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Gaining volume with Kyrpa

IPR opposition

Now first of all I am really happy to help anyone who thinks that I may have some capability for doing such.
I will be generously helping in the future as well with all I have to do so.

What I would like to say for everyone sending me questions after reading few of my first posts. Don´t send.

I know there is hundred pages ahead, but the thing is the understanding of the PE growth process has evolved far from the initial frame adopted from the IPR methodology.

The concept I am embracing does not involve deliberately causing trauma at any level of the tissue structure.
Secondly we re not in any circumstances dealing with exceeding elastic limits or reaching the plastic zone.

Reading the whole thread you know what I am all up to. And what the science field is all up to trying to elongate and expand connective tissue for inducing growth responses.

We are not dealing with healing the wounds. Either we are not obligated to cause structural or cellular level damages to engage the healing responses to further inducing growth.

It really would be helpful if the first post of the progress log stayed editable for the OP forever.

I will start a new progress log for the second coming during following months, with a freshened thoughts of my own, filtered from the available knowledge around.

Adaptation responses seen as a growth via mechanotransduction. I don´t mind letting the wounds heal on their own wherever they tend to show up, but I am not into it.


START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)


Last edited by Kyrpa : 12-17-2020 at .

Apoptosis does not sound good. When I think about it, I’m predestined for it with my clamping setup. Even when I break the tension, the glans is still clamped off. Even loosening the screw doesn’t do much because the therapeutic band is tight. Or do you mean that clamping off isn’t a problem, just the pull?
I really need to revise my setup.

What should be the maximum pause between phase 1 and phase 2?

I’ve found that the post-BPFSL doesn’t last long and the pre-BPFSL quickly sets in again. What is your experience? How long does "your" post-BPFSL last?

About the silicone materials:
These silicones are 2-component materials. You mix them 1:1 and stir for 3 min. The material is then poured at room temperature and it cures chemically, so without the effect of temperature. Temperatures around 80 °C accelerate the curing, but are not necessary.

We have to evaluate the mechanical properties. There are huge differences in these silicone materials and I think we have to use different materials if we want to build an US phantom or a cock ring.

The viscosity of the material alone determines the manufacturing process. The DragonSkin20 is quite tough and needs to be trowelled in my opinion. I found a video about this (here the DragonSkin10 is processed, which is a bit firmer with 23000 mPas):

Privacy info: Clicking on this image will enable content from www.youtube.com. Privacy friendly version via Piped.

From 2:16 comes the part with the processing.

The DragonSkin20 seems to be very well suitable for US phantoms. However, looking at the material properties, I think other materials are better for such parts as a cock ring.
There is still the DragonSkin10 very fast variant, which has to be processed within 4 min and is demoldable after 30 min. Theoretically, you could use the stretched penis as a mold and spread the stuff on the dorsal side while stretching. So you would get a perfect US phantom that fits perfectly at least for a certain time. But I think this is exaggerated.

If you want to make special parts for your torture tool (extender), you should use stronger materials that can be molded (6000 mPas). Spontaneously, I find two products from Wagnersil (28 LE and 35 LE). With 28 and 35 Shore A, respectively, they are significantly harder than the other materials, so I assume it is better for this application. The two materials have an elongation at break of 500% (28 LE) and 450% (35 LE) and can withstand a stress of 5 MPa.

I have tabulated the data to give an overview. Everything I write here is my interpretation from the data sheets and some Youtube videos. I haven’t processed the material myself yet, but find it very interesting. I am still considering whether to cast or trowel my Phantom, then I will decide which material to buy.

Materials 2020-12-17.webp
(40.3 KB, 150 views)

Originally Posted by Rocco25
Apoptosis does not sound good. When I think about it, I’m predestined for it with my clamping setup. Even when I break the tension, the glans is still clamped off.
Even loosening the screw doesn’t do much because the therapeutic band is tight. Or do you mean that clamping off isn’t a problem, just the pull?
I really need to revise my setup.

What should be the maximum pause between phase 1 and phase 2?

I’ve found that the post-BPFSL doesn’t last long and the pre-BPFSL quickly sets in again. What is your experience? How long does “your” post-BPFSL last?

