Wow, a lot of talk about scientific aspects of PE. But no “real” evidence (that is, peer-reviewed studies). I’ve searched a little and come up with a few that has relevance to PE. Here goes:
Penile elongation and thickening—a myth? Is there a cosmetic or medical indication?
Austoni E, Guarneri A, Gatti G. - Andrologia. 1999;31 Suppl 1:45-51
This study looked at a relatively new way of increasing penile size. The authors conclude that for length, the dual attack of suspensory ligament surgery and stretcher device is suggested. For girth they discuss a new method which is about making an incision in the tunica albuginea and enlarging that structure with grafts. That allows for more space for the corpus cavernosa to expand into as they should after the stretch stress of the stretcher device. After a 9-month follow-up, the increase in girth was 1.1-2.1 cm. Unfortunately, the regimens of stretching were not revelead, neither when they were begun (considering the post-operative recovery process) but as with all penile enlargement surgery, the stretching is vital to success (see below).
Penile enlargement surgery.
Alter GJ. - Tech Urol. 1998 Jun;4(2):70-6
To cite the revealing text of the abstract “Penile lengthening is performed by releasing the suspensory ligament of the penis followed by use of penile weights”. Why the weights? Because without that stressor there has been no statistically significant gain in length after surgery, some even experiencing a slight reduction in size. Proof enough that just the stretching should give benefits.
Microphallus: eventual phallic size is dependent on the timing of androgen administration.
Husmann DA, Cain MP. - J Urol. 1994 Aug;152(2 Pt 2):734-9
This study can be taken with a grain of salt as it was performed on mice but they are the number one labratory animal. Any reserach you read about cancer treatments are done on mice (because of the ethical problems of human subjects).
So why am I talking about the abnormality of microphallus? We’re looking for bigger penises, right? Well, this is, in a roundabout way, a small case for the inclusion of natural aphrodisiacs and testosterone boosters in penis pills. The authors of this study conclude that pre-pubertal hormonal therapy (mainly androgens) didn’t reverse microphallus in genetically altered mice. However, post-pubertal therapy did. This means that the size was increased after adult size was achieved. Now, androgen therapy can’t reverse gene-altered states but rather had to have increased the penis tissue size by other mechanisms, most probably by increased blood flow (or so they theorize).
Topically applied testosterone and phallic growth. Its effects in male children with hypopituitarism and microphallus.
Ben-Galim E, Hillman RE, Weldon VV. - Am J Dis Child. 1980 Mar;134(3):296-8
This is an old study but as far as I know it hasn’t been refuted. Here’s the gist of things: “Application of testosterone locally for this brief period is a safe, effective, and simple means of stimulating phallic growth. Our findings suggest that topical testosterone causes penile growth predominantly through its systemic action.” One can question the efficiacy of oral testosterone and testosterone boosters because the resulting increase in testosterone can naturally be used by other tissues. However, the increase in excitation caused by the PE exercises should make the predominant part go towards increasing penile size and also increase blood flow which is what we try to do anyway (by hyperemiation).
Let the discussion continue.