Possible reason for PE induced growth

So it’s better to stretch the hell out of our dicks for a short period of time instead of long periods of stretching?

Low level but steady action probably beats heavy duty cycles. I tried both and I think that most of my (moderate) length gains came from low weight hanging and ads.

Less potential for negative PI’s and injury also speak in favor of low level activities.

Hear me, Mr. Happy?

I agree. I guess it’s only human and I am personally at risk. However, I couldn’t say that I mad good gains while overdoing. Usually, overdoing causes some kind of negative PI’s or nmild injury like blisters and so on. This requires rest to heal. Time enough for your dick to recover and get back in shape (it’s shape before overdoing).

Someone said in this forum that the the body has the tendency to maintain it’s form, shape etc.

And that that is what our lives (bodybuilders’, or athletes’) lives so hard. To overcome this tendency requires hard work.

It can be accomplished. Good luck with you all’s efforts in the new year.

I don’t believe that tension-induced remodeling favors one way over the other, per se. It responses to all kinds of stress, including injury. Your suggestion of a short very intense workout will probably stimulate more fibroblasts causing more of a collagen making response causing a collagen fiber diameter increase over length. This would be like a muscle tendon exposed to a great contraction stress. It would increase fiber size to prevent it from being ripped off bone or other injuries with continued high tension stresses.

ttt - I agree. I’ve also had my best gains at lower intensities. But that hasn’t stopped me from going high intensity as well.

Hi Stormy,

The story on Relaxin as I have been told, and to a degree, followed up on, was that the hormone Relaxin was duplicated around 10 years ago, but that there was no interest in it from the obstetrics field. Later, some researchers wanted to try it as a possible treatment for Scleroderma, and some was made. It did not help the Scleroderma patients, and the remaining stocks were sold of to some orthodontists who wanted to try it with adults to see if it would help them in remodeling the gums. Dr. Adams tried to get the remaining supplies, but the orthodontists beat him to the punch. Apparently it was not helpful to the orthodontists, so no more was made.

As far as I know, Magnumforce never posted pictures. As I recall, Magnumforce was not small to begin with, but I do not remember what his starting size was. I think that his erection strength was quite strong, but again, that is must memory.

Girth gains so far after 1 1/2 years have been a bit over 1 inch in circumference.

The question about body types is a very interesting one to me. I don’t think that with the use of IGF-1 that it makes any difference, so the girth gains from that method would not seem to be of any effect. The length issue has been discussed and there has been a number of interesting comments about it. (I know the next question is where was it discussed, and I have forgotten that). The thread had a lot of interesting discussion about the construction of the tunica. According to the thread, some of us do not have as much collagen in the tunica as others do, hence, elongation is easier to achieve. That certain people are more successful in PE seems to be validated here on this board. If the cause is the relative differences in tunica composition, well, I don’t know, but it would be interesting to get more discussion on. I suppose that the only bad thing here is that we can’t change our genes, so while we may have a thick tunica, there is not much that can be done about it.

As to other factors such as blood type or other areas of the body, well, I don’t know. Again, it would be interesting to get some opinions from others.

Best of luck,

Stage

Nice job, pudendum! Carry on!

I just can propose analogy with obesity.
Why it’s so hard to lose weight? Because there are a huge amount of newly formed fat cells throughout the body. The only natural way is to reduce lipid content within cells of fat tissues by diet or energy consumption through training. Otherwise you need to remove these cells by liposuction.

I strongly believe that PE gains are reached for the most part through tissue growth rather than tissue stretching as rubber. Even in case of ligament stretching while the inner part of cavernous bodies is released. This inner part also should grow to be released outside. The common theory is that cavernous bodies grow due to sinusoid dilation. But I hope for new sinusoid formation. All the more endothelial cells (inner vascular cells, vascular lining) like smooth muscle cells (vascular wall cells) and fibroblasts (connective tissue cells) have outstanding ability to divide and form new blood vessels.
Thus, maybe PE and “cementing” are achieved because of whole penile cell number increase? And insignificant decrease of penile dimensions during erection or PE exercises free period is due to extracellular matrix (network of collagen, elastin and other structural proteins) degradation?

I’m agree that the limiting factor of PE is tunica albuginea. The substance of erection is counteraction between blood pressure in cavernous bodies and elastic resistance of the tunica. Erection for tunica is the same load as hard working on tendons. In contrast to puberty when long-lasting erections appears to be PE stimulus, in maturity erections can only induce penile tissues strengthening. The ADS is unusual and sparing load that probably can provide tissue remodeling appropriate to our PE aims.
There is not only tendency to maintain body’s form, but also tendency to maintain composition and quantity of cells in tissues. That’s why it’s so hard to bring cells to divide and harder to bring them to die (in regard to “cementing”).

