The Penis (smooth) Muscle Theory

Remek, Xeno:

Good job, looking forward to see a ongoing discussion at this thread about the smooth muscle theory.

BTW, now every “sex expert” has to say something else than “It’s impossible, the penis isn’t made up by muscles”

I think this could lead to a scientific proof of PE power.

Great guys!

/Swensk

Most of the body builders workout each part of their body once ~ three times a week.

So shouldn’t penis should be exercised 1 ~ 3 times a week? Or can it be different, because it’s a soft muscle?

This shouldn’t influence your routine much. Carry on doing what gives you gains, silver. :)

IMO, the tunica needs to recuperate too. IF we don’t let the tunica recuperate (or decondition), then sooner or later the gains will come to a stop and we hit a plateau (most of us, anyway).

However, this is all theory. Perhaps the tunica isn’t as big of a factor in PE as we have precieved it to be?

Also, xeno, if we find out what were up against, then we find out how to fight it best. Simple strategy.

I hear you rem. We just find out what we’re up against in different ways. You read, research and make theories based on what you discover about rats, rabbits and other people’s tissues.

I’ve experimented, iterated, and made a model based on what I’ve discovered about my tissues.

IOW, I’ve let MY tissues tell me what I’m up against. Smart strategy.

It appears that hypertension (high blood pressure) is associated with increased smooth muscle mass in human arteries:

http://www.pnas .org/cgi/conten … ract/78/12/7759

The above study seems to have concluded that growth in this experiment was due to hypertrophy, rather than hyperplasia.

The implication is that the mass of smooth muscle grows as an adaptive response to hypertension. Perhaps this is one reason that impaction exercises (jelqing, clamping), which raise blood pressure within the penis, are effective at increasing penile mass.

Here’s a study that explores hyperplasia: http://www.gale nicom.com/medli … 91/jo:0009-7322

It also looks at the role of serotonin, which might be interesting to anybody who takes prozac (fluoxetine) or other SSRIs.

Various others, provided for reference but not studied in detail:

http://www.ncbi .nlm.nih.gov/en … t_uids=15059929

http://www.find articles.com/p/ … 121/ai_84666848

http://content. karger.com/Prod … ArtikelNr=78785

http://www.chil drenshospital.o … Physiol-Lee.pdf

http://www.phas sociation.org/M … 5/serotonin.asp

http://www.pubm edcentral.gov/a … gi?artid=218758

http://www.fac. org.ar/scvc/lla … ett/arnetti.htm

http://www.cvph ysiology.com/Bl … 0Flow/BF011.htm

In my mind, there is no question over whether we can stimulate growth of the penile smooth muscle but the way I see it, we are still facing the same problem, the tunica.

The following paper demonstrates that PE would probably be much easier without the tunica:
Lengthening Shortened Penis Caused By Peyronie’s…

I recently read the following article:

and it got me thinking about how my penis seems to have something analogous to muscle memory. If I don’t PE for awhile, I loose some of my gains but they come back really fast when I resume.

I also know that the penis adapts to exercise. We all know that if a newbie does extreme clamping on day one, they will make a bruised mess of their dick so the forces we apply to our dicks somehow result in a protective biochemical "reinforcement" response.

So what do we do? We can use collagen softening agents but I worry about the veins and arteries because they are also surrounded by a collagen coat.

We can keep applying greater and greater forces but we know that seems to lead to injures including blisters, thrombosed veins, difficulty with erections and various other negative P.I.s.

So that brings me to two schools of thought that I plan to further investigate and those are the patent and Xeno’s penis tale:

Patent Pending Chemical PE

The Holy Grail of PE is found!!!

xenolith - Finding xeno: a penis tale

By design, one determines the time point at which gains slow in Xeno’s approach and the forces are always kept low in the the patent protocol, but the forces are applied over a long time period, perhaps allowing for healing in the extended state.

The patent might be complete crap (although Potaba is interesting), I really don’t know. Also, I am not certain that Xeno or anyone else can take the necessary measurements with the precision that would be necessary to accurately find the inflection points in his analysis but both are certainly worth further analysis in my humble opinion.

So, in conclusion, yes, I think we can induce smooth muscle hypertrophy but I think that is the easy part because nothing is going to happen until we expand the tunica, the probably limiting factor.

Im certainly no scientist when it comes to PE, but I do agree with PS here.

The observations I have made in the last few years of the methods that do/ do not work for me point squarely at the tunica as being the limiting factor in PE.

Im going back to reread my notes where I started to develop exercises to target the layers of the tunica seperately, try to develop them further, and see what I can achieve.

Can percentage rate of the smooth muscle explain why some can gain longer times /(and more) than others?

/Swensk

The principle observation that contradicts the notion that the tunica is THE limiting factor in PE is that BPEL is greater than BPFSL in many people.

Very interesting discussion. I did a little searching myself and found this..

http://www.casc adebio.com/pdfd … M231%20SMGS.pdf
http://www.casc adebio.com/inde … Content&CID=543

Now these products are for scientific purposes NOT human consumption but still, if you look at the ingredients doesn’t it strike you as something that could have possibilities down the road?

I don’t know how I let this one slip past me, but I must say this was a most interesting read. The condensed version is that surgeons treated men for Peyronie’s Disease by making a circular incision around the tunica, removing the fibrous plaque, and inserting a graft of venous tissue. Postoperatively, the men were instructed to use a vacuum pump for 30 minutes per day. Men grew an average of 2" or so after 18 months. Their only postoperative complaint was soft erections.

This study is certainly strong evidence that the tunica is a limiting factor—especially for people with Peyronie’s. What’s not clear to me is whether this procedure would be equally effective for guys without Peyronie’s. Postoperative lengths, even after the substantial gains, brought the guys back to only "average" or so length.

For guys without Peyronie’s, I don’t know whether the same results could be obtained by a similar procedure, or whether other limiting factors would enter the picture.

Thank you very much for this excellent article. I really, really appreciate it ;) .

It is entirely possible that relaxation of the smooth muscle may be required for an optimal BPFSL measurement but that doesn’t mean that there is a contradiction because the tunica is still the best candidate as the limiting factor during erection. It is the best candidate because of its structure (longitudinal and lateral fibers) as well as the priapism study findings which conclude that megaphalus results from a loss in tunica elasticity:

penismith - Experimental new type of clamping: journal of development

http://www.inge ntaconnect.com/ … 1jjour1w6.alice

as well as the tunica incision paper linked a few posts back.

Seems to me that the loss of elasticity would be a result of the priapism, not the cause of priapism. ‘Splain it to me gents.