Gaining volume with Kyrpa

“Growing a few cm3 of new tissue a year for such a big capillary bed can’t possibly be as fast as the process can go.”

Yeah well even if this were so, who is to say that a counter - say protective or otherwise limiting measure wouldn’t be triggered?

Do you have any references for the idea that elastin reduction results in a less firm erection. I’m not suggesting that I have anything arguing the other side, but it seems highly counterintuitive. The penis essentially being a highly elastic volume restricted by a more inelastic liner. The elastin would provide the ability to enlarge, but the collagen would provide the firmness.

I suppose he is pointing on studies like this one. Am I right Sigmoid?

https://www.ncb i.nlm.nih.gov/p … ihms-503716.pdf

There are actually drugs to increase elastin content, but whether or not that new elastin will be organized as it needs to be isn’t clear and the existing drugs I know about come with serious side effects from long term use.

I think the elastin plays more of a role than just facilitating the contraction of capillaries via SMCs, allowing them to not be collagenized which would limit the ability to increase the blood flow when needed for erection. I think elastin plays a huge role in the inner fascia. The inner fascia needs more elastic qualities so it can expand easily from the initial increase in blood flow to the CC. If the inner fascia can’t expand more easily to compress the veins against the outer fascia, full erection isn’t possible without something like a cock ring. If the inner fascia of the CC loses its elasticity I think it will shrink through remodeling without using something like a cock ring or pump, followed by the shrinking of the outer fascia.

Eventually someone will find a species with a similar penis and run tests. Or maybe the answers will come from data aggregation about who bought penis pumps, traction devices, etc. and examinations of their cadavers. We could probably gain a fair amount of info from high resolution ultrasound imaging.

The human penis has two relevant fascia for erection. The inner one has a grain of fibers running girth-wise around, it restrict girth expansion. The outer one has fibers running shaft-wise, it restricts length expansion. If the inner girth fascia cannot expand enough to compress the veins running between the two fascia, you cannot have a full erection since the restriction of blood flow out of the penis is primarily due to the mechanical pressure of being compressed between these two sheaths. If the inner fascia isn’t elastic enough to expand from increased blood flow, it can’t compress the veins to reach maximum cavernosal pressure.

Comparative analysis of the penis corpora cavernosa in controls and patients with erectile dysfunction - PubMed

Computerized measurement of penile elastic fibres in potent and impotent men - PubMed

Characterization of Erectile Function in Elastin Haploinsufficicent Mice - PMC

"The flow rate required to maintain a set ICP (200 mm Hg) after the use of a smooth muscle relaxant, papaverine, was compared. Eln+/− mice required a higher flow rate to maintain 200 mm Hg ICP as compared with Wt mice."

The above quote is important to notice, but could be easily missed. The mice with a missing elastin gene were not impaired by SMC dilation, but the amount of blood flow necessary to reach a certain cavernosal pressure (ICP) was greater because of a weakened elastic qualities of the penis fascia.

Forgive me if there’s some mistakes in what I’m writing today, I didn’t drink caffeine yesterday and still have a terrible headache.

Yes. Though when I first read up on the subject a while ago I don’t think I’d seen that study. I seem to remember a study on rabbits and a few on humans. So there are definitely more backing up this idea.

Like I previously mentioned, we are guessing just like them. They do not know if the low elastin content on tunica or the missing elastin in cavernous tissues is the main reason.
Your view on this is that the inner layer with poor elastin content is the strongest suspect objecting the expansion and not compressing veins.. My guess is that the cavernous tissue with low elastin content has poor smooth muscle cell relaxation capacity which leads to need for higher in flow rate to build enough pressure to push the inner tunica fascias compressing the veins against the Buck´s fascia.

The studies available do leave the interpretation completely to readers. There is nothing conclusive yet.
What I do know is that the 18 months on the program has improved my erectile features in every aspect at high rate. I am willing to bet that I have increased the collagen content in both tunica layers. Not in cavernous tissues in same percentage if at all. We just don´t know without histologial examinations.

If someone is thinking what we are talking about in the first place , here is the physiology of the erection.
We are talking about the phase blood flows in . If the infation is poor there is no chance for the peak phase of the erection where there pressure builds up by the compressed veins cutting the outflow.
https://www.ncb i.nlm.nih.gov/p … les/PMC1351051/

Sigmoid , may I ask if you are worried about PE methods driving us to state addressed in this posts?
Remodelling the tunica too dense with increased collagen content or the SMC mass surrounding sinusoids getting too massive to relax enough?

Kyrpa, got a question for you. I’m not sure, could be my imagination, but towards the end of the US session today it felt like the cord on my shaft (dorsal part, related to septum I think) was softer and possibly thinner. As in, there is a thicker hard part at the start, and it seems to reduce nearer the end…. something softening up?

