Cialis or Viagra for recreational use.

There is such thing as drug metabolism, ie how quickly your liver processes the compound.

Metabolism roughly translated is the ‘breaking up’ of compounds to produce energy or waste products.

Happy and crabapple, your both right, check my post earlier today. 10inch how did you damage your chambers?

Pepper

Very intense length-training regimen. I’d hang heavy weights for two hours a day along with 24/7 ADS/ANS extension. I got to the point where I wasn’t even hanging anymore, and I was just wearing my ADS strap around my waist. At the end of the day the fatigue in my penile chambers was unreal and also painful. It had a constant throbbing feeling. I had a hard time getting erect and I lost almost an inch in length and half an inch in girth. To give you something to base my pain on, it felt like when you are wearing your cockring for too long and your chambers start throbbing from lack of new oxygenated blood coming into the penis.

So to circumscribe the length fatigue belief: Yes, it IS possible to overtrain length.

Are you using the ED drugs as some type of rehab?

Pepper

Exactly. Let me get you a link on a motivating factor for me.

Here is something I copied from an MOS member, Bionic:

World J Urol. 2005 Dec;23(6):385-92. Epub 2005 Nov 5.
Treating erectile dysfunction by endothelial rehabilitation with phosphodiesterase 5 inhibitors.

Department of Men’s Health and Clinic of Urology, University Hospital Hamburg-Eppendorf, P.O. Box 202101, 20214, Hamburg, Germany. Frank.Sommer@me … and-health.info

A large body of evidence has accumulated demonstrating that a common pathway in conditions such as hypertension, atherosclerosis, hypercholesterolemia, diabetes mellitus, and erectile dysfunction (ED) is endothelial dysfunction. Although a complete pharmacological cure for ED is currently unavailable, the phosphodiesterase 5 (PDE5) inhibitors sildenafil, vardenafil, and tadalafil are efficacious oral therapy for ED. Results from recent studies suggest that regular treatment with a PDE5 inhibitor may lead to enhanced erectile function (EF) beyond that observed with on-demand usage, possibly through improvement of endothelial function. Such an effect may be viewed as rehabilitation of damaged erectile tissue. The present review focuses on several recent studies which provide evidence for the beneficial effect of regular PDE5 inhibitor administration on the improvement of EF by rehabilitation of vascular endothelium.
PMID: 16273418

J Sex Med. 2006 May;3(3):504-11.
Tolerance to the therapeutic effect of tadalafil does not occur during 6 months of treatment: A randomized, double-blind, placebo-controlled study in men with erectile dysfunction.

Australian Centre for Sexual Health, Sydney, Australia. Cmcmahon@acsh.com.au

INTRODUCTION: Tolerance can cause a decrease in drug efficacy during chronic therapy, possibly leading to treatment failures. AIM: The aim of this article is to determine whether tolerance developed to the effects of tadalafil on erectile function (EF) over a 6-month treatment period. METHODS AND MAIN OUTCOME MEASURES: Post hoc analysis of data from a multicenter, double-blind, randomized, placebo-controlled, parallel group study was performed. Men (> or =18 years of age) with erectile dysfunction (ED) were randomized to treatment with placebo (N = 47) or 20-mg tadalafil (N = 93) taken as needed for 6 months. This report focuses on efficacy assessed with the Sexual Encounter Profile (SEP) diary (diaries were collected after a 4-week treatment-free run-in period [baseline], and monthly for 6 months), and with the International Index of Erectile Function (IIEF) (administered at baseline, and at 3 and 6 months). RESULTS. The mean per-patient percentage "yes" response on SEP question 3 (SEP3, successful intercourse) was 33 +/- 4% at baseline, 74 +/- 4% after 1 month, and 78 +/- 4% after 6 months of tadalafil treatment. The IIEF EF domain score was 16.2 +/- 0.7 at baseline, 24.3 +/- 0.8 after 3 months, and 24.3 +/- 0.9 after 6 months of tadalafil treatment. In a subgroup of patients who took tadalafil > or =3 times per week (N = 24), the SEP3 score was 87 +/- 4% after 1 month and 93 +/- 3% after 6 months of treatment, and the IIEF EF domain score was 27.3 +/- 0.9 after 3 months and 28.5 +/- 0.4 after 6 months. Of 16 tadalafil-treated patients who discontinued, three cited a lack of efficacy. CONCLUSIONS: Tadalafil treatment significantly improved SEP3 and IIEF EF domain scores. The efficacy of tadalafil, taken as needed, was maintained over a 6-month treatment period in men with ED.

PMID: 16681476

Int J Impot Res. 2007 Mar-Apr;19(2):200-7. Epub 2006 Aug 31.
Relationship between chronic tadalafil administration and improvement of endothelial function in men with erectile dysfunction: a pilot study.

