Originally Posted by SteadyGains
So using the term, compliance. I believe that stress on the tunica over time increases the compliance at high pressures
I think a better way to say it is that our goal over the long term is to reach the point of lowest tunica compliance (stiffest, less pliable) at an ever longer stretch length both longitudinal (length) and circumferential (girth). That is the crux of PE directed at the tunica.
Originally Posted by SteadyGains
This can disproved by demonstration very easily. Even if you think a portion of the increase is fluid, the increase is substantial, not “very very small”.
I’m saying tunica radius at peak erection, not overall penis. This is our goal anyway. The edema will be removed quickly.
In LaPlace”s law, one of the factors is wall thickness (in this case the tunica thickness). Tension is inversely related to wall thickness, that is when the tunica thins with stretch tension increases. This thinning at peak erection is small and I will grant you that with this small thinning and a small increase in radius that the tension will rise. Does it cause tension-induced connective tissue remodeling at a longer collagen fiber length and/or diameter? How much? You’ll have to answer that. The degree of lengthening over strengthening may be the factor of variability that everyone is presenting
I believe you can obtain greater tensions if you start to pump at lower levels of erection and bring the penis up to an “artificially” high radius. What I am trying to say that by causing a pressure gradient across the tunica with negative pressure, you will be drawing more blood into the penis and increasing the radius. As the tunica starts to become stiff (because of compliance) and the external pressure effects becomes less, the pressure in the hardening penis will rise as well at a higher radius than if you had started to pump at peak erection. This is the concept I’m trying to get across.
Am I making sense, or am I just full of it?