Originally Posted by Monty:
So it appears that the question here now becomes: If the layers are splayed which would account for expanded girth measurements during a clamping session, do they fill in with new fibers in between the originals or do they fatten which would also result in increased girth?
Because of the perpendicular arrangement of the fibers between the circumferential inner layer and the longitudinal outer layer of the tunica, they appear to work at cross purposes. Clamping at peak erection will place a very high tension on the tunica throughout its entire length - mainly circumferential, but also longitudinal to a lesser degree. Splay does not account for the expanse of girth measurement during clamping. The predominant force in the tunica will be on the circumferential layer placing a tension along the long axis of the circumferential arranged collagen fibers. With this force, the expansion of girth puts stress on the fibers of the longitudinal layer forcing (or splaying) them apart to some extent, the amount of splay will be limited by the ability to stretch the cross links.
The rate limiting step to girth enhancement will be in the element with the greatest resistance to tension-induced stretch; this would be the collagen fibers in the circumferential layer, not the cross links in the longitudinal layer. So if clamping is to work, it must induce collagen fiber elongation in the circumferential layer to increase girth. I don’t think that you can say that girth expansion is solely the result of splay.
When tension is applied to tendons or ligaments (depending upon force and duration) there can be either collagen fiber lengthening (increasing flexibility) or fiber thickening (to resist tendon rupture). Since the stress induced by the splaying of the fibers of the longitudinal layer as a result of cavernosa expansion is not along its long axis, it should not affect either elongation or hypertrophy of fibers in this layer. The stress will be on the cross links, will they decrease or increase over time as a result? Good question.
There is no real space to fill in and the character of the tension stress should not result in “…new fibers in between the originals…”
Originally Posted by Monty:
I’m not sure that length is enhanced all that much because the ligs would still be the limiting factor overall. What is left to be determined is the recovery rate of the lattice to it’s original state and how often expansion needs to take place for growth to be accomplished.
I have to disagree with you here. I believe that penis enhancement is multifactorial, both in length and girth enhancement. There is substantial evidence and experience in this forum that tunica stretching maneuvers (v- and inverted v-stretch, etc.) result in lengthening independent of ligament stretch.
I am also not sure what you mean by a lattice and where this lattice resides.
Originally Posted by Monty:
It seems, for the sake of argument, that these layers would need expansion very often and on a regular basis. How fast they could restore themselves is what seems to be key here to change.What is fascinating to contemplate here is if one guy has three layers, and another has one layer, does this explain why some guys can experience ballooning where another cannot?
The layer question (remembering that guys with 2 layers are far, far more common than are either 1 or 3) is a very interesting one and has been since it was first presented in this forum (and it has been presented several times in several different threads). We can but theorize. Makes for interesting discussion.
The question of how often and by what force one would have to expand these tunica layers is a good one. High stress on tendons or ligaments results in thickening. Our goal is not to thicken collagen fibers as this would inhibit any lengthening of the collagen fibers (either longitudinal or circumferential).