With all due respect,You may be skipping the whole point, I understand what you mean, and it makes sense but I think your missing the following.
Yes mesomorphs are well built from the start, but it’s not just their frame (bone structure) that makes them gain weight (muscle or fat) easier.
From what I understand (and have studied) it’s the genes of the mesomorph that can gain mass faster than an ectomorph, and not just the frame, I have studied about the theory and the genetic metabolism for each group.
You mention that penis size is not related to somatic types, which is correct, but the fact still remains that tissue gaining potential does relate to soma types.
So who’s to say that gaining penis tissue isn’t the same as gaining muscle tissue (after all the penis is made from 50% muscle)
An insight on muscle (tissue):
Muscle is the contractile tissue of the body and is derived from the mesodermal layer of embryonic germ cells. Muscle cells contain contractile filaments that move past each other and change the size of the cell. They are classified as skeletal, cardiac, or smooth muscles.
I have also studied a lot from Remek at the PE gym, who has in turn studied a lot about the penis being 50% muscle. If this is the case and the penis being 50% muscle and the mesomorph being able to gain muscle easier than lets say any other genetic body type, then shouldn’t we at least entertain the chance that they could hold the gene to easy PE gains?
Penis tissue:
I have put some interesting facts below about the smooth muscles of the penis, maybe this will explain to anyone who didn’t realise that the penis is 50% muscle:
The important role of corpora cavernosal smooth muscle in potency has been known since Goldstein et al reported the first examination of erectile tissue. 8 Normal smooth muscle content and function are necessary for the initiation and maintenance of erection. 6 Published reports suggest that the average intracorporeal smooth muscle percent is between 40% and 50%. 9 Our unpublished data confirm this rate with the finding of an incidence of smooth muscle of about 49% in normal potent males in the general population. In contrast, patients with veno-occlusive dysfunction show a much lower percent on microscopic examination. A prior study suggested that these patients have a smooth muscle percent of 10% to 36%. 9 Concomitant intracorporeal fibrosis results in abnormal smooth muscle function, increased venous leakage and eventually impotence.
* Because the penis is compromised of 50 percent smooth muscle, and smooth muscle has a lot of the basic properties of skeletal muscle (the biceps, triceps, and all the other commonly known muscles), we can presume that it might react to stress the same way normal muscle does. Which is a no brainer for us all! What are we doing here? Exercising! Moreover, nearly every single guideline we have is based off body-building/exercising/weightlifting concepts in one way or another! Think about it — Bib, presumably one of the biggest gainers (if we accept his claims as facts), used a weightlifting concept known as "progressive overload." Peter Dick, another big gainer uses a common weightlifting program known as "muscle confusion" (in which he keeps the muscles guessing). And now, more recently (thanks to guys like you [xeno], Shiver, MX, Modesto, Wadzilla, and many more) we are now realizing cyclic training using deconditioning breaks helps us keep the penis in a responsive state. And cycylic training is a very popular weightlifting principle (if not the biggest).
* Albeit, this doesn’t particularly mean we can attribute the growth to smooth muscle growth — but regardless, if the penis grows, the smooth muscle must grow too. The article I quoted above stated: less smooth muscle = higher chance of ED. Obviously this isn’t the case for us PEers. Most of us report harder erections.
* Also, It is well documented that smooth muscle can grow due to stress. I have a whole book on the subject, in fact. It is called [you]
Hypertrophic Response in Smooth Muscle[you], by Charles L. Seidel and Normal W. Weisbrodt. It appears this book has numerous scholarly articles on the growth of smooth muscle. Link: http://www.amaz on.com/gp/produ … .Fencoding=UTF8
* I also think this can explain why PE gains are permanent (for the most part), opposed to traditional muscular growth gains. Think about it like this: the smooth muscle in the penis is exercised when we jelq, stretch, and incorporate other exercises. These exercises take the penis (and the smooth muscle) beyond it’s normal threshold. But what is the normal threshold? An erection. Masturbation, sex, anything that involves an erection is exercise too! It’s typically just not enough to cause growth. Therefore, after the gains are cemented, and we retire all we need to do to keep the gains is have frequent erections — in which the erections are enough exercise to keep the gains. It is also well documented that as men get older, and they use their penis less, the smaller it becomes. Note: I don’t have a reference for this off-hand.
In an essence, this article went over (in detail, down to the chemistry) the smooth muscle’s role in the penis. The bottom line: smooth muscle is very, very important. Or, at least that is what I perceived it to be. The smooth muscle causes an erection (which is set off through chemical reactions) by completely relaxing. "Compression of the relaxed trabecular smooth muscle against the fibroelastic tunica albuginea causes the closure of the emissary veins," and thus results in an erection. If this smooth muscle is unable to completely relax, then an erection can’t take place… . .
Theory: (a and b)
A: A mesomorph has greater ability’s (genetically) to gain tissue overall than the other groups of morphs.
B: The Penis consists of tissues.
Are there any points of view on this post.