How could the tunica weaken after continious(increasing or stable) pressure from inside?
Quote:
"We propose an alternative mechanism for this sequela. The increase in penile circumference follows a sudden and permanent loss of elasticity of the tunica albuginea, brought about by a particularly intense priapism and a very engorged organ. The loss of elasticity of the tunica albuginea releases constraints on the corpora cavernosa, which then expand like a sponge. This expansion, helped by some subtunical venous impairment that is secondary to the remaining elasticity of the stretched tunica, results in pooled, deoxygenated blood in the corpora cavernosa consistent with our BOLD-MRI findings. BOLD-MRI defines the presence of deoxygenated blood, eliminating the need for an invasive procedure."
Megalophallus as a sequela of priapism in sickle cell anemia: use of blood oxygen level-dependent magnetic resonance imaging
No deoxinated blood in pumping I suppose.
The sudden loss shows it can happen probably sometime when the blood is deoxygenated to a certain level.
As with a "non ischemic" it probably still isnt totally deoxygenated as fresh oxygen follows which then decreases the chances of a defunct penis.
Of course experementing with this is highly dangerous and can result in a defunct Penis.
But in its mildest form resembles many intermediatery PE exercises performed by conditioned exerciseerss.
There are some examples of people pushing their limit regarding oxygenation that had some good gains and documentation. Like Redzulu. Or big girtha. They for sure created internal pressures for a long time. They all had a long conditioning before venturing into such extremes.
I would suggest for anyone getting out of control priapisms to immediately visit a doc to relax the situation. The chances of a defunct penis are probably too high to not get to the doc..
There might be the possibility to get control over it with training the pelvic floor if the high in flow isnt created/originated by lets say the brain or spinal cord…
hope someone chimes in and this has some truth in it ..
Is the inflow continiously increasing the pressure and size(and maybe correlated pain?) of the organ? Or is it at some point stable? Polymer ,can you recount that?