Increasing smooth muscle mass in the penis

There is a study around which has been quoted by others which has shown on cadaveric studies that young men have a little over 50 % smooth muscle mass n the penis and as we grow old our smooth muscle mass goes down to 30 %. Related to the loss of smooth muscle is a loss of erectile strength. It has been suggested that a sufficient amount of smooth mucle mass is essential for optimal erectile quality.

The problem is that we have little knowledge about how to increase smooth muscle mass. While it is easy to exercise skeletal muscle mass (bulbospongiosus and ischiocavernosus muscle by kegels) there is a hypothesis in the scientific world that the body practices smooth muscle during spontaneous nocturnal erections. They occur at the end of the REM sleep phase, approximately per night, the first 20 seconds after the first REM phase, the last 90 seconds after the last REM phase.

Aging man not only loose muscle mass and erectile strength but also experience less and less intense nucturnal erections.

One rather new approach to treat erectile dysfunction was to administer viagra every evening before going to sleep during one year. Erectile quality improved during that year, and persisted several months after discontinuation of viagra.

It was postulated that this was a result of increased smooth muscle mass.

This approach could be useful for those pe’ers who believe that an increase in smooth muscle mass is the basis, or essential for penis growth.

Testostrerone replacement in testosterone-defficient men also improves erection quality and the frequency of nocturnal erections.

That’s some pretty interesting stuff, TTT.

Thinking about these mechanisms ballooning or edging makes more sense to me than previously: basically it would be a prolonged erection, so to say a smooth muscle exercise session. Jelqing at a low erection level would make less sense than at a higher erection level.

It has been my hypothesis for a while now that good, hard and frequent nite wood may be part of the cementing process, and a possible mechanism of gains itself.

What I have found is if you dial in you PE properly AND strengthen your muscles with kegels, that most guys will see greatly improved nite wood.

As per Stagestops Chemical PE thread, he has found that if you increase smooth muscle mass, it will expand the tunica.

This is really interesting in light of what I’ve suspected. Good information!

Wouldn’t it be funny if some of the main function of PE is to greatly increase your nocturnal erections, and THAT then is one of the main mechanism of gains?

It is my understanding that the smooth muscle in the penis works to control the flow of blood through the arteries. When the smooth muscle relaxes, it allows the flow of blood to enter the penis and results in an erection. The following is from a previous post I made on this:

Smooth Muscle In Arteries
How the smooth muscle in arteries works:

http://health.h … com/viagra3.htm

QUOTE:

The penis is one of the places in the body where the brain needs to be able to turn the blood flow on and off with a valve. To understand how the brain controls this particular valve, let’s start with the basic concept at work: How does the brain control blood flow to different parts of the body?

Turning Valves On and Off
In the human body, the "valves" open and close using muscles in the walls of arteries. When these muscles relax, the arteries open up and blood flow increases. The valves respond to chemical messages that the brain can control.

The mechanism that the body uses to "open a valve" in any part of the body involves four steps:

The brain sends a signal down a particular nerve fiber. This nerve fiber ends in an NANC nerve cell in an artery, somewhere near the point where blood flow needs to change. NANC stands for nonadrenergic-noncholinergic, and what it means is that the NANC nerve cell is able to create nitric oxide.
The NANC nerve endings inject nitric oxide into the blood and surrounding cells.
The nitric oxide stimulates an enzyme called guanylate cyclase in nearby cells, and this enzyme starts producing a chemical called cyclic guanosine monophosphate (cGMP).
cGMP tells smooth muscles that line an artery to relax. When they relax, blood flow increases.

So how does this relate to an erection?

When the brain gets aroused, it sends a signal to the penis. Nerve cells in the penis’ corpora cavernosa start producing nitric oxide, which leads to the creation of cGMP. The cGMP causes arteries in the corpora cavernosa to dilate, causing lots of blood to flow into the penis. The extra blood flowing in causes the penis to inflate like a balloon. An erection occurs.

UNQUOTE

So you can see, there is no benefit involved in trying to strengthen the smooth muscle tissue in your arteries and in fact it can’t be done. The erection mechanism is triggered when these muscles relax, not when they flex.

Sorry to be a dumb head about this, but it’s all very well to let extra blood into the arteries of the penis, but surely the outlet through the veins needs to be constricted, otherwise there would be no pressure. Do both of these things occur during an erection?

Mine, too, sparky.

gprent said: “Turning Valves On and Off
In the human body, the “valves” open and close using muscles in the walls of arteries. When these muscles relax, the arteries open up and blood flow increases. The valves respond to chemical messages that the brain can control.”

But, if you can bypass the the off/on system controlled by the sympathetic nervous system (Sympathetic discourages erections) with a moderate dose of an ED drug like Viagra or Levitra at bedtime, frequency and duration of nocturnal erections greatly increases. At least for me. And, I agree not only with the theory the “cementing” becomes more effective, but that general penile vascular health improves during these nocturnal “exercise” times.

