Priapus Shot Received

Marinera,

Again, you are not reading my posts properly. I never said that more SM means less EQ. I think its possible to have more SM and good EQ as everyone who has gained on this site demonstrates.

I never assumed that the penis had more SM content at the base. I was assuming that the act of swinging the weight around would impose trauma on the base not felt by the rest of the dick. This could potentially explain base gains. Trauma has been associated with hypertrophy in other medical contexts.

I never claimed that there were less elastic fibres at the base.

If you believe that its “immediate expansion” then the likely cause is lymph build-up or some kind of swelling.

I am very open minded. If you can prove me wrong using rational argument then I will admit it. You managed to change my mind about the mechanism behind clamping and about the uselessness of T derivatives in PE. Please stop interpreting my arguments as personal attacks.

Since hangers get base girth gains AND lower EQ through intensinve hanging, and since you think girth gains are caused by smooth muscle increase, what you are saying is that more smooth muscle cause bad EQ.

Have a good night.

Guys, let’s be nice. Marinera you are smart as fuck and you KNOW I respect your opinion and knowledge. London, you sound like someone who will find something very very productive in terms of chemical PE, you remind me of another internet friend I have.

I wanted to add in that Marinera is very right about extensive hanging taking EQ down. I think this is usually when one increases sets too fast, or goes up in weight too soon. Otherwise, hanging actually BOOSTS my EQ, beyond me as to why though. And yes hanging made my base girth even more drastic.

Ok. Listen : it was ME saying that there are less elastic fibers at the base. This is actually true. That’s why base is more easily deformed - so you have base girth gains from exercises working on length.

The ‘likely cause’ of immediate expansion can’t be lymph. There isn’t any lymph build up using an extender, or manual stretching, or even hanging - hangers wrap their penis, so no lymph there.

A few hangers swing the weight, where base girth gains are experienced by the vast majority of hangers. No fluid build up either in the area under the clamp. No immediate hypertrophy due to trauma.

Filling your penis with SM won’t increase appreciably girth because the funciont of TA is exactly to constrain CC. TA is way, way stronger than any amount of SM can stretch. Beside that, how fast an increase of SM can happen? Your TA will get stronger faster than that.

I don’t feel personal attacked by you. I don’t dislike you either at all. Actually, what I find annoying is that you are blinding yourself; you could make nice posts, but you are so in love with this idea of SM as The Way to enlarge the penis that your posts are a bunch of contradictions, arguing out of the realm of rationality and hypothesis with no contact with direct experience. So, I think I’m wasting my time trying to explain where you are wrong, because you don’t want to listen - you don’t like the outcome. Probably with enough time you’ll change attitude, but for now you look like a guy who is over 6 feet tall but wants to walk like a dwarf.

That sounds logical and reasonable. And London, you aren’t a dwarf either.

Hey London I like you KEEP up the GOOD work Bro! You tell um bro!

The world does not have to conform to any theory especially black and white either or on off theories. Existence is far more dynamic then that. Analinear logic is the way to go much of the time if not most of it… The world can be seen as flat or round from a certain point of view, but short sided people will argue one way or the other, something of a tail chase in the end really, but oh well such is life… A great many things are probablistic and proportionate, it may work a little it may work alot, which may seem like not at all or way to much, a lot of things are just sort of relative. I dont think its very productive to spend most of your time doubting becase it implies to me a black and white expectation in the out come of most things, when there is now law saying it has to. Who says a white crow wont hatch tomorrow or already hasnt at some point in time? [Or cant be made to be LOL\ BioChemPE anyone???]

There is no logical connection.

I was saying that base girth gains (could) be caused by SM increase.
SM increase is unrelated to EQ
Hangers get bad EQ for other reasons.

BTW ron over at ChemPE is starting to get gains after having none for 2 years via Oxytocin shots, I shared the data with him on how it causes angiogenesis now he feel he may be done with PGE-1 and replace it with Oxytocin! Things that make ya go hmm, someones real a pratical experience or some web aticle about the facts…

“Don’t go trying to confuse everyone with the facts, now!!!”

…Go on…

I know nothing about PRP. This may be a stupid question, but high platelet counts have been implicated in thrombosis. Is there a risk of thrombosis when using PRP?

I would not think so, more likely its due to the result of the injury from thrombosis calling in more platelets so when they collide with the site of injury the growth factors are activated. Which is essentially whats going on with PRP\PRFM. PRP\PRFM simulates an actual injury when injected the PRP is activated and the PRFM forms a fibrin matrix, sort of like a scaffolding. Its along this scaffolding that all the growth factors travel.

Check out the effect of oxytocin where angiogenesis is concerned, also check on the role of each growth factor in PRP\PRFM healing cascade and see how it can impact PE, and or hyperplasia, angiogenesis, and things the break down and build up collagen

PS check the effects of oxytocin where ED studies are concerned. Also check the role it has in the Production of NO [nitric oxide], need good choline levels and something else or its not as effective. Also when PRP is activated it calles in M-Stem cells… These do their work for 3 months… but get seeded for the first 3 weeks…

Pardon me for jumping in here. I would like to add two alternate hypotheses for hangers’ base girth gains:

1) Depending on an individual’s anatomy, the pubic bone can serve as a fulcrum to place quasi-circumferential stresses on the base of the penis, especially when hanging in the BTC position. Anecdotally, as someone who has hung almost exclusively BTC for two years, I can attest to base girth gains precisely along the portion of the shaft that bends over my pubic bone (and perineum, to a lesser degree). I believe Marinera cited fulcrum stretching at midshaft increasing midshaft girth in similar fashion. Bib and others who have used the RSDT fulcrum have indeed claimed improved midshaft girth. This would likely imply that tunica deformation is the driving force of such growth.

2) The ischiocavernosus and bulbospongiosus muscles attach at the base of the penis and are composed of fibers which may hypertrophy under tension (of course these are not the same as the smooth muscle within the corpora). I cannot say exactly how far they extend over the visible portion of the shaft as this may vary based on an individual’s fat pad, exit point, and position of suspensory and fundiform ligaments.

However, I think we should be cautious to dismiss the possibility of smooth muscle involvement (as London has offered) at least as a supporting actor in any gains process. Corporal sinusoids will fill to greater volumes given the tunical freedom to do so, as saphenous vein graft surgery has demonstrated; yet the surgery should also demonstrate that exclusively increasing one component of penile architecture without a concomitant increase in others may result in loss of EQ or potentially even ED. In fact, it doesn’t seem unreasonable to suppose that the extent to which EQ is lost due to hanging is a dynamic, ever-changing balance between reduced contractility of the stressed tunical fibers and changing ratios of smooth muscle under new penile volumes.

Oxytocin is a legal hormone right? If so, where do you get it?