Yeah in your setup the tissue is all the time compressed. The apoptosis has been studied widely happening under compressive forces as well. With the protocol of mine before the use of the latest setup that wasn´t an issue. At the point releasing the phase 1 stretch the vacuum pressure had already subsided and had to be re-applied before the heated phase. And for the third phase I usually used manual pulls anyways.

With the recent setup the noose sits tight the whole process of 60 minutes if not released briefly after the phase 1 , which mentioned to be beneficial, as for the glans blood circulation refreshment basis as well.

I don´t know how long the post -BPFSL lasts, as I have not paid attention for it.

In the literature they have used various recovery times between singular elongations when determining elastic behaviour.
It is different of course depending on the tissue and the strains caused.

This is off the records as I am speaking from the memory basis without checking it out ,but some cases they have mentioned 15 minutes being full recovery attained for tendons for example allowing to do non biased second attempt.
I will come back to this when writing up the full optimized protocol at some point.

As long as we stay no longer than few minutes apart we are still benefiting the most out of the first phase results.

Surely one thing more to consider if aiming to optimize every single aspect of the procedure.
Not really able to confirm many of these nuances things in reality these optimizing efforts stay highly theoretical of course.
Keeping the focus on the main concept is the priority.


START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)

Thank you for your answer, Kyrpa. I was passive here for a long time because I didn’t want to disturb the process of the thread with my questions. But the gain in understanding since I’ve been writing here is overwhelming. Thank you so much for the information and especially for your patience in discussing everything in detail. I hope I can give something back in some small way with my contributions.

To be honest, I am disillusioned. In the end, my approach was wrong and I need to rethink everything. We want to have more cells in the dick and not witness cells dying due to apoptosis.

How do you want to deal with this in the future with your new setup? Finally, you increase the risk for apoptosis if you go through 1 h.

I really don’t have a solution up my sleeve, but am already worrying about how things will go from January. My clamp setup is not a solution according to my current understanding. Therefore, I’m thinking about looking into the vacuum again. An example could be that one part is clamping, and another vacuum. In which order I do not know yet. For Vacuum during stress relaxation speaks the low load. Against it speaks IR. During US, transducer accessibility is better with Vacuum, but loads are higher. But it is 10 min less. So they are both lazy tradeoffs.

I’ll have to sleep on it again until I find a clever answer. But whichever way it goes, I think based on current evidence that we should generally take a little break after stress relaxation so we don’t run the risk of experiencing apoptosis.

The idea of casting silicone parts ourselves opens up new options. I already have one or two ideas, but they are not yet suitable for the forum here. As soon as something usable comes around, I’ll share it here.

Originally Posted by Rocco25
Apoptosis does not sound good. When I think about it, I’m predestined for it with my clamping setup. Even when I break the tension, the glans is still clamped off. Even loosening the screw doesn’t do much because the therapeutic band is tight. Or do you mean that clamping off isn’t a problem, just the pull?
I really need to revise my setup.

Few words about the apoptosis. Now the strain percentage has lot to do how the cells react to the tensional stress.

We shouldn´t put too much of a emphasis on phenomena in the early stage of the workout as the phase 1 is all about to take take “loose out of structure”,
straighten the undulation of collagen fibers, align the fibril, fiber and cellular structure(ECM) with the direction of the applied force.

At that point the mechanotransduction really is transfering the external stress to the mechanosensing fibroblast cells with full capacity. It is a condtioning phase for the circumstances to be potentiated as optimal.

So the relevant emphasis should be directed to the phases 2 and 3, the duration to be limited to 30 minutes.

Keep your focus on the priorities. Maximize the strain in each phase with threshold loads and you will enable growth as a permanent elongation.


START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)

I think that stretching is not a problem in terms of apoptosis. Even if phase 1 should not be overrated - as you write - there is still the option to take a short break before phase 2. This is manageable in my eyes.

I rather see the problem in the clamping. Both your new setup and mine challenge the thing for 1 h, unless we intentionally put it down in between so the blood can circulate again. That’s where my thought of separating the force application came from (e.g. Vacuum and clamping).

