Microtears Result in Plastic Deformation

Tonight while searching for the correct spelling of the “Blachoe Ring”, while replying to another thread, I came across the Penis Enlargement Wiki. I decided to look at the information on the Wiki. I noticed the “Growth Theories” link, one which I have looked at in the past. I then clicked on “microtears”, because that is what I am finding occurs in connective tissue in order for permanent elongation to happen, according to the science behind physical therapy. In many cases, the term Microtruama is used. As I read the description of “Microtears” is see that the author states that plastic deformation and microtears are not one in the same, and that the two are clearly different.

Well, that lead me to this point. I’ve been wanting to debate whether or not plastic deformation occurs here on the forums, even though it has been debated over and over again. You see, I believe that both parties are right, to an extent, and I will clarify what I mean by that.

What misleads people is that they refer “only” to engineering mechanics when considering the definition of plastic deformation. In that case, the molecules of a material are stretched, creating permanent added length, but no new or extra molecules are added/created.

I also held this same definition of plastic deformation when I first discovered the term and the theory as it applies to PE. I reasoned that if my penis had 1 billion cells, and it permanently lengthened, then there is no possible way that it would still have only 1 billion cells. So, by that analogy, I disagreed with plastic deformation PE theory. But, I was not 100% sure that it did not occur in the ligaments. So, I kept the term in mind, even though I suspected the body would add new cells (extra replacement cells/more than the original amount) in the long run over a years time or so.

Now, lets take a closer look at connective tissue. According to information that I have found, about 47% of the penis is made up of connective tissue. Connective tissue is made up of collagen, and is very much like a ligament/tendon. I’m not sure how many members agree with me, but I believe that the connective tissue in the penis is the LIMITING FACTOR to developing gains.

Permanent elongation of a ligament “as a whole” is known as Plastic Deformation in biomechanics. Biomechanics is the science that orthopedic doctors and physical therapist refer to. So, to say that a ligament, or other form of connective tissue has become plastically deformed, is to say that it has become permanently elongated. Plastic deformation is not meant to explain what occurs on the molecular level in biomechanics. In both engineering mechanics and biomechanics plastic deformation is a result of MICROTEARS. The difference in engineering mechanics, the mircotears are not healed, and new molecules are not placed in the stretched areas, while in biomechanics, the body heals itself. The end result is new tissue, or extra collagen, which occurs by the process of inflammation, proliferation, and remodelling.

Notice, tissue damage occurs in the region permanent lengthening is achieved.

Now with the next bit of info. Since tissue damage has been created, the body must now heal itself. When the microtear or microtruama occurs, the contents of the cell spill out into the extracellular matrix, known as the ECM. This signals to the body that healing is needed, and this is the onset of inflammation. If the injury is large enough, swelling and redness are symptoms (this is in injuries such as sprains, and such a large injury is not a goal of PE).

In that quote we see that healing of connective tissue is done by tissue repair. Remodelling, the “R” in IPR is mentioned in this quote, but inflammation and so on are also mentioned on that page as the process is outlined.

The rest of the penis is made of smooth muscle within the corpus cavernosa, and corpus spongiosum. This smooth muscle accounts for about 48% to 50% of the penis. The remaining 3% - 5% must account for the skin, veins, arteries, nerves, and capillaries. The structures of the penis mentioned in this paragraph or the ones that can be overworked. This is why high intensity routines require rest days. These structures are “more” responsible for engorgement of the penis during erection than the connective tissue is. This is the basis for negative PI’s occurring when these tissues are overworked. I am not sure of the process by which these tissues become permanently elongated. The skin, and the walls of veins and arteries are also made of of collagen, but I believe that mitosis may happen in the skin.

In closing, we now have evidence that plastic deformation is merely interchangeable with the words permanent elongation, and it is not used to describe what is occurring on the molecular level. Microtears/microtruama occurs on the cellular level in connective tissue, which is healed by the process of IPR, and results in permanent elongation, which is plastic deformation of the connective tissue.

The link to the site is:

Here is another link. This link has presented me with the most scientific information that “I believe” explains PE theory.

In this information, the letters “CT” stand for connective tissue.

Here the term “microfailure” is used instead of microtears or microtruama.

Also, notice that here we also see more evidence that the process of IPR occurs as connective tissue heals.

This astounding link is:

The article is entitled, The Effects of Manual Therapy on Connective Tissue. This article is under the category of physical therapy at thefreelibrary dot com. I believe that were are merely practising physical therapy on our penis.

This thread has turned out to be more of a PE theory thread than I expected. I guess that I always draw things out and get into detail.

Generally, I would attempt to avoid posting back to back, but since I’m providing more information, I’m going to post again.

Here’s more information from a different website.

