Sorry for my English
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At one time I wore a vacuum stretcher 12 hours a day for three months. And instead of length, I gained 1 centimeter to my girth. Uniformly throughout the length. What do you think is the growth mechanism involved here?
Sorry for my English
Although I appreciate your enthusiasm, your assertive feels premature. It might make sense to tone it down a bit as you explore this idea.You should definitely pursue your hypothesis in whatever ways you can, but perhaps consider softening the certainty and assertiveness of your language as you explore it. It’s still just an idea at this point and honestly I can’t think of a way that one might ever prove anything here as definitive.
Since you want a method that is grounded in science, I think it’s important that you take a scientific perspective and hold hypotheses as such, and not as established fact. The rationales you present as possibilities as the biomechanical stimuli for growth may well turn out to be reasonable but there is little way for us to ever be certain based on actual peer-reviewed data, either from controlled studies involving subjects or bench work evaluating tissue markers of growth and/or its underlying biochechemical mechanisms. In the end all we will ever have is anonymous self-reported protocols and results, and the variations in exercise, recording and reporting techniques will be too much to ever make firm, much less definitive, conclusions about. Published research about similar tissues, while interesting and possibly revealing, may or may not apply. It is hard to know.
Remember that scientists pick a theory and then try to disprove it as much as find supporting evidence and a great scientist is humble and unattached to a theory. I can tell you from direct observation that the scientists who end up in dead end research are the ones who are the most dead certain about their hypotheses being fact and the least open-minded and curious about being wrong.
I can also tell you (again with a significant amount of high-level educational and professional understanding) that as a general matter that humans are mostly all the same, but there is a LOT of anatomic, physiologic and biochemical variation. Connective tissue variation is no exception. I’m not making any claims about the penile connective tissue properties as either uniform (and uniformly responsive) or not. But that’s because I try not to claim things as fact that I can’t be certain are actually facts.
Being skeptical is one of the most powerful tools of the exploring researcher, and it is absolutely the most powerful when confronting our own scientific assumptions.
I’ll follow this thread with interest.
Well said and 100% agree
Although I appreciate your enthusiasm, your assertive feels premature. It might make sense to tone it down a bit as you explore this idea.You should definitely pursue your hypothesis in whatever ways you can, but perhaps consider softening the certainty and assertiveness of your language as you explore it. It’s still just an idea at this point and honestly I can’t think of a way that one might ever prove anything here as definitive.
Since you want a method that is grounded in science, I think it’s important that you take a scientific perspective and hold hypotheses as such, and not as established fact. The rationales you present as possibilities as the biomechanical stimuli for growth may well turn out to be reasonable but there is little way for us to ever be certain based on actual peer-reviewed data, either from controlled studies involving subjects or bench work evaluating tissue markers of growth and/or its underlying biochechemical mechanisms. In the end all we will ever have is anonymous self-reported protocols and results, and the variations in exercise, recording and reporting techniques will be too much to ever make firm, much less definitive, conclusions about. Published research about similar tissues, while interesting and possibly revealing, may or may not apply. It is hard to know.
Remember that scientists pick a theory and then try to disprove it as much as find supporting evidence and a great scientist is humble and unattached to a theory. I can tell you from direct observation that the scientists who end up in dead end research are the ones who are the most dead certain about their hypotheses being fact and the least open-minded and curious about being wrong.
I can also tell you (again with a significant amount of high-level educational and professional understanding) that as a general matter that humans are mostly all the same, but there is a LOT of anatomic, physiologic and biochemical variation. Connective tissue variation is no exception. I’m not making any claims about the penile connective tissue properties as either uniform (and uniformly responsive) or not. But that’s because I try not to claim things as fact that I can’t be certain are actually facts.
Being skeptical is one of the most powerful tools of the exploring researcher, and it is absolutely the most powerful when confronting our own scientific assumptions.
I’ll follow this thread with interest.
Thanks for your response.
I’ve not made anything certain, and I’ve tried to make it clear that these are just theories with a few scientific studies to back up methods. I apologize if I sound too certain. My only goal here is to get people talking on the subject of girth gains and to question the original method of clamping when there may be something more efficient. All I’m trying to do is make others understand that there has to be something we don’t understand about girth gains yet, and it needs to be questioned.
