Here is the link to the full study:
http://144.92.4 8.140/files/loc … trinsic2005.pdf
look what it says:
"After a Grade III injury (complete rupture) to ligament, a
region of scar tissue is deposited to provide a provisional
matrix for connecting the two residual ends of the ruptured
tissue (Frank et al., 1983; Jack, 1950). Biochemical analysis
of this scar tissue reveals changes in total collagen and glycosaminoglycan content within the scar
tissue out to 40 weeks post-injury (Frank et al., 1983).
………………
Mechanical evaluation of ligaments after subfailure
injury revealed a substantial strength deficit and increased
deformations within the strain-stiffening toe-region of the
load-deformation, consistent with previous reports (Provenzano
et al., 2002a,b) and indicating a significant tissue
injury. This early portion of the stress–strain curve is
physiologically relevant. Changes in mechanical behavior in
this region indicate a longer functional length prior to load
bearing…
Results herein point toward an intrinsic process of collagenous
ECM remodeling that relies more heavily on
replacing type I collagen directly than producing a predominantly
type III collagen provisional matrix (scar) to be later
replaced by type I collagen.
Furthermore, proteoglycan
mRNA levels indicate significant increases in conjunction
with increased collagen expression. Since proteoglycans
such as decorin, lumican, and fibromodulin are associated
with collagen fibrillogenesis (Birk et al., 1995; Chakravarti
et al., 1998; Ezura et al., 2000; Jepsen et al., 2002; Svensson
et al., 1999; Vogel and Trotter, 1987), as well as cell
behavior (see Kresse and Schonherr, 2001), it appears likely
that they are, at least in part, up-regulated to help modulate
the fibrillogenesis of the newly synthesized collagen…"
http://144.92.4 8.140/files/loc … trinsic2005.pdf
According to this link, an Achille’ tendon takes 6 months to fully heal:
http://achilles blog.com/files/ … re_mass_gen.pdf
Summint it up, grade II injuries (no inflammation neither scar deposition) are repaired through more ‘healthy’ collagen deposition and new fibers. Being this tissue similar to the normal tissue, it can be elongated again, ‘small’ injuries (grade II) to this tissue will cause new collagen deposition and so on. The whole process can take few weeks. Scar tissue, instead, is less elastic and requires more time to disappear. Without even thinking to the pain, inability to have intercourse etc..
Beside that, how can you pull on your ligs without pulling the penis? And again, stretching ligs would add about nothing to erect length. In penis surgery, where the ligs are cut, there is a gain in EL of about 1/4"; some people don’t report any lenght gains at all from this procedure. If cutting ligs can’t do much, what stretching them can accomplish?