Hi Kyrpa, I was wondering if you’ve ever read this article

https://www.pai nscience.com/ar … issue-depth.php

And if so, what are your thoughts on the conclusion that surface heating using a heat pad for example is more effective at raising temperatures than utlrasound?

Article quoted below:

Quote
Dry heat is more easily tolerated, so the hope was that it would be more effective, and it was: dry heat on the hands and feet raised tissue temperature at a half centimetre depth to 9˚ and 5˚ respectively, somewhat more than hot wax or water. The authors make the case these methods are superior to the “penetrating” heat of microwaves or ultrasound because they can be easily applied to larger surface areas. And so they concluded

That surface heating modalities are much more effective in producing elevated temperatures than is ultrasound therapy or diathermy at depths of up to 1.2cm.

Source: https://www.pai nscience.com/ar … issue-depth.php

Do you interpret this differently?

I see the distinction they make considering large areas vs small areas, but for me personally, wrapping my unit with the heating pad while hanging can keep it so hot that it’s almost uncomfortable, and when I remove the pad, I actively feel the heat emanating off. A stark contrast compared to US as I hardly feel my unit would feel warm at all if I didn’t have the heat pad between it and my leg while using my US device.

Another small comment, I’ve noticed significant reduction in strain if I DON’T use the heat pad between my leg and unit during the US heating.

Another thought I had.. Have you given any thought to elongated heat cycling? For example, 10 minutes heat/3-5 minutes cool while hanging or using a band, followed by stretched US heat.

Originally Posted by TimeIt
Hi Kyrpa, I was wondering if you’ve ever read this article

https://www.pai nscience.com/ar … issue-depth.php

And if so, what are your thoughts on the conclusion that surface heating using a heat pad for example is more effective at raising temperatures than utlrasound?

Article quoted below:

Do you interpret this differently?

I see the distinction they make considering large areas vs small areas, but for me personally, wrapping my unit with the heating pad while hanging can keep it so hot that it’s almost uncomfortable, and when I remove the pad, I actively feel the heat emanating off. A stark contrast compared to US as I hardly feel my unit would feel warm at all if I didn’t have the heat pad between it and my leg while using my US device.

Hello,

Happens to be that I have interpreted the study earlier on my own. And I suggest you to do the same as well.
Overall I am seeing this whole out coming with such a strong claims, being biased at least because of the fluidotherapy group interests.
Looking in the study the proportions are taken out of the context to serve their cause.

The 5C and 9C temperature raises are taken from joint capsules in thumb and big toe , having very poor blood supply to cool it down.

The muscular temperature raise stays much more moderate level. Similar you can be expecting with heating penis.
I highly doubt you will be expecting to reach no more than 39C urethral temperature no matter how hot you feel your skin being.

The hand muscle temperature is showing similar resting temperature to urethral temperature.
The superficial blood circulation in the penis is very effective cooling system. Also the intracavernous blood circulation is likely to multiply trying to heat the unit externally or internally.

The study they are referring attached below. Also contrasting numbers to what you are referring from hand muscle seen in the graph.
With foot muscle the effect was even worse.

I am more than happy to see if anybody brings us the urethral temperatures they are reaching with the heating methods they use.
I am doing my homework and have no interest to do everyone else´s part as well.

There is absolutely no need for opinion of anything, mine or anybody elses if the data is easily available.
There are enough variables in PE which rely on opinions, data magnitudes harder to get than heating efficiency.

Please prove me wrong with the data , as we don´t have it. Even plethora of anecdotes is not science, data is.

I am not starting arguing about the superiority of any method, what I am saying that me and few others with me, are taking steps into measured data basis decision making on PE. Out of the realms of multi variable feel basis mess it has been for decades. We need to know.
It would be good to know the effect of your method in numbers also, it would be very helpful for many.


START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)

Originally Posted by TimeIt
Another small comment, I’ve noticed significant reduction in strain if I DON’T use the heat pad between my leg and unit during the US heating.

Another thought I had.. Have you given any thought to elongated heat cycling? For example, 10 minutes heat/3-5 minutes cool while hanging or using a band, followed by stretched US heat.