Everyone is thinking of clamping at the base to fill the shaft with blood in hopes it’ll slowly expand due to the outwards pressure and engorgement. If that’s the case, then why is the majority of people gaining girth at the base at a significantly faster rate than the shaft? Why is it that people gain where there is no increased blood flow? It’s something that doesn’t make sense according to the widely accepted clamping approach.
And you are right, not every body is exactly the same, and there are many variations. Although I believe that in PE, there is no way we can ever find a method for every single person who varies too significantly from the average human’s physiologic and biochemical responses. That’s why we need to find a routine that is backed by science and works for the majority of humans. Will there be people who are unaffected due to a slight variation of some biochemical or physiological response? Absolutely, but we can’t let that stop us. All we need is a theory with scientific studies as its foundation and have people test it.
I’m on my decon at the moment, and I’m currently following Kyrpas and longerstretches’ approach to length gains, and I will continue that until I reach my length goals. They are a perfect example of a theory that is scientifically backed and works for the majority of human bodies. I’ve already gained 0.5" in 3 weeks by doing it, and I’ve been doing PE for slightly more than 3 years now.
When I’m done and my length goals are accomplished, I would definitely practice the theories I’ve shared here. But it may be a couple of years before that happens, as my length goals are quite ambitious. Maybe people can start their own theories in this thread that show why gaining girth and length at the same time is beneficial. If that’s the case, I would gladly practice the girth theories. But until then, most community theories based on collagen science seem to go against it, and I don’t want to risk hindering my future length gains.
No penis is different. What works for one, will work for another.
At one time I wore a vacuum stretcher 12 hours a day for three months. And instead of length, I gained 1 centimeter to my girth. Uniformly throughout the length. What do you think is the growth mechanism involved here?
I’m not too sure. By vacuum stretcher I’m assuming you mean an all day stretcher. Here are some possibilities you should think about:
- You made a mistake in measuring. Its 1 centimeter and its easy to inaccurately measure at such a number. I would say this is the most likely situation.
- The ADS was too tight. Some people have been known to gain girth through heavy hanging. Generally ADS aren’t used to keep the penis at a high tension, most aren’t even capable of doing that. Although the ADS I wear can definitely be used a "hanger" although I don’t recommend it as its difficult to know what weight the tension is.
I cant think of anything else, and I cant provide a definitive answer as I just simply couldn’t know. But you are a unique case if you did indeed gain and didn’t make a mistake with measurements.
No penis is different. What works for one, will work for another.
I believe this to be the case. Blood vessel proliferation due to obstructed and under-oxygenated where the clamp is placed.
There is another factor that might be at play and that is anti-lox which are produced under asphyxiation, leading collagenous fibers to a state much like the EDS (Ehler Danlos Syndrome) which is more elastic. I have this theory that people with EDS have pretty fast and huge gains due to this condition. However they don’t live long for that same reason (heart issues).
That is interesting! I’ve not heard of that condition but ill research more into it. Maybe there’s a way we can intentionally give ourselves this condition to improve our PE gains :D
No penis is different. What works for one, will work for another.
Hey everyone!
I was going through old threads hoping i could find some theories based off studies and I found this:
VEGF, AKT and You
This poor guy wrote all this in 2021 and only got 1 reply which was 2 years later.
I’m impressed with him trying to think outside the box and escape the conventional thinking. Its cool to see him show an interest in the growth factors that come into play during a hypoxic environment, and its even more awesome to see him connect the dots and mention clamping. Its just unfortunate that nobody seemed to show any interest in the topic. He was obviously ahead of his time.
No penis is different. What works for one, will work for another.
Scientifically speaking, girth has been solved. Now some might consider this "solution" as an approach with some asterisks, but the fact remains that significant girth is attainable in relatively short time with relatively low risk (keyword: RELATIVELY, since similar yet alternative methods can have disastrous consequences).
In 2011 I managed to gain 1-whole inch in circumference in 1-hour’s time. For another 3 to 4 years I had subsequent appointments for more girth and for aesthetic revision (to make it appear more natural). I have not lost any size size since and I have not had any partner (in the double digits) ever question my enhancement.
