leaves the CC and CS/glans via small veins which connect and drain into larger ones. The dorsal vein is one that you can see. There are others.]

[There are many web sites that explain all this in great detail. Use a search engine like Google and look for ‘physiology of erection’ or ‘how an erection occurs’.]

2. Turtling- I took an adderall today and notice my unit turtle to about an 1 inch and half (EDIT: probably about 2 inches…nonetheless it is still extreme turtling). While I know amphetamines hurt my EQ, and I don’t completely understand the relationship of amphetamines to my penis, the point i’m looking at is with turtling in general. When you turtle, is the penis retracted into yourself as a whole? [No.] Or does the corpus cavernosum/spongiousm deplete itself, all the while it is being retracted? [Most of the blood in it is expressed by the contraction of tiny muscles within the CC called the trabecular network. These muscles respond to different chemicals to contract or relax. If they contract the CC shrink to the smallest size possible when the blood is all expressed. Since the trabecular network itself does take some space within the CC, there is a limit to how small the compacted CC can become.] Does it shrink in size, thus affecting the tunica, making it much, much, smaller (no blood in penile tissue to expand it). [The tunica is the outer covering of the corpora cavernosa. It will shrink down along with the other parts of the CC when the trabecular muscles contract. It’s ability to expand during an erection or to be stretched during PE exercises is not affected by the CC compression called turtling.] How counter productive is this to “general PE or penile enlargement (literally)” especially AFTER a penis workout? [I’d say not at all. Others think that it makes a difference. If you’ve stretched the tunica during PE exercises and cause the small “micro-tears” that promote new cell growth, then having those tissues compressed afterward is not going to “reverse” the micro damage done. I think you’re worrying too much about this (perhaps as a result of your amphetamine ingestion).]

3. I’ve been reading more and more about the role of cold/heat, in the ligaments. I’ve read about elastic and plastic deformation. This is where it gets interesting.

Scenario A. Suppose I was to heat my penis to the optimal heat, for stretching. I would finish heating, then I would stretch. I would stretch for a while, and let my penis cool down gradually (while stretching). When my penis is room temperature, I am finished. Would this be the optimal scenario to stretch?

Scenario B. I perform my heating, then I stretch. But I keep heating, and keep stretching. When I am finished stretching (I was heating all the while), I am finished all together. No cool down.

Scenario C. I heat my penis, then stretch. I am finished stretching, and, in a elongated state, cool my penis using a cold object, e.g., cold towel, ice, cold water.

In scenario A, which seems to be what most people here at Thunders do, is this the best scenario to achieve plastic deformation? Is scenario B the scenario most likely to achieve elastic deformation? Although the result of length gain would obviously be dependent on the force exerted, the elastic deformation would then become plastic, but in a much greater time? Elastic, then plastic? In scenario C, the heat would be conducive to elastic/plastic deformation. Then, while temporary elongated, if one were to apply a cold object, would this help “solidify” gains? What is the science behind this? Obviously we know that heat helps the ligs become elastic, but what about cold?

[My opinion (others may be different) is that heat is important to make the tissues pliable. Deep heating with moisture (a hot, wet washcloth) is the way to do this because you cannot get the proper effect by simple skin surface heating. After your exercises additional heat may help them remain in the stretched position. Eventually you have to get back to normal body temperature (not room temperature unless you’re dead), but some application of heat (the “warm down”) helps. Cold, again in my opinion, does nothing positive.]

4. When an erection is obtained, the penile arteries become suffocated. If one were to add a cold wrap around the penis, would the cold constrict the penile arteries further? Thus aiding in blood retention in the penis? [No. During an erection penile arteries relax allowing the blood pressure to force blood into the normally nearly empty erectile chambers (both CC and CS). Veins drain this blood away. Outside temperature shouldn’t make any difference except applying cold to your penis might make your body decide it isn’t the right time for an erection.] If so, one could kegel more and more blood into the penis, expanding the amount of blood that the penis could usually hold, to a greater amount. But if the cold doesn’t have this blood retaining effect, would the cold harm the erection quality? [The kegel will temporarily expand the erectile chambers making your erection harder, but it’s only temporary. Temperature would not make any difference.]

Lol duhhhh. Thanks for the response, Westla. I will right my response tomorrow, I am going to bed myself. In all my reading, its funny to think I screwed up arteries/veins. Makes me look like an idiot! I do understand how an erection takes place though, for the most part, even though I don’t know some of the anatomical terms.

Is it my computer or is Westla’s response not showing up in blue?

Phenomenal post westla.

Thank you.

Can you see me now?

Red
Green
Blue
Yellow

Must be your monitor.

Westla, isn’t it known that cold causes vasoconstriction? So applying cold to the already erect penis, wouldn’t further constrict the veins? Possibly a cold clamp?

Temperature does make a difference, doesn’t it? e.g., Heat allows for great engorgement and causes vasodialation, isn’t that the point of warming up before a PE workout? Cold causes vasoconstriction. Theoretically, why wouldn’t an applied “cold clamp” or such, cause more trapping of the blood within the chambers?

What a great resource we have here in Westla!

Invictus, you ask some good questions.

Deformation of materials can not adequately be described in simple terms of elastic, plastic or tearing. The change of stretched length over time is a viscoelastic property. Viscoelastic nature of a material can be observed in behaviors such as CREEP (the property of a material to slowly grow longer when a constant stress is applied), or STRESS RELAXATION (the characteristic of a material stretched to a constant length to reduce it’s tension over time).

The penis does have some viscoelastic properties as you describe.
You found a great thread by Hobby. I was thinking of plastic vs. elastic in terms of typical engineering materials but I know that the terms are in fact commonly used in medicine when referring to tendon and ligament stretching.
Whatever we are doing when stretching our penises is happening on a micro scale, not a macro scale. Stretching can cause rearrangement of collagen which you could call plastic deformation even if it is not a measurable change in shape. Stretching can also stimulate growth as you described:

I view this growth stimulation as separate from plastic or elastic stretching. The act of stretching causes a response in the body to add tissue. There are lots of discussions around here about the different mechanisms for growth (actual stretching from collagen rearrangement, vs. growing new tissue). I suspect both things are happening.

The question of ice is interesting. I have read guys say positive things about it. Like you mentioned, can it cause vasoconstriction, therefore causing a more plump flaccid for a longer period of time after PE? Another theory I read in someone’s thread is that heat puts the collagen into a more fluid gel like state, so that after you stretch, ice can be used to freeze the collagen in its new stretched (rearranged) configuration. Interesting theories. I haven’t played around with ice much, so I don’t know.

What are you trying to do, get a larger, harder erection, or do PE exercises? If you’re exercising then there is no need for a full, hard erection. If you want a larger, harder erection for sex I suggest that having a cold member would be somewhat uncomfortable for your partner.

The veins in question (the tiny ones draining the CC through the tunica) are located throughout the length of the corpora cavernosa. For your theory to work you’d have to make the whole shaft cold. A clamp works mechanically, not by temperature. Having a cold clamp would not make if work more efficiently since mechanical compression of veins wins over cold induced vasoconstriction.