This is the best article/link that I’ve found yet. I’ve saved the information to notepad on my computer, and will likely copy it to a disk.

To take the information one step further, I’ll tell you that we have to enter the plastic range in order to create permanent elongation. In order to enter the plastic range connective tissues must be stretched to about a strain of 3%. In other test, the yield point has been established to be at 5.14% which I covered on another thread. Here’s the information about that 3% strain.

Also, it is important to distinguish stretching in the plastic range from stretching in the elastic range. The amount of force will determine which range you are in, or the amount of weight. I need to mention this information on the pound per minute theory thread. Now, we know that a minimum threshold of weight/force is required to create plastic deformation. Nonetheless, using an ads or golf weights can be supported by this information. If ADS’ing works in the elastic range all the time, it may make it easier for us to reach that 3% strain. In other words, for a rough example, we may be able to gain from less weight than would have been required without the extra use of an ads. Pre-exhaustion is a good term for working in the elastic range. This is just a theory of how ADS’ing works, but here’s the info.

While all of this information supports PE theory behind intense moderately intense work, it does not seem to represent what occurs when using a stretcher for long periods. What about the few guys that gained from only ADS’ing? Yet, after thinking about it for a while, and putting together some other information from other articles, I can see how this still applies.

From another article I learned that a strain in the elastic range takes 10 times the amount of “loading time” for it to revert back to normal. So, if you wore a stretcher for 10 hours, the very slight strain or small increase in you FSBPL would take 100 hours to disapear. If you again used the stretcher before the 100 hours was up, possibly about 16 hours after removing it on the previous day, you may be able to pile the very small amount of strain from one day on top of the next day. Eventually the additional stretch in the connective tissue will pass the 3% mark, and would likely be best to continue till passing the 5% threshold over days/months.

The link to this wonderful article is:

Here is a graph corresponding with the information. I did not create this graph.

Kojak, that’s a very interesting read, thanks. And although it was long, it provided some great info into what we’re doing here on the scientific level. Now, after reading that, I have a question for you. The article states that repair of the tissue takes place within the 48 hours after stress. Would this then be evidence that you should only PE 3-4 times a week or every other day? Back to back days would not be wise cause you would be preventing the tissue from healing, increasing in size and thus possibly hurting your gains?

Most of the veterans I think suggest every other day or 3-4 times a week anyway.

I feel that this PE theory is compatible with many, if not all aspects of PE.

I mentioned how overworking all of the other tissues in the penis besides the connective tissue results in negative PI’s. I believe that extremely overworking the connective tissue could also result in negative PI’s, but this is rarely done. This would be similar to a bad sprain.

Also, this information is compatible with our findings in the PE community that many different types exercises and schedules result in gains. Because it is merely that we stretch the connective tissue far enough, jelqing, stretching, hanging, etc… have all yielded gains for different individuals. Even enlargement from priapism (megalophalus) fits in these findings. I’ve gotta say that I believe clamping really fits this info well.

Another point that I’d like to cover is that many members believe that we are all different, and that what works for one may not work for another. To address this, I’d like to say that gains are established by the same processes on the cellular level as everyone else. The things that will vary are the amount of force required, the amount of time the the force is applied, and the amount of recovery time needed. These things will vary from individual to individual.

The fact that we are each more proficient at different exercises or routines makes some more effective for us than others. Also, some members require that more force be applied to their penis than others, so certain methods are more effective for each individual.

Age also plays a role. The amount of force required for an older individual to cause plastic deformation in their connective tissues is lower than the amount of force required for a younger individual to do the same. This is due to the fact that younger people have more elasticity in their connective tissues. Genetics may also affect this, and for example, not all 35 year old guys will have the same amount of elasticity. Less elasticity would mean that a guy would need less force to enter the plastic range. Older individuals generally require more recovery time, but that is a general estimation. That covers my opinion on how we vary from one member to the next.

I don’t that we will ever be able to identify an exact amount of weight/force or amount of time that is perfect for everyone. This would be wonderful, but the amount of force required for John to reach the 3% strain may be more than is required for Bill. Nonetheless, both will have to reach the 3% mark for permanent change to be occurring.

We have been able to give ranges and estimations of force required, time of applying the force, and recovery time. I believe that we will be able to narrow the ranges down some in the future, but as I said before, there will always be a range.

Kevin12, I have not focused my efforts on determinig the optimal amount of rest days, yet. I would recommend that you follow what is commonly recommended on the board. Even though there is some variance, the vets here know their stuff.

Also, about the 48 hours, the information states that tissue repair begins within 48 hours. That has a different meanig from “the repair takes place within 48 hours”, which sounds like it may have been completed, but that is not the case.

I will be testing various schedules myself. I’m currently on a break.

