Originally Posted by Janus Bifrons
Greetings Raybbaby,Janus Here,
My apologies,
I take for granted my knowledge base at times. While I was developing everything, I designed three experimental models of which the Angion Method was apart of. Out of the three methods, two showed the most promise. It was during the compiling stage that I repeatedly noticed how stretching caused an increase in collagen density, especially when done in a dynamic fashion. While static does not produce this effect, I repeatedly noticed it tended to cause points of weakness in collagen structures that increased the likelihood of catastrophic failure(tearing).
Do note that intermediate collagen density will find embedded in itself healthier endothelial sprouts than those found in lesser densities. In low density extracellular matrices, more sprouts develop (greater angiogenesis) but they are also weaker and less stable. On the other hand, if density is too great then sprout formation is inhibited. As with anything, it’s probably best to shoot for the golden mean.
What Janus says about "points of weakness" caused from static stretching is fascinating. I would be interested to see if the effects of dynamic stretching on extracellular matrices would suggest a way of continuing to target the ligaments and while simultaneously fine-tuning density to ideal ranges (1.2 - 1.9 mg/ml), all without lending itself to the creation of "points of weakness."
This would be difficult to measure, though. But there is good reason to think that too much static stretching/hanging could be detrimental to AM.
Source: http://web.stan ford.edu/group/ … lshornBauer.pdf
Perhaps the ideal route would be to prioritize AM, then after some time integrate newbie routine-level stretching? This seems a good way for a beginner to start out. For intermediates and advanced users, a de-conditioning break (proportionate to how tough one’s penis is) would yield similar results, I imagine, in so far as it is supposed to simulate the newbie phase and its associated gains.