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Gaining volume with Kyrpa

Originally Posted by Kyrpa

My cumulative BPFSL gains have been exactly 6,5% and 6% at the point the strain percentages have started to diminish. BPFSL still continued to increase up to 7% and 7,5% while being at the second parts of the Periods. There is definitely something in it.

I think the stress strain behavior is better explained in the cumulative effect of a cycle (1 week, or various sessions), and not just 1 session, as I have predominantly focused on.

In a 1 day context I think you are right we might just be in toe region and transitional.

But in a cumulative context I think we reach the linear region. I feel the yield point at the 5-6% cumulative strain point to be quite strong that I need to up the stress to get past it. I think this may be the point were one taps into the plastic region past the yield point.

The studies we refer to are usually conducted over short periods, sometimes long, but mostly within 1hr or less than 24 hrs. We are conducting our sessions over days. So although our tunica UTS may be high, the weakening effect of days of stretching etc, and puting it through hell, it could be that we do reach the higher regions of the curve.

I have been experiencing this displacement event ever since I started and it happened again today. I was trying to get past 21.8 post BPFSL. My cumulative strain for this cycle was 3.3%, and finally managed 22.2. Which I’ve hit before in my macro cycle 3, but nonetheless felt good to hit again. My cumulative stress is now 4.72%.

Anyway, this concept of cumulative strain seems to be important as many data points coincide at 5-6% for me. And seems to be similar to yours. And remembering the rat tail study, and other studies, they mention the 6% was plastic region IIRC. or was it anything above 4%? I can’t remember.


Genesis 2006 = 5.8" x 4.7" /// Round 3 2019: Hanging again = 7.99" x 5.5" /// ST Goal 2019 = 8" x 6" /// End Game 2020 = 9" x 6.5"

Originally Posted by manko007
I think the stress strain behavior is better explained in the cumulative effect of a cycle (1 week, or various sessions), and not just 1 session, as I have predominantly focused on.

In a 1 day context I think you are right we might just be in toe region and transitional.

But in a cumulative context I think we reach the linear region. I feel the yield point at the 5-6% cumulative strain point to be quite strong that I need to up the stress to get past it. I think this may be the point were one taps into the plastic region past the yield point.

The studies we refer to are usually conducted over short periods, sometimes long, but mostly within 1hr or less than 24 hrs. We are conducting our sessions over days. So although our tunica UTS may be high, the weakening effect of days of stretching etc, and puting it through hell, it could be that we do reach the higher regions of the curve.

I have been experiencing this displacement event ever since I started and it happened again today. I was trying to get past 21.8 post BPFSL. My cumulative strain for this cycle was 3.3%, and finally managed 22.2. Which I’ve hit before in my macro cycle 3, but nonetheless felt good to hit again. My cumulative stress is now 4.72%.

Anyway, this concept of cumulative strain seems to be important as many data points coincide at 5-6% for me. And seems to be similar to yours. And remembering the rat tail study, and other studies, they mention the 6% was plastic region IIRC. or was it anything above 4%? I can’t remember.

Taking in account the cumulative elongation and the fact collagenous tissues of our penises have not healed, and considering this whole period as one long lasting exercise , then we shall reach the proportional limit relative to the initial baseline.

Yes at the 6-7 % cumulative elongation on BPFSL after few weeks on program we hit the wall and we are forced to stop stretching, and the healing have to start.


START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)

Originally Posted by capernicus1
You guys might enjoy this thread if you haven’t already seen it.
Possible reason for PE induced growth

Thanks,

There have been some great researchers in the past, this guy being one of them. I have been reading lot of them before I decided to go the way you are seeing here. Your linked thread included.


START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)

Originally Posted by Growing4it
DocJ, using the link you provided, and using the table for optimal heating of muscle with 3mhz 2.5cm deep I should be able to effectively do my heat routine in 28 minutes at .08-1 W/CM2. I think the question is how does heating a penis with good blood flow compare to heating muscle tissue. I don’t believe I have been overheating, but I believe for the sake of safety I need to find a good way to check the temperature. At 3mhz the heat should penetrate 2.5 centimeters, and that is more then my stretched flattened unit. I wonder if a thermometer strip between my leg and dick would be effective with a good amount of ultrasound gel?

Hey Growing4it, sorry for the delay responding been traveling out of the country. That sounds like a good idea to use the thermometer strip - I was going to do the same for myself. Remember, 3MHz is 3x the energy and heats 3X faster than 1MHz so you need to be mindful of the amount of energy and can’t be compared to the same watts as those using 1MHz. Really my only concern here is clamping while heating with US. During clamping the blood is confined/constricted to an area where there is no flow and the blood is under pressure, which increases with adding more heat. Adding intense energy with US in this situation, in my mind, you run the risk of coagulation of the blood which could be a really bad situation. I would heat first with US, remove the US, then clamp which I am going to try myself down the road. FYI, I do 2.0W at 3MHZ for 2 10 minute treatments as part of Kyrpa’s routine, which is higher than the recommended published therapeutic dose, but I feel my tissue can tolerate it because there is no pain, only heat as long as the transducer is kept in motion. Take what I’m saying with a grain of salt, there is no science on US applied to the penis and I’ve only done this for less than two weeks and I got derailed from a bad blister of applying too much vacuum to the tip of my penis, I can’t wait to start again next week. I just don’t want anyone here to get an injury, US at 3MHz is powerful. Were doing our own “science” here and I appreciate all of the feedback from everybody using US for PE.

