I will preface this by admitting I only read the first 5 pages and the very last page of this somewhat old thread. But I want to comment, because I think perhaps the valuable data is nothing new here, and there are statements in this theory that are definitely false.
First, I think the analogy of the tube in the tire is a decent one for what is being examined in the research. You need enough “tube”/cavernous tissue to adequately expand against the tunica to create the necessary pressure required to stiffen the connective tissue of the tunica and get a good quality erection. Makes sense, less smooth muscle/cavernous tissue, less engorgement of said tissue, less internal pressure, less erection hardness/quality. There is, however, no definitive causal relationship here, so the loss of smooth muscle could just as easily be the RESULT of erectile dysfunction. Either way, all the researchers are saying is that it is a good predictor of ED and until someone shows that by directly increasing the ratio of smooth muscle you improve erection quality, that’s all it will be.
Is this theory and prescription really new information for us in the PE community? Absolutely not. When we are talking about how to enlarge the penis, we must keep our frame of reference restricted to how to enlarge the HEALTHY penis. What is being talked about in this research is a deficiency. And, with any deficiency, we should concentrate on correcting that before attempting to make forward progress. If you are two steps behind you have to catch up before taking two steps forward, so to speak. And when we have poor EQ what is usually the recommendation? Jelq, or similar exercises, that have more or less been anecdotally shown to improve upon this problem. Whether this is from increased circulation, improved smooth muscle tone, an increase in cavernous tissue, or a combination of those factors we do not definitively know, but, whatever the reason, it appears to be effective. :) Nothing new here people.
If you have a deficiency, absolutely you should fix it first, but don’t mistake that for the guidelines to layout your PE future in accordance with even after correcting the deficiency. This is where this hypothesis and prescription really goes off the rails, because in a healthy penis, smooth muscle cannot be the limiting factor. Plastic deformation and permanent expansion/elongation happens relatively quickly in all soft tissues, including smooth muscle, when compared to tough, collagenous connective tissues. Those connective tissues will always be the limiters - the last to budge. It HAS to be this way, otherwise connective tissue would be ineffective in its role in the body: to support other structures of both soft and hard tissues. Connective tissues have to be tough and resistant to deformation.
I appreciate the work and research that went into this those years ago, but if you are considering making this a model to establish your future PE in accordance with, consider what the research is actually saying. Correct your deficiencies, sure, but then you have to target those tough tissues to growwwwwww.
Almost forgot, the research speaks about a RATIO of smooth muscle to non-smooth muscle. Smooth muscle is correlated, if not causal, with erection quality. So even when going for length it is important to keep your EQ good, or if it is not important to you it should be! This is why hangers are recommended some light jelqing in addition to their hanging. Whether this works to directly maintain that smooth muscle ratio, and thus EQ (the causal scenario), or to maintain EQ through other avenues and thereby maintain a good smooth muscle ratio (the non-causal scenario), or a combination (the partly causal scenario), you should be doing some light work to keep EQ high even if you don’t desire to increase girth.
Roots