Medical Pathophysiology, University of Rome La Sapienza, Rome, Italy. Antonio.aversa@uniroma1.it

Men with erectile dysfunction (ED) frequently have a disproportionate burden of comorbid vascular disorders including atherosclerotic disease. We investigated whether scheduled tadalafil is better than on-demand (OD) in improving endothelium-dependent vasodilatation of cavernous arteries in men with ED and whether this effect is also exerted on markers of endothelial function. We did an open-label, randomized, crossover study including 20 male outclinic patients aged 18 years or older (mean age 54 years) who had at least a 3-month history of ED of any severity or etiology. Tadalafil (20 mg) on alternate days (ADs) or OD was administered for 4 weeks. Primary end points were variations of basal inflow (peak systolic velocity (PSV)) and flow-mediated dilatation (FMD) of cavernous arteries compared with baseline at penile Duplex ultrasound. Secondary end points were variations of Q13-SIEDY scores regarding morning erections and of markers of endothelial function, that is, vascular cell adhesion molecule (VCAM), intercellular cell adhesion molecule, endothelin-1 (ET-1), insulin and C-reactive protein (CRP). PSVs and FMD were higher after AD treatment when compared with OD and baseline, respectively (P=0.0001), and improvements were maintained from 2 weeks after discontinuation (P<0.005). Patients receiving tadalafil AD experienced a significant improvement of morning erections as compared to AD treatment (P<0.0001); ET1, VCAM and CRP showed a robust decrease after chronic vs OD regimes (P<0.05), with concomitant increase in insulin levels (P<0.05), without any variation in blood pressure and other laboratory parameters. Chronic but not OD tadalafil improves endothelial function with sustained effects from it’s discontinuation. Chronic treatment also produces a dramatic increase in morning erections, which determines better oxygenation to the penis, thus providing a rationale for vascular rehabilitation.
PMID: 16943794

http://www.life stages.com/heal … ction_2006.html

Bionic also stated:
"The take home message: a little daily tadalafil is pretty damned good for your Johnson and general health and does not create tolerance."

Listen,

They are talking about ED due to endothelial disfunction, caused by clogged arteries, diabetes, etc.. These are degenerative disorders causing artery wall disfunction. So they use the nitric oxide producing aspects of the ED drugs to assist the artery walls in becoming flexible again. Your situation is traumatic. Two totally different problems. When did this happen to you?

Pepper

I’ve been doing that routine since October 2006 and just recently decided to totally stop PEing for at least a month in late June. I should have stopped months before, however.

For men with ED, whether from diabetes, surgical trauma, medication interactions, vascular insufficiencies, a more robust regimen of the PDE5 drugs is often suggested - like daily. Doesn’t matter if it’s V, C, or L.. Gradually endothelial cell function tends to improve, and in many cases.

For those of you who get good boners without the erectile drugs and are just tinkering around with these drugs for “fun,” consider the long-range possibilities of psychological “addiction.” True, “tolerance” is not usually an issue, but do you need a monkey on your back when you don’t in any way need a monkey on your back?

Taking these drugs is actually counter productive to what your penis needs right now. You are experiencing ED because you injured your penis and it wants to heal, it’s irritated and inflammed and it doesnt want to be used, part of the healing process is loss of function, so the ED is actually protective. I wouldn’t do anything to it for 1 month, no ADS, no sex, no masturbation, try not to even get erections. Leave it alone and it will heal, it will contract a little yes, but once again, it’s part of the healing process. The ED drugs attempt to force erection, thats exactly what you don’t want. Does this make sense to you?

Pepper

For anyone doing PE, pain and disfunction should be used as a signal that the level of intensity of their program needs to be decreased or serious injury may occur.

Pepper

The erections are the only thing that feel therapeutic. Also, the liquid C doesn’t make me want to get an erection any more than when I’m not using it. As far as not getting hard or beating off, well, I just can’t resist playing with my toy every now and then. I’m going based off of intuition here. It is important to note that ED drugs do NOT make you want to get an erection.

Good point. But I have always been a responsible user of drugs and I think liquid C is no different. It’s not like I get bigger/better erections necessarily, it’s just that I don’t have to keep stroking to maintain an erection like I normally do. However, I don’t know if I’ll continue to use it regularly or not. I view it as a supplement. If it’s not going to hurt, why not take it? Just like protein when working out.

Yes, but sadly many, most notably girth (clamping!) workers, tend to let that rule slide. I remember back when I was clamping my erections wouldn’t be as good, but I set that aside because I was gaining in girth. Although, I have some bad discoloration from it now. I did get to my girth goal, however.

When I start back up I’m definitely not going to be going overboard. If I can’t get a rock hard erection I need to cut back some. It’s hard to do though, especially when you think more PE equals more gains.

You will be able to regain the lost size fairly easily once you’ve healed. Once erections are back to 100%, you will know your better and then you can start again, but this time be smarter about it. Avoid the ED drugs also because they will make you think you are healed when you may not be. When you break an arm, you immobilize it, wait awhile, then start moving it little by little, right, you don’t pull on it, stretch it, play with it or pump it up with drugs to make it swell. Please be careful whose advice you take, your young and want that thing to work for a lifetime. Does anybody else have anything else to add?

Pepper