Could very well be. The article shows that mechanical loading has complex effects both on muscle extracellular matrix and smooth muscle. There are several parts who are interresting; for example:
"Static stretch has been reported to increase SMC synthesis of tropoelastin, the precursor of elastin [78] . Three studies in particular have directly compared the effects of static and cyclic strains [11,79,80] . In the first study, cyclic strain (10%, 0.86 Hz), compared to 10% static strain, caused a 3-fold increase in total protein and collagen synthesis [79] .
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All three of these studies clearly demonstrated that static strains promote ECM degradation and adhesion molecules whereas cyclic strains enhance SMC synthesis of fibrillar ECM proteins."
As I read it, sounds like continous static stretching works remodelling tissues, whereas cyclic stretching works making them grow.
"….cyclic strains applied to cardiac fibroblasts have been found to modulate collagen synthesis and to cause the secretion of various growth factors into the ECM. When these cells were subjected to cyclic loading, their collagen I gene expression increased up to 4-fold compared to no loading conditions [37–40] ."
This accords with what posted elsewhere; anyway, as the author underlines, cardiac tissues are highly specific because they, by their very same function, are subject to cyclic stress, differently than tissues in the penis.