Girth theory: Pumping vs. clamping

That’s great info pud, thanks.
When I compared to a wetsuit, I wasn’t referring to the actual structure, just the idea of how it can seem really tight, for example around your arm, but if you flex, it still stretches.
What I am wondering about is how the drawing you posted translates into 3 dimensions. In other words, how pulling longitudinally effects the fiber orientation in the radial direction, or visa versa.
Also, I wonder how that structure is modified from PE induced growth. I have heard the theory in the past, that breaking the elastin fibers causes a gain in flaccid size by allowing the unfolding, but what exactly needs to happen to cause erect gains?

Well this is very confusing with positive and negative arguments and a allot of great information to absorb. I want to buy a pump one minute while reading and then I think that clamping is the answer. Can we do a pole on what worked for the brothers on thunders. I don’t know how to establish a pole on this site. A pole may aid in a members decision to pump or clamp.

example:

Did pumping work for you in girth gains? yes/no
Did clamping work for you in girth gains? yes/no

This will allow us to get stats on both.

When the pressures exceeded 750 mm Hg, the undulation orientation of the collagen and the elastic fibers is destroyed and does return to the rest orientation position. (See the bottom panel of the attached file, the broken elastic fibers stick out like hair).

I do disagree regarding the 102% erection and do agree with ttt that it can be achieved. If you measure your penis at peak erection several times without penis rings and get a consistent average, this average will be your 100% value. Any increase above this is going to be greater than 100%.

There is a difference. When you stress your hamstring muscles (and specifically the tendons and sheaths) to touch your toes, you are stretching against an unloaded muscle. The tunica at peak erection is loaded. The difference is critical. The tendons elongate (as I have mentioned before) from connective tissue remodeling at a longer fiber length. The tension added to the tunica at peak erection leads to tension-induced connective tissue remodeling at a larger fiber diameter not length (stronger not longer).

Are you saying the tunica is not pliable? Or not pliable under maximum stress? I guess I cannot under stand how it can be disputed? I have clamped to over an inch or more of solid base girth with absolutely no fluid.

Well this is very confusing with positive and negative arguments and a allot of great information to absorb. I want to buy a pump one minute while reading and then I think that clamping is the answer. Can we do a pole on what worked for the brothers on thunders. I don’t know how to establish a pole on this site. A pole may aid in a members decision to pump or clamp.

Example:

Did pumping work for you in girth gains? Yes/no
Did clamping work for you in girth gains? Yes/no

This will allow us to get stats on both.

And you seem to misunderstand the admonition to avoid turtling. That has nothing to do with your mythical “102% erections.” It’s simply the concept of trying to maintain semi-inflation (“plumpness”) for as long as possible post-workout. And, guess what.that’s in a non-fully-erect penis (with, therefore, a more compliant tunica).at the risk of drawer further sarcasm from you, YES, I also wrote about that before: mild to moderate, long-term stresses being uniformly maintained against the tunica in a semi-erect state slowly deforming the tunica - or causing “creep,” whichever word you prefer. :)

I am with you, Steady.

Pudendum is an academic mind (smart ass, as ThunderSS noticed), but without experience in neither pumping nor clamping if I remember correctly.

Pe is pretty much an empirical “science”. We are trying to work on the theoretical background in this thread, but must keep in mind that the theories should not contradict our experience.

Nope. I’d rather you found a College of Bullshit at this particular point in time ttt. I seem to be running into a lot of that lately. Nothing to do with you or this thread though. But I could be the Dean of Bullshit with no problem. :)

I hope you don’t mind, but I’m going to correct something you said. As I said in an earlier post, lymph is the fluid in lymph vessels that drain to lymph nodes and ultimately back into the blood circulation. Fluid outside cells is called extracellular fluid (ECF). When ECF increases and causes swelling (aka, fluid build up) it is called edema. This edema fluid will overtime enter the lymph channels and “become” lymph. It may sounds like a case of semantics, but this is the more correct terminology. It’s kinda like capitalizing Kegel, it’s just what it’s called.

In a couple of posts ttt and I talked about the factors that cause fluid to leave the blood vessels (at the capillaries) and become ECF (and if too much, edema). It is a balance between 2 factors. One is the pressure difference between inside of the capillaries (which is 30 - 50 mm Hg) and outside the capillaries (which can be between -3 to 5 mm Hg). This difference would cause the fluid in the blood to exit the vessels and become ECF. The other factor is caused by the differences between the amount of proteins inside (high) and outside (low) the capillaries. Blood proteins do not pass out of the capillary. This difference in proteins tends to draw water back into the blood vessels by osmosis and “holds on” to it; water travels back into the capillary because amount of water is higher outside the vessel and lower inside the blood because of the proteins. (Mravg - this is a difference between hydrostatic pressure and oncotic pressure).

The balance between these 2 factors in normal situations keeps fluid inside the blood vessels. There is a small leak normally and this is why there is a lymphatic system in the first place; take ECF back to the blood and keep the space outside of the cells relatively “dry.”

Pumping and clamping alter this balance.

Pumping causes the pressure outside the capillaries to become very negative. This causes the pressure difference between inside and outside to become even larger. This causes fluid to exit and increase ECF. As pumping is sustained, the negative pressure difference remains high and over time fluid builds up (edema).

Clamping increases the pressure in both the arteries and veins so that the pressure difference across the capillaries gets higher and fluid would leave into the the ECF. The difference between clamping and pumping is that the clamps keep the ECF from moving around and leave the penis. Because of this, pressure outside the capillaries rises quickly, balancing the pressure difference and the fluid loss will be small. The other thing is that clamp times are short, so the time for fluid loss is also short.

I think this is the reason for the differences in fluid build up (edema) between pumping and clamping. I hope this is clear.