Newbies, please follow the newbie routine. This theory even supports what goes on for newbies. Newbies don’t have to apply as much force to their penis in order to create microfailure or microtears. The tissues in your penis are fresh. Collagen has not yet alligned itself in the most optimal way to resist stretching.

BINGO! In my opinion the most important factor is to determine work/rest ratio(how much to PE and how long to rest-on micro and macro scales) for each of us. Moreover-I suspect that such ratio is not ‘once given’ and it changes throughout our PE journey…it is like chasing the rabbit with eyes closed…but here is the secret in my opinion and here lays the key to gains.

So Kojak would you say that newbies should take is easier cause it will be better for them in the long run? Meaning that if you start slow you have a better chance of gaining over a long period of time cause your penis will take longer to build up an immunity to the various exercises and greater force that you give over said period of time. I hope that makes sense.

BTW, I’m currently doing a one on/one off routine.

Just wanted to bump this. Kojak have you discovered any new information or support in your findings since you last reported on this?

I’d like to see a more clear definition of “plastic deformation” as it applies to the penis.

And point out that we are all different, on both a chemical and molecular level. Is the process the same for all ages/strength of force? I am not at all sure it is.

Kojak10…

Very interesting stuff. Wonderful contribution. Thanks.

I have two questions:.

Your documents say that “micro-failure begins at 3% elongation.”

My normal flacid approaches 6” or 152.4 mm, but a static stretch gives me 8.5” or 215.9 mm

My first question is this: 3% elongation … of which value? 152.4 or 215.9

I assume that it must be the latter.

On that assumption, my second question is this: it takes a bit of force to stretch my 152.4 mm to 215.9 mm,

more than a typical ADS provides.

3% elongation of 215.9 = 6.48 mm, or just a hair over 1/4”.

The force to get that last 1/4” in order to have 3% elongation is quite large.

I don’t think one could get that with any type of ADS, yet

your analysis (if I am reading it correctly) suggests it should.

Am I reading you correctly?

What are your thoughts?

DD.

That quote you include….tearing, healing, scar tissue, etc….has, I believe, nothing to do with actual PE growth:

(1) If collagen actually “has the property of gradually shortening when it is truly formed,” then PE is an exercise in futility….OR….collagen has little to do with PE gains.

(2) If “new scar tissue will always shorten unless it is repeatedly stretched,” then my gains have had nothing to do with scar tissue formation (read: I’ve gone an entire YEAR, cold turkey, with no PE whatsoever - and only lost about 1/8” of the approx 2” of length gains I’d achieved; and, only about 1/8” or so of girth).

If our gains are about scar tissue formation, and I’ve more than doubled my erect size (volumetrically), then - according to your theory - my penis must be a mass of scar tissue. But it’s not. My penis looks the same (except a lot bigger!), it feels the same to the touch, and my sensitivity seems the same it always was [none of those qualities should be present if my penis were a mass of scar tissue].

The article was packed with info, I just don’t think it addresses the unique situation that we are involved with.

I second wadzilla : scar tissue = non functional tissue, so, according this theory, we all had to be impotent.

It is a bout cell replication. Are all the components there to grow dick cells. The stem cells that are formed in the bone marrow that will eventually become penis cells go to replace dead cells. The object is to have more cells grow than die. Now how do we encourage that?

So genetics designed a certain length and shape to the penis. But we have proved over and over again that we can grow and thicken it. Cell hypertrophy, how do we encourage this?

First we stretch the ligaments and then we work their tunica for growth. Growth slows how do we speed that up again?

Constant rotating and changing PE may be the answer. Dick gets bored of the same ol same ol. The key to this how to find the right flavor of the week. The right PE routine that will spur new growth.

I think the key is telemeres, telemeres shorten become frazzed from distress. Exercise rebuilds the telemeres in the body. PE renews the telemeres in the penis.

Good nutrition must be present. Good diet high in anti oxidants to kill off free radicals. Zinc and vitamin “C” to to normalize the hormone testosteron. “A” and “E” for skin and “B” vitamins for metabolism. Minerals are necessary to in the right amounts. Magnesium, calcium, potassium, sodium, Chloride, don’t forget phosphorus or vitamins are useless without minerals.

Protein in adequate amounts for hypertrophy is needed. Not just enough to get by but enough to encourage cell replication,repair and cell health.

Biblically speaking we should be living to 120-130 years. To have the issue of donkeys and the flesh of horses like in the Bible we must be missing something what is it?

Hypertrophy isn’t the multiplication of cells, but the growth of existing cells.

New cells born in a tissue in four cases, by what I know:

1) when cancer developes;
2) when scar tissue is formed;
3) when tissue is broken (ex. bones);
4) when hyperplasia developes: but hyperplasia phenomenon never has been observed in humans.

So, I’m pretty skeptics that PE is related in someway with those studies.