Wow. This started out as a really interesting thread. With no disrespect intended toward those engaged in the present controversy, I’d like to get back to the substance.
I have long held a belief that cell division has little to do with PE gains. The reasons are as follows.
1) The envelope of the penis is composed mostly of collagen. Collagen is not a cell, but rather a product of cells. Thus, I’ve surmised that the tunica and ligaments can be stretched, and even repaired and strenthened, without requiring any new cell division. There is likely a limit to this type of growth, however, which I suppose is imposed by a need to maintain some minimum ratio of cells to cell products in the connective tissue.
2) The enlarged envelope (tunica and to some extent ligs) is filled primarily with blood. Blood, being a liquid in relatively large supply, will rush in to fill the newly enlarged tunica.
Thus, both the envelope and the contents (blood) can be enlarged without the need for cell division.
A possible weakness of this theory is that the trabeculae that make up the inner spaces of the CC’s include blood vessels and smooth muscle, as well as collagen and elastin. As the CC’s grow, the blood vessels and smooth muscle may need to enlarge as well. Some of this enlargement may require cell division.
Incidentally, this last fact may help to explain why many hangers experience gains in FSL faster than EL. First, connective tissue is stretched. Later, blood vessels and smooth muscle grow to enlarge the trabelculae and thus enable blood to fill the extra space that hanging has provided (that’s a wild-assed theory).
Great thread. Let’s keep it going.