I do think that you are not getting to therapeutic window soon enough with US only.
Similar what I found using the one transducer only method my early stages. Having the rices sock to keep certain temperature level it worked better.
So using any additional heat source will help you getting there. The core is hard to get heated enough.
Later on I had no need for it once starting the dual transducer application.

Many thoughts have been pondered. As long as I have been successful with the established one , I have not tried.
But once it does not produce anymore, instead of increasing load or workout volume I will.

Like you suggested, or using multiple shorter heating cycles by the US alone. Letting the tension off between and in the las t set only applying the cooldown stretch. There is great amount of things to do upgrading the intensity of the workout still not putting more weight or piling up workout hours and volume.

Now that you have the strain percentage as your guide you can easily make the decisions. Easy to make the difference. I encourage you to explore.

I did forget to attach the picture on previous post.

Temp Raise fluidotherapy.webp
(51.7 KB, 150 views)

START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)

Thank you Kyrpa, your thoughts make sense. I am no scientist but trying to learn the process of experimentation and data collection as well so I can contribute based on my experiences as well.

One question, can you offer any tips or advice on being more accurate with taking measurements? So far it’s been frustrating to try to standardize it at all. Right now, stand exactly one foot distance from a wall (one of my feet lengths so I’m always squared up against the wall), straighten my body 100% and lean forward with my head against the wall, then I measure with the ruler level and the end against the wall. Something like this. Even this is slightly unreliable, but I think it’s the best I’ve got so far. Maybe someone else can offer suggestions or find this helpful.

LOL measurements.webp
(3.0 KB, 15 views)
LOL measurements.webp
(3.0 KB, 10 views)

Originally Posted by Kyrpa
Even plethora of anecdotes is not science, data is.

Amen!

Originally Posted by TimeIt
Thank you Kyrpa, your thoughts make sense. I am no scientist but trying to learn the process of experimentation and data collection as well so I can contribute based on my experiences as well.

One question, can you offer any tips or advice on being more accurate with taking measurements? So far it’s been frustrating to try to standardize it at all. Right now, stand exactly one foot distance from a wall (one of my feet lengths so I’m always squared up against the wall), straighten my body 100% and lean forward with my head against the wall, then I measure with the ruler level and the end against the wall. Something like this. Even this is slightly unreliable, but I think it’s the best I’ve got so far. Maybe someone else can offer suggestions or find this helpful.

BPFSL is the most accurate measurement.


Period 1: 06/08/2020 BPFSL: 22cm (8.66") BPEL: 22cm (8.66") EG: 15.8cm (6.25") => 09/07/2020 BPFSL: 23.9cm (9.40")

Period 2: 05/01/2021 BPFSL: 24cm (9.44") BPEL: 22cm (8.66") EG: 15.8cm (6.25") => 07/24/2021 BPFSL: 25.4cm (10.00") BPEL: 23.5cm (9.25")

Goal: 1 Foot x 7.5 Inches (30.48cm x 19.05cm) NBPEL

Merry Christmas already.

Forget the dick for few days , your life does not depend on it.


START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)

Originally Posted by Kyrpa
Yes that can be done with ultrasound , but if it will happen with the intensity available after attenuated in the shaft before reaching the fat pad is another thing.

After using it one and half years like that absolutely no fat has seemingly have been destroyed on the top of my quads. None.

BUT I tried to combine the studies concerning the thing on my lower abdomen fat pad which is thick I can tell.
I used two transducer focused on the same spot having in theory 3.2 w/cm^2 of focal ultrasound . Keeping it in a spot to the point of feeling pain before moving on.
I used it for the approximately 8x 20 cm area for 30 minutes a time.
Before the vigorous heating I had used ESWT to prepare the area banging it with as much as joules I could possibly tolerate for 5000 shocks.

I was able to replicate the results they had with pig abdomen fat and similarly in rat studies where they had 0.5 mm reduction on skinfold caliper measurements per treatment .

Plausible yes. With lots of dedicated and extremely painful efforts to do so . Happening as a collateral damage by chance absolutely no way.

Kyrpa you used the US pro to reduce fat? Do you think a single US pro on the pubic fat pad could achieve the same thing?

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