The Penis (smooth) Muscle Theory

This thread is a continuation of a discussion begun in the thread entitled, Need help from all good gainers!.

I have always been curious how the penis works — and if I could enlarge it. Along time ago, I was rather upset when I found out that the penis wasn’t a muscle, and therefore it couldn’t be enlarged. Thankfully, since then (due to this site and others), I have found out the truth — penis enlargement is very real. It works, yet, no one seems to know exactly how.

I was rather dumbfounded when I learned that the penis truly is a muscle — not completely muscle, and not a normal muscle — but approximatelly 50 percent smooth muscle. Below are two posts of my notes and research about the smooth muscle of the penis.

Xeno, there is plenty of basis in this forum. I hope I haven’t offended anyone by for-bringing what I recommend to the table. After all, sparkyx did ask for my opinion. Perhaps you meant no harm in your reply, just busting my balls, eh? Either way, in actuality I’m truly not a scientist, yet.

Science is mysterious itself. Even scientific facts aren’t always fact. So I presume that my recommendations could be wrong, but I believe in them. But I will always follow the evidence, at least to the best of my ability. My recommendations are also based on hours (and hours, and hours) of reading over PE forums, and scribbling down my own notes.

But more important (at least right now), do you disagree with me? I know you think decon breaks help, and I also know that you believe moving up in intensity is the best way to go. Or, at least that is what I am under the impression (from your posts and your journal). I’m all for discussion if you think I’m wrong. I think that is why Sparky created this thread. He, the thought provoker he is, wants to create more guidelines for men to follow.

Anyways, here is a reference for what you requested:

If you want the full article, email me (akemmer@gmail.com).

Personally (this is my opinion), I think the smooth muscle plays a monumental roll in penis enlargement. Healthy smooth muscle in the penis is required for an erection.

This is well documented in:

I only have this one in print, so I can’t email it to you. However, if you would like to look at this 20-something page article (an exercise in itself to read ;) ), then I would be willing to mail it to you.

But here is a tidbit:

In an essence, this article went over (in detail, down to the chemistry) the smooth muscle’s role in the penis. The bottom line: smooth muscle is very, very important. Or, at least that is what I perceived it to be. The smooth muscle causes an erection (which is set off through chemical reactions) by completely relaxing. "Compression of the relaxed trabecular smooth muscle against the fibroelastic tunica albuginea causes the closure of the emissary veins," and thus results in an erection. If this smooth muscle is unable to completely relax, then an erection can’t take place… . .

Here are my current thoughts on the subject of smooth muscle:

• For the penis to enlarge, the smooth muscle (cells and sinusoid spaces) must enlarge too. This is how I think penis enlargement takes place. I don’t have the time to go into complete detail right now, and I won’t for the next 1-3 months (another reason I gave my quick opinion in this thread), but I do plan on doing more research and writing a few "Internet" articles on the subject.

• Because the penis is compromised of 50 percent smooth muscle, and smooth muscle has a lot of the basic properties of skeletal muscle (the biceps, triceps, and all the other commonly known muscles), we can presume that it might react to stress the same way normal muscle does. Which is a no brainer for us all! What are we doing here? Exercising! Moreover, nearly every single guideline we have is based off body-building/exercising/weightlifting concepts in one way or another! Think about it — Bib, presumably one of the biggest gainers (if we accept his claims as facts), used a weightlifting concept known as "progressive overload." Peter Dick, another big gainer uses a common weightlifting program known as "muscle confusion" (in which he keeps the muscles guessing). And now, more recently (thanks to guys like you [xeno], Shiver, MX, Modesto, Wadzilla, and many more) we are now realizing cyclic training using deconditioning breaks helps us keep the penis in a responsive state. And cycylic training is a very popular weightlifting principle (if not the biggest).

• Albeit, this doesn’t particularly mean we can attribute the growth to smooth muscle growth — but regardless, if the penis grows, the smooth muscle must grow too. The article I quoted above stated: less smooth muscle = higher chance of ED. Obviously this isn’t the case for us PEers. Most of us report harder erections.