If you couldn’t already pick up what I was putting down: I had "work" done via cosmetic medicine, or more technically known as phalloplasty. (Note* this term over the years has morphed into becoming almost exclusive to transgender reassignment surgeries, versus actual penis enlargement surgery). I had dermal fillers injected into my shaft skin and I’m the thickest most (if not all) my partners have had.
There are other means and methods of enlargement that have been refined over time to produce higher efficacy as well, some temporary, some permanent. Of course, like all things in PE (exercises, surgeries, or supplements), nothing is without risk. Both complications and aesthetic irregularities can occur. And it would be ridiculous if I failed to mention that it comes with a price tag that far exceeds organic PE (i.e. the kind of routines you find on this site). These were the asterisks I mentioned in my opening remark.
Injectable phalloplasty, dermal grafts, and the P-Long Protocol are proven girth enhancers (with the first two methods being guaranteed); Ligamentolysis and the P-Long Protocol are also proven lengtheners (although these require PE exercise routines in addition, so a bit more hit-or-miss and not guaranteed if you don’t have the commitment). These are performed by medical professionals and the materials/fillers involved are FDA approved and can be used off-label for things like girth enhancement. Note*- avoid high volume injections of silicone as well as any rigid silicone implant; they tend to possess the most challenges & complications.
Regarding the P-Long Protocol: this is a non-surgical method backed by a study (albeit with a small sample size) that aims to increase length & girth. Sure, it may not be the industry-breaking Study I was hoping for, but certainly a step in the right direction. This involves a prescribed routine (a.k.a. Protocol) tailored by a Urologist (Dr. Brandeis) involving a stretcher & pump (of medical-grade quality, which they supply), a monthly PRP shot, and supplement(s) with a (presumable minimum) 6-Month commitment. The P-Long Protocol suggests 1-inch in length and 0.5" in girth after the completion of the program. Affiliate physicians around the country have began offering it so I cannot speak to its longevity/permanency yet.
To be frank, I wasn’t sure what to think about P-Long but I figured if guys can already achieve size with PE exercises alone, why wouldn’t a tailored routine combined with "medical enhancements" backed by a Study not have just as (or more) merit than standard organic PE? Especially by a Doctor who studies Sexual Medicine. Furthermore, a long-time veteran of my site (who I know well and trust) has begun the Protocol and has already seen gains in length & girth, and he’s not even halfway near completion. So it is exciting to say the least — if it weren’t to be the success it was intended to be, it would still open avenues on how science & medicine can modify and tweak the Protocol to prove more successful for a greater number of men (because what I do notice is how some techniques yield results for some guys while the same technique fails to provide gains for others).
In summary, if it is girth science you want, modern male phalloplasty is a demonstrable success. Some future day this will likely be a stem cell advancement, and even more into the future, bionic dicks!
If you're ever considering a surgical (or non-surgical) route for penis enlargement:
PhalloBoards: A Forum Devoted to Penis Enlargement Surgery & Non-Surgical Procedures
Scientifically speaking, girth has been solved. Now some might consider this "solution" as an approach with some asterisks, but the fact remains that significant girth is attainable in relatively short time with relatively low risk (keyword: RELATIVELY, since similar yet alternative methods can have disastrous consequences).In 2011 I managed to gain 1-whole inch in circumference in 1-hour’s time. For another 3 to 4 years I had subsequent appointments for more girth and for aesthetic revision (to make it appear more natural). I have not lost any size size since and I have not had any partner (in the double digits) ever question my enhancement.
If you couldn’t already pick up what I was putting down: I had "work" done via cosmetic medicine, or more technically known as phalloplasty. (Note* this term over the years has morphed into becoming almost exclusive to transgender reassignment surgeries, versus actual penis enlargement surgery). I had dermal fillers injected into my shaft skin and I’m the thickest most (if not all) my partners have had.
There are other means and methods of enlargement that have been refined over time to produce higher efficacy as well, some temporary, some permanent. Of course, like all things in PE (exercises, surgeries, or supplements), nothing is without risk. Both complications and aesthetic irregularities can occur. And it would be ridiculous if I failed to mention that it comes with a price tag that far exceeds organic PE (i.e. the kind of routines you find on this site). These were the asterisks I mentioned in my opening remark.