I’m at an age when much of this nocturnal activity should have leveled off years ago. But I’m still getting many erections during the sleep cycle. I see these as very good events.

During a successful erection (eg., firm) as the penis lengthens and widens, there is increasing pressure against the internal veins which effectively constricts them and seals them off. There is always some flow in and flow out, thus assuring that we’re getting newly-oxygenated blood in and venous flow out back to the heart and lungs so that we don’t have tissue damage from oxygen starvation during a prolonged erection. I’d guess that for most of us, anything over a half hour could fall into the “prolonged” arena.

That isn’t how I’ve read that thread (Chemical PE progress report), SparkyX.

Stagestop is injecting inside the corpus spongiosum . He has commented on the ‘stuffed sausage" sensation he has gotten after injections (especially when he injected too much). I think that Stagestop’s hypothesis is that he is getting growth from the inside out, as the corpus spongiosum grow and put stress on the tunica and the smooth muscles to expand. I don’t think Stagestop has been going on the hypothesis that he is primarily trying to increase smooth muscle mass, or that he is seeking to increase smooth muscle mass to expand the tunica.

Hey Lamp!

I am refering to girth increases, and I am fairly confident he injects the CC and them main mechanism is increased smooth muscle.

post 413
“…We grow the CC, it puts constant tension on the skin and tunica, and gradually, it yields to that force. The sausage effect is when you get too much growth in the CC and the tunica has not had time to stretch enough to accommodate that growth…

In the penis, since stretching has already shown itself to be a cause of growth, but starting our program with that, we only accelerate things by adding IGF-1. This is for the girth side of things naturally, as IGF-1 does not seem to do all that much length wise.”

post 393

“With there being separate, or perhaps it is better to say strings of smooth muscle, coupled with the total amount of smooth muscle in the penis, there seems to be a heck of a lot that IGF-1 can attach to when it is in the penis. I feel that this is the explanation for the growth in girth that I have experienced.”

Interesting I have another hypothesis though that can be added in. It is not designed to subtract from this theory.
We speak of nocturnal erections. Some guys like me don’t get nocturnal erections. However I have gotten more nocturnal and spontaneous erections this year than in the first two years of PE combined. In othe rwords they are becoming more frequent.

I say create your own spontaneous erections, not nocturnal but planned. When ballooning or edging let you erection subside almost completely. I do this almost every session I let it die down to near nothing. Then build it back up again through the process of ballooning. I think these session have actually may have increased smooth muscle growth.

Of course you have to factor in the use of Ginkgo Biloba, Ginseng, Green Tea and AAKG (I’m not currently taking AAKG, will will resume soon)

If we can crack the code on increasing smooth muscle, how much of a role will this play on reversing other vascular conditions?

Post 393: stagestop - Chemical PE progress report
Post 413: stagestop - Chemical PE progress report

You are completely right about Stagestop injecting into the CC corpus cavernosum , not the CS. The first quote you cite suggest what I was getting at: the CC grows, and puts tension on the tunica and the smooth muscle. Good citation of post 393 about IGF-1 attaching to the smooth muscle as well (not that I’ll be sticking needles in my penis right away, in any case).

My understanding is that the IGF-1, when injected into the CC, primarily causes the smooth muscle to grow, which leads to the “sausage” effect. This is because the increase of smooth muscle, which creates some increased internal pressure, which over time expands the tunica.

If that is indeed correctly summerizing the main effect of chemical girth gains, then it sheds a new and revolutionary light on PE and what we may be doing.

It may very well be that ONE of the mechanisms of girth gains is not directly stretching the tunica, rather stimulating smooth muscle growth WHICH IN TURN causes tunica expansion, at least in width.

To me, this explains why some of our Vet pumpers, with moderate vacuum levels over time have really had great success with girth.

It also brings to mind that some of the clampers that have really increased girth talk about a slow steady progress, that it takes time.

Further, it seems IIRC, most of these guys also report very hard, frequent nite wood.

If this turns out to be a major mechanism, it certainly will allow us to shift away from trying to directly stretch the tunica and re-examine some of the hypothesis of what stimulates smooth muscle growth, like increased blood pressure’s effect on arterial smooth muscle thickening and mild hypoxia.

Which translates into moderate vacuum pumping and clamping for periods of time that not only increase pressure but cause some mild hypoxia.

[I don’t remember where I saw the references for the hypertension being a mechanism of smooth muscle growth or hypoxia causing smooth muscle growth, TTT maybe you are familiar with them?]

I for one am very excited about the implications.

spark, I posted those references in the TGC theory thread, here is the link:

Iguana - Focus Your PE and Gain! TGC Theory