• Also, It is well documented that smooth muscle can grow due to stress. I have a whole book on the subject, in fact. It is called [u]

  Hypertrophic Response in Smooth Muscle[u], by Charles L. Seidel and Normal W. Weisbrodt. It appears this book has numerous scholarly articles on the growth of smooth muscle. Link: http://www.amaz on.com/gp/produ … 5Fencoding=UTF8

• I also think this can explain why PE gains are permanent (for the most part), opposed to traditional muscular growth gains. Think about it like this: the smooth muscle in the penis is exercised when we jelq, stretch, and incorporate other exercises. These exercises take the penis (and the smooth muscle) beyond it’s normal threshold. But what is the normal threshold? An erection. Masturbation, sex, anything that involves an erection is exercise too! It’s typically just not enough to cause growth. Therefore, after the gains are cemented, and we retire all we need to do to keep the gains is have frequent erections — in which the erections are enough exercise to keep the gains. It is also well documented that as men get older, and they use their penis less, the smaller it becomes. Note: I don't have a reference for this off-hand.

Seems you may be more of a scientist than you think :) . Thanks for the thoughtful reply. My first comment was referring to your recommended training schedule. Is there some basis for its configuration? Or is it just what you FEEL is right? IOW, is there any evidence that it is an effective schedule?

With regard to my second comment, the request for a reference, thank you, very well done. I’m delighted to have learned something from your reply. I think you would do the Forum a service, not to mention assisting your credibility, certainly, at the least, in my eyes, if you’d support your statements of fact with supporting evidence from the get go. That’s what real scientists do.

BTW, as far as I’m concerned, I have “figured it all out”. And I’ve shared it here.

Nice work, Remek. I have been under the impression that smooth muscle does not respond to exercise the same way skeletal muscle does. I’d be very interested in learning more about this.

Holy shit! This site says smooth muscle, unlike skeletal muscle, retains the ability to divide throughout life: http://www.biol … 9/10_muscle.htm . Let’s add hyperplasia to the hyper list for smooth muscle.

Here’s an interesting read on a similar subject from another forum:

http://www.mens -network.com/fo … .php/t-648.html

Very interesting find!

Very interesting Modesto…the comments in that biology-online link coincide very closely with what I was going to say here…

The I in IPR stands for Injure (well, if you’re sqeamish, its Inflammation) fellas. This is good information, about 50% of a penis is comprised of smooth muscle, presumably primarily in the form of the CCs and the CS…that can grow through hyperplasia (a process that I think we’ve also referred to as distraction hystogenesis), and then there are some other smaller percentages of tissue made up of collagenous material, primarily in the form of the tunica (and the less important for this discussion, ligaments) being the structural limiting factor for growth, and miscellaneous more stretchable things like nerves and veins and such and then some fluids.

So the IPR training gig goes like this: strain (Injure) the limiting factor, the tunica, and then take advantage of a bit of a volume problem, where subsequent to straining the tunica, there’s a finite amount of ‘new volume’ (ΔV) available for the contained (largely smooth muscle) tissues to occupy. So with a bit of jelqing or pumping we “encourage” our existing SM tissues to deform/shear in the direction of the tissue density gradient, from occupied space toward finitely, relatively ‘unoccupied’ space by Injuring them in that preferred direction.

Then we apply light tension in the direction of the ‘new volume’ (ΔV) in order to keep that ‘new volume’ open while hyperplasia (distraction hystogenesis) takes place to fill it in. This is Proliferation.

The tunica proliferates too as a result of the strain to it. With growth of Type I collagen, which is a bitch to deform. So we let (all of) our tissues rest, while that pesky old Type I collagen is reabsorbed and replaced with that gullible little Tpe III collagen that we know how to deform (if ever so finitely). This is Remodelling.

IPR hanging + jelqing I phase training followed by ADS or ADC P-phase training cycling at both micro and macro scales is certainly consistent with a strategy that would accomplish the making and filling of ‘new volume’, while transitioning from the former to the latter process through the course of each cycle, thereby maximizing the effectiveness of each phase/process while still accommodating a smooth transition between them. And it sure has worked for me, he says ΔV= 0.88 later.

Is there anything more we need to know?

Oops, it was the mens-network link that coincided with my thoughts, not the biology-online link :) .

Thank you Modesto. I see you got it to work ;) . I appreciate the moving around.

:up:

Good show, Remek.

:up: :up:

I’d appreciate it if my original “first” and “second” comments (from the other thread) that I refer to in what is now this thread’s post #3 were also included in this thread in order to provide the proper context (in the same thread) for my follow up comments that now appear in what is now this thread’s post #3.

Also, on a more chemical level, Nitric oxide is required for the relaxation of the smooth muscle.

This is noted in this article (and many, many, others):