Originally Posted by Kyrpa
But what have started to puzzle me, is that if there is stress dependency along with needed elongation still for the top of all things?
The minimum amount of recruited and stretched collagen fibrils needed?
I really don´t know. Therefor I will use manual hand grip and + 6kg loads for the rest of this Period 3 and continue experimenting in the future with this aspect.
At least I know my glans will be thanking me.
Measured pre BPFSL 23.4cm and post BPFSL 24 cm. Strain of 2.6%. (avg)
Fantastic results man. Your continued success gives us all hope that gains are in store given effort is put in, and there is no end in sight apparently thus far.
Those questions have always confounded me as well. My take is there is dependency at some level. However you know my main focus has always been increasing load, so there is some bias. And this is in line with traditional PE for length. However, you have proven thus far that not much load is needed and look at your gains. So it is a point in favor of not much dependency.
For the second question. In my understanding, the cyclic loading recruits less fibers than static loading. The cyclic loading recruits the shorter fibers first over and over each cyclic stretch, causing the shorter fibers fatigue and thus breaking. Once broken, the mid length fibers are recruited to support the same load. This happens again and again until a bigger cross section of the tunica is recruited. However, take a look at static stretch, the shorter fibers are elongated until mid are recruited and then long are recruited. Because there is no repetitive fatigue in the short fibers, these may just elongate but not tear to the length of longer fibers. It may not answer the question, but I think with cyclic loading there comes a point where the fibers “snap”. Say you have day 1, day 2, day 3 using 6kg and all are BPFSL at 24cm. And you can’t break through. It may be these shorter fibers have not been fatigued enough to break past 24cm. By increasing the load above normal levels, as the studies show that at 60% UTS cyclic loading causes greater fatigue and shorter fibril rupture, you may break past 24cm by increasing to “60% UTS” (to reference that study) which we don’t know what that would be, but say just for the sake of this example, 10kg (probably nowhere near 60% UTS but still more than 6kg, it may be more or less, whatever is required to break through, this is the point). You fatigue it with 10kg until short fibrils rupture and the mid length fibers are thus fully and primarily recruited. They were secondarily recruited before, now they are primarily recruited. You break past 24cm. Now you have a 24.5 cm dick. There was a displacement that occurred in the stress strain curve to the right, thanks to the larger stress and cyclic fatigue, your shorter fibrils are broken and coiled up (appearing to be crimp but actually just broken, as in that study showed). Now you can go back to your same normal load of 6kg and target the mid range fibers, now that the shorter fibers are out of the picture. Now your mid range fibers may need more than 6kg, because they are longer, they can bear more load before fatigue breaks them. So you may need to increase this to 7kg lets say. Day 1 day 2 day 3 BPFSL is 24.5cm. Repeat the 10+kg fatigue and break through to even longer fibers. Now you at 25cm lets say. Once you are at your larger fibers, there may be diminishing returns. Or simply the weight needed to fatigue these is too painful, etc.
Because you are always manual cyclic stretching and can’t gauge the load accurately, and always at the top of the length of the shortest of your fibrils, this could be the reason why your fibrils fatigue, break, and lead to gains. Because each cycle you are breaking the fibrils that are shorter and moving on to the longer fibrils.
Anyway that is my two cents attempt to try and answer these questions.
However, my answer if true begs the question, once broken what happens to the shorter fibers? Does this lead to collagenous buildup? Repairs are done and integrated to the longer fibers? Or they wither and die and stay coiled up forever? How does this affect the load for the future? Does it increase the required load or no effect?
I have a feeling my static loading the shorter fibers never ruptured, they were elongated only, to 22.8 in my case. Then after a break, they recoiled back up to 21.8. Now with cyclic loading I will aim to break these, so the recoiling can’t happen. And have a permanent length increase.