Can someone put this in simple layman terms or street people? Does this mean that we should focus on longer or shorter stretching? I thought there was another thread that said longer and low weight stretching=better.

I think so.

We just try to understand natural laws before recommend you something.

The concept of tension-induced connective tissue remodeling is well documented in the medical literature. There are no specific studies on the connective tissues of the penis and there won’t be. Our goal in this thread is to understand how tension in other connective tissues brings about growth and how this applies to the penis.

If you come to this thread looking for answers for what specific type of PE or intensity is necessary, you will be disappointed. The mechanisms presented here definitely apply to the connective tissue of the penis (suspensory ligaments and tunica). It would appear that longer lower intensity PE might be more beneficial since stronger tension tends to cause tendons and ligaments to get thicker and stronger, not longer.

I’m sorry you are disappointed, but answers unfortunately tend not to fall in our laps. This stuff is also complex. I and others have tried to explain this as simply as possible. Somethings are explained several times in different ways to get people who are not in medical fields or the sciences a better chance to catch on. It’s going to take continued thought and debate to try to ferret this all out into usable recommendations.

If as you read this entire thread you have specific questions or comments that will add to this discussion, please chime in. Ideas are the key to advance our understanding.

Necessity of cementing gains is in someway not well explained by the mechanisms here exposed, seems to me, but I can say that I experienced lossen of gains when had no chance to do that “cementing”. Maybe there is something else involved in PE gains.

Just to guess: maybe penile cells are regulated by a “last in, first out” principle, so the new cells are the first to be affected by the “disuse atrophy” you was speaking about, pudendum.

About jelq effectiveness, maybe I’m going to be somewhat paranoid on this argument.

The sole effect of blood-pressure seem unable to explain, at some degree, why with jelqs one gets girth and length gains in a balanced way, where, with clamping (blood-expansion work) the main effect is on girth.

There are 2 kinds of jelqs:dry-jelqs and wet-jelqs. The first one seem to work basing on that blood-expansion way: it is well known that dry-jelqs give mainly girth gains.

Wet-jelqs are based on two actions: a) blood-expansion, that forces the tunica; b) finger-massage. This second stress, I suppose, can ..uh.. split or smear (not secure about the right word, sorry) the tunica tissue longitudinally - this could aid the length gains.

Basing on that, I’m trying a new exercise (well, I don’t know if it’s really new, but haven’t found nothing similar posted before) : when I do fulcrum stretches, I push with knuckles on penis, doing a longitudinal massage; I figure this could be more effective for that fiber-smearing I speaked above.

I tried it yesterday for the first time, and after about 30” I found that I was able to stretch the penis about 1/4” more than habitually can.

I want to try this exercise on erect penis, and even with clamped penis, but I have a bit of fear.

What you guys think about?

The losses that happen with disuse are losses in connective tissue fibers. But something causes the fibers to be deconstructed. Whether this is a function of fibroblasts or other inflammatory cells is unknown to me. I think you’ve it onto a very interesting subject to investigate. I’ll get back to you.

There are two layers to the tunica of the penis. The inner layer has circular fiber arrangement (circumferential), the outer layer has longitudinal arranged fibers.

I think that balanced effect from jelqing is a problem. Many of us have had a baseball bat shape to our penises from imbalanced growth from the pressure and volume characteristics of the stroke. Blood accumulates in front of the stoke causing the greatest tensions the further up the penis you jelq. It requires other girth exercises to bring balance back (I’ve done reverse jelqing).

My belief is that jelqing gives far greater girth effects than length, that is the greatest tension is circumferential. I would think that the elongating effects would be towards the end of the power stroke as you reach the far closed ends of the cavernosa. Here is where tension would be both circumferential and longitudinal.

Any method that allows you to direct the tension longitudinally should be effective.

I would be concerned that if you did this at full or peak erection, that you would generate tension that would lead to fiber width increases but not lengthening. Do you think you are generating enough longitudinal tension without being fully erect? I still think that the best possible tensions are generated when you start with your penis at less than full erection. Starting higher than this, you are stretching a poorly compliant tunica and tension-induced connective tissue remodeling will probably give you the thicker fibers that will “protect” the tunica against injury with future similar stresses. In other words, stronger not longer.

Let us know how this method is working and if it does (and no one else has posted it), you need to post a thread.

Heat. I think warm ups and relaxes the tunica. Example, when doing fowfers the penis is tucked snugly between a testicle and a leg. In time it warms up as it stretches. Longer the warm up period the better for stretching and jelqing or even hanging. Warm up makes the PE more effective.

Be careful when working on your erected dick with your knuckles. Particularly sensitive areas are the top sides (blood vessels and nerves) and the underside (urethra). I know that you know that, it just comes to mind, for the sake of protecting curious newbies (and others).

Hear me, Mr. Happy, my friend?