Injectable phalloplasty, dermal grafts, and the P-Long Protocol are proven girth enhancers (with the first two methods being guaranteed); Ligamentolysis and the P-Long Protocol are also proven lengtheners (although these require PE exercise routines in addition, so a bit more hit-or-miss and not guaranteed if you don’t have the commitment). These are performed by medical professionals and the materials/fillers involved are FDA approved and can be used off-label for things like girth enhancement. Note*- avoid high volume injections of silicone as well as any rigid silicone implant; they tend to possess the most challenges & complications.
Regarding the P-Long Protocol: this is a non-surgical method backed by a study (albeit with a small sample size) that aims to increase length & girth. Sure, it may not be the industry-breaking Study I was hoping for, but certainly a step in the right direction. This involves a prescribed routine (a.k.a. Protocol) tailored by a Urologist (Dr. Brandeis) involving a stretcher & pump (of medical-grade quality, which they supply), a monthly PRP shot, and supplement(s) with a (presumable minimum) 6-Month commitment. The P-Long Protocol suggests 1-inch in length and 0.5" in girth after the completion of the program. Affiliate physicians around the country have began offering it so I cannot speak to its longevity/permanency yet.
To be frank, I wasn’t sure what to think about P-Long but I figured if guys can already achieve size with PE exercises alone, why wouldn’t a tailored routine combined with "medical enhancements" backed by a Study not have just as (or more) merit than standard organic PE? Especially by a Doctor who studies Sexual Medicine. Furthermore, a long-time veteran of my site (who I know well and trust) has begun the Protocol and has already seen gains in length & girth, and he’s not even halfway near completion. So it is exciting to say the least — if it weren’t to be the success it was intended to be, it would still open avenues on how science & medicine can modify and tweak the Protocol to prove more successful for a greater number of men (because what I do notice is how some techniques yield results for some guys while the same technique fails to provide gains for others).
In summary, if it is girth science you want, modern male phalloplasty is a demonstrable success. Some future day this will likely be a stem cell advancement, and even more into the future, bionic dicks!
Thank you for your reply D2G!
Absolutely, penis enlargement procedures are only going to get better. I think there wasn’t too much talk about it until the last 10 years or so. I could be wrong though, but science does progress fast! As for me for me, that type of approach is something that I’ve not thought about, and its definitely not attainable with my financial situation. So for me, plain old PE is what I have to work with.
Besides, PE has actually become a lifestyle now. It passes time and I enjoy the research and application of methods. For the rest of this thread though id prefer to keep this discussion solely based on traditional PE methods.
No penis is different. What works for one, will work for another.
Here is something that just occurred to me related to your clamping theory: in light of your theory that focal compression with clamping results in focal girth increases, wouldn’t you expect focal increase in girth in the region of compression hanger attachment, which is typically in the mid-to-distal shaft?
Lots of guys use pretty high compression forces at the attachment segment during hanging, and there is typically some degree of blood trapping distal to the compression device, like with clamping. In fact, blood accumulating distally in stasis is one of the challenges of compression hanging, since it requires repetitive removal and reattachment, like clamping.
Lots of guys report increasing base girth from hanging, but I don’t know if there is any indication that there are lots of reports of mid-to-distal shaft girth increasing focally at the compression hanging attachment point.
So what do you make of that in light of your theory about base girth and clamping location?
Rock out with your cock out!
Here is something that just occurred to me related to your clamping theory: in light of your theory that focal compression with clamping results in focal girth increases, wouldn’t you expect focal increase in girth in the region of compression hanger attachment, which is typically in the mid-to-distal shaft?Lots of guys use pretty high compression forces at the attachment segment during hanging, and there is typically some degree of blood trapping distal to the compression device, like with clamping. In fact, blood accumulating distally in stasis is one of the challenges of compression hanging, since it requires repetitive removal and reattachment, like clamping.
Lots of guys report increasing base girth from hanging, but I don’t know if there is any indication that there are lots of reports of mid-to-distal shaft girth increasing focally at the compression hanging attachment point.
So what do you make of that in light of your theory about base girth and clamping location?
My shaft is swollen at the point of hanger attachment a few minutes after a set. Looks awesome. Maybe minor future gains from it, i dunno. But im committed to a 3 year timeline. Im really curious how 3 years of pulling and clamping will look.