I have observed similar things. It seems you have effectively heated the thing.
It feels thinner when palpated because the whole penis gets very soft during proper heating like the cord itself.
I have used manual stretching at the end of the heating and the first pulls feels in the hands like stretching rubber band. It normalizes after few initial strokes as the unit cools down couple of degrees quickly but I was amazed when this happened first time.

OK good to know your experiences, thanks

I’m not so worried about it since I suspect if elastin is spread too thin we can always just allow the penis to shrink back in size some and the elastin fibers may be reconnected enzymatically, restoring erection strength.

But I am curious about what role elastin plays and, if this is a problem for establishing larger sizes that are sustainable, how more elastin could be added to the areas it is needed. That would be interesting if just adding more elastin to the mix alone would gradually allow the penis to remodel to be bigger when erect. Perhaps if elastin is doing the job of the connective tissue effectively, the tissue removes unnecessary collagen.

So I’ve been thinking about the heated stretch time under US… was there a rationale to why you ended up pulling with 3-4kg under US rather than more? Was it due to the limit of the vac head device, or due to something else? I’m thinking that since we pull with much more force at the end manually but without US as it’s cooling down (I think you were measuring 9kg or so), why would we not do the same during the US stretch time? i.e. pull with 9kg. With a BiB it would be possible. If the reason is something to do with the effectiveness of cyclical stretching with more force, there could also be a way to cycle tension up and down from say 3 to 9kg throughout the US heating part?

Well, I have been promoting the idea of everyone taking the load - strain relation out of their units. We can´t just pick any load we find in someone else log.
I have made the test now several times to determine the loads which I need for every step to maximize the strain.

I can get a certain level with lower weight in given time. Given time has here the importance of glans bearing the vacuum cup about 60 to 70 minutes maximum.
Given time is such an important variable when talking about visco-elastic tissues in general, it has to be always taken in account when talking about the load.

So I can get the maximal elongation at phase 1 , stress relaxation stretch with 2kg - 2.7kg kg in 30-40 minutes. And recently I found out that I can get the elongation at this phase whether or not I am using rice sock heating (graph SS_combined).

Phase 2 is heated with ultrasound. Earlier test performed last year I found out that I needed slightly more load to max out the US heated stretch in the given 20 minutes window. ( Strain 3).
I chose the 3 - 3.8 kg because that was enough to give the elongation and I struggled to stand the stress build up during 4kg loading.
Lately I have been forced to increase the loading a bit during the heated stretch compared to situation year ago .

Phase 3 cooldown stretch is performed by manual stretching. And the load is average as the load naturally varies while doing the stretch manually.
Looking the stress strain 3 graph you can see that there in no reason to pull with the 9kg load, as there is no additional strain achieved.
Therefor I have stuck with the approximately 6kg loading with the exercise.

I can´t bear such a loads in constant stretch. It would be painful and the glans would be in blisters.

With the manual stretch I can do the necessary cooldown stretch with the load enough, glans doing fine with the manual grip.
If we do the cooldown in the fixed length extender the stress builds up to the level of loads mentioned due the internal forces in the collagenous tissue.
While cooling down from + 4% strain the structure, most likely the ECM contracts with a such a powerful magnitude that it is hard to manage without slippage in any vacuum or noose style extender. If we were to put load cell measuring the load during the cooldown we would see loads similar I use in manual stretching.
I know that there is lots to read but I think I have been talking about these things few times already in this log.

Taking the time for examining the load bearing and elastic capabilities of each one´s own unit, there should not be too low or too high loads anymore.
Just optimized and effective penis elongation.

Without converting the load for stress units we can´t straight out compare the load some other is using. The load in grams divided by the thinnest cross sectional area of the penis during the stretch indicates the stress level in some of these graphs included.

GRAPHS:

Stretch, is a showcase of the three phases of the workout session , and the illustration at which region of the stress strain slope we are operating.

Strain 3 , shows three different workout sessions with datapoints merged in one graph.

SS_Combined , shows two different exercise merged in the same graph . One performed cold and the other with heating. Load has been converted to stress unit of g/cm^2 .

Stress_Strain united data , shows the comparison of data from cold stretching sessions of Alex2.0 and me merged in together. Loads have been converted into stress units for the leveled comparison.

Additional answer.

Yes with the Bib it should be possible to use such a loads.
I would still be very cautious introducing such a high loads during the heat ing with US.

There is thermal transitioning happening at the temperatures we are aiming. Structural weakening allowing the elongation with lower weights which other wise would need multiple times higher loads and structural damages which we are not having.

For the cooldown the Bib with the fixed length extender would be perfect.
Fixed meaning not loading with the springs put setting up the maximal length with initial load (~5-6kg) and leave the penis cooldown. Pure stress relaxation setup.