Goal 7.5 x 6.5
Start 4/22 6 x 5.25 BG
Current 11/22 6 x 5.5 BG 4-7/8" MG
I would like to add some extra information.I’ve never seen many people talking about it before, but length is limited by the urethra, and girth is limited by nerves that are responsible for glans’ sensitivity.
In Brazil, there’s a famous urologist called Paulo Egydio who is recognized worldwide. He actually performs what those who do P.E are after (tunica expansion), but surgically. His surgical method is meant for men who have lost both length and girth due to fibrosis, and as he claims, the expected result is what one had before. Moreover, he claims that the tunica is a very tough tissue, and even surgically, it demands a lot of work to expand it, and methods like stretching and Jelqing will hardly or never make it expand.
Well that’s unfortunate to hear. Id like to point out that he’s either wrong or lying so that he can get more guys to see him. Me and many others have gained. Penis enlargement working is not a question anymore. Maybe there is a limit to how much you can gain which is what he could be saying.
No penis is different. What works for one, will work for another.
Here is something that just occurred to me related to your clamping theory: in light of your theory that focal compression with clamping results in focal girth increases, wouldn’t you expect focal increase in girth in the region of compression hanger attachment, which is typically in the mid-to-distal shaft?Lots of guys use pretty high compression forces at the attachment segment during hanging, and there is typically some degree of blood trapping distal to the compression device, like with clamping. In fact, blood accumulating distally in stasis is one of the challenges of compression hanging, since it requires repetitive removal and reattachment, like clamping.
Lots of guys report increasing base girth from hanging, but I don’t know if there is any indication that there are lots of reports of mid-to-distal shaft girth increasing focally at the compression hanging attachment point.
So what do you make of that in light of your theory about base girth and clamping location?
There are actually a few posts on Reddit r/gettingbigger where individuals have noted girth gains while using a compression device. Some knowledgeable individuals in the comments have pointed out that it could be due to hypoxia and related growth factors.
To answer your question about why there aren’t many reports; compression hanging is significantly less popular than vacuum hanging and extending. Out of the small number of people who do it, in order to hear any feedback from them, firstly, they would have to be active on Thunder’s, secondly, they would have to be consistent enough to actually see results, and thirdly, they might not even be aware of the slight gains they make (if they’re hanging, the chances of them expecting girth gains are small).
No penis is different. What works for one, will work for another.
Id like to let you all know that I am now incorporating these theories into my routine. I will be experimenting with Theory 2 (Clamp Compression Theory)
My current routine consists of 10 mins clamping 1.5" down from the glans. Then clamping at the mid shaft for 10 mins, then base clamping for 10 mins.
I have already done this routine once, but clamped 15 minutes per site and damaged my glans badly (bruised it black and blue, it looks terrible and it hurts).
I am also hanging before each girth session.
To aid me in my girth routines, I have been taking Cialis 5mg daily. I will also be using an 660nm & 850nm NIR heating pad. The 850nm setting on the heating pad has numerous studies showing its ability to increase cell regeneration.
Ask me any questions. And feel free to give me any tips or interesting information. Let the theories begin!
No penis is different. What works for one, will work for another.
I’m surprised there isn’t more interest in this topic? I thought I had raised some very interesting points. I suppose people are set with their current routines and are waiting for someone with a larger reputation to confirm the methods presented here before people commit any time to them. Now that I’ve had this post moved to the topic where majority of the vets frequent, so I’m hoping we get some scientific and experimental brains turning.
I am currently practicing theory 2, but I’m somewhat of a beginner when it comes to clamping, so one clamping session sets back my EQ for a day or 2.
No penis is different. What works for one, will work for another.
Id like to report something very interesting thats not been discussed before. While I’m using my ADS, sometimes i get an erection, but the erections i get while the ADS is on are much thicker than normal. Much thicker than if i were to clamp or pump. Normally im 5" MSEG but when i measured my erection while in the ADS i was 5.5" MSEG. How could this be? Im not clamping, im not pumping, and yet my natural erection was able to thicken by 0.5". Its not a permanent change, when im outside the ADS my erections are just normal.
Could this be a case of erect hanging causing gains? Or stretching while erect actually increase the circumference of the shaft? Maybe one of you should try it intentionally and see if it happens to you.
No penis is different. What works for one, will work for another.