Cycle 6 of the Period 3

Day1 the bad

This round was not that good, already having not so good strains at cycle 5, this cycle started in similar fashion. The unit felt stiff and nonelastic.
Stretching did not give the normally felt exhaustion on tissues

Measured Pre-BPFSL 23.4cm and Post – BPFSL 23.9 cm. Strain 2.4%

Day 2 the ugly

This time my schedule forced me out of my protocol.
But I was able to wear my ADS setup stretching around the waist and down to leg. Used luggage scale to gauge load and stretched with ~2 kg .
Felt stretched and fatigued for the entire shaft after 120 min total stretching time.
No measurements taken.

Day 3 the good

This was normal exercise. Felt like being elastic and healthy again having normal feel while stretching.
Procedure was the same as have been during Period 3.

Measured Pre-BPFSL 23.5cm and Post – BPFSL 24.1 cm. Strain 2.9%

Days 4 and 5
I have to give additional comments for rest days being very nice with really good EQ and multiple spontaneous erections during these two days.
I am excited to start next five days cycle from these feelings.
Already measured BPFSL 23.6 cm during rest.


START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)

Diminishing returns or just a pothole on the road?

During Period 3 (41 days so far)

BPEL 21.4cm ->21.9 +0.5cm
BPSFL 22.7 cm->23.6 +0.9 cm
MSEG* 14.8 cm->14.9 +0.1

*Documented mseg is average from base (15.7), midshaft (14.7.) and behind the glans girth (14.3) for more accurate volume calculation.

Strain during exercises is reaching its top at 24.1 cm which is timewise thereabouts and in line with previous rounds.
What is new is that I am reaching the top at 6.2 % elongation calculated from the starting pre exercise BPFSL for this period (P3 ) 41 days ago .
At previous times I managed to push further as the same value was 7-8 % at this point and stalled there.
I will reproduce one more cycle trying to go further, but if it sticks with 24.1 cm , I will turn my focus on girth for the rest of the Period.

Is it the point of diminishing returns at the macro scale, maybe strain wise it is. If it is for the longrun, i really don´t know.

BPEL progressing identically as previous rounds, BPFSL gains just about falling outside the margins at this point.
Signs are still surely pointing in the direction of BPFSL gains approximating the point where the steep slope declines in to diminishing returns.
It is predictable and because still gaining at high rate with BPEL, there is no real cause to worry about it.

Day 1 Cycle7
Conditioning stretch with ADS. First attempt to exercise failed due interruption. Kept it 60 minutes.
Second take 60 min conditioning. Otherwise normal procedures.
Longer conditioning bringing no better stress relaxation or strain than 30 minutes in precise controlled stretch.
Day 1 Measured pre BPFSL 23.6 cm and post BPFSL 24.1 cm. Strain of 2.6%. (avg)

Day 2 Cycle 7
Normal protocol. Cond. 30min. Heated stretch 20min. Manuals 10min.
For extra I gave it a try for cooling down under tension.
Length fixed to maximum elongation on extender with icepack and kept there 10 min.
Pretty much tension as the tissues cooled.
BPFSL shrunk rapidly below pre-exercise measurement after releasing and felt elastic when tried to stretch it.

Measured pre BPFSL 23.6 cm and post BPFSL 24.1 cm after manual stretch. Strain of 2.6%. (avg)
After cooldown BPFSL 23.5cm

Day 3 Cycle 7
Just normal rituals.
Measured pre BPFSL 23.6cm and post BPFSL 24.1cm. Strain of 2.6%. (avg)

Progression_18_8_19.webp
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START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)


Last edited by Kyrpa : 08-18-2019 at .

I do find. And to some extend find it backing my story here also. Hope all readers find and are able to view this PE context easier by the simplifications presented in the links you provided.

The problem is that they are simplifications, missing few crucial characteristics seen when stretching penis. Every collagenous soft tissue organ has its unique stress strain curve when stretched. To simplify permanent penile elongation is not so easy task. Simplifying anything this complicated is never easy.

I took the privilege to tune the stress strain curve found in the links, to show the more detailed view of the reality we are experiencing when elongating penis with any loading procedure.
This will hopefully help to bring the content of the links provided better in to this context

Few things I would like to point out as my try on simplify :

1. We are not going to reach plastic region or even yield point in a single exercise no matter the load. Even if we ever could, there will be no flow region.
Something breaks badly soon after.
2. We are not going to reach proportional limit in a single exercise, but when achieving length gains in within certain time window we might.
This would result as permanent gains on BPFSL .
3. In a single exercise penile tissues stiffen to certain point until we reach the m - slope ( proportional(elastic) range, the slope where the tangent modulus is stable)
(in fact same characteristics are found in some ligament studies as I have previously pointed out)

In the attached picture picture I have pointed out the place we are operating on the stress strain curve with any form of PE at its best.

desimplification.webp
(85.4 KB, 194 views)

START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)

Heat of the moment

I have already mentioned elsewhere that phenomenological modelling is the description for my contributions theory wise.
To establish direct relations between the external stimuli via stretching and the observed tissue response without trying to explain the mechanisms behind the observations.
But Tutt and Manko have somehow got me involved and I am kind of forced to open thinking behind the things I have chosen do with my dick and why.

Here is my take on studies referred in the book: Science of flexibility by Michael Alter. I had previously captured screenshots from this and took a while to remember the source again but here it is. There are few things which I rely for being crucial for the growth I have achieved with my concept.
Heat is the key, so many times mentioned here at Thunders, but I think it is not clearly understood how high temperatures are actually needed.

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous when it is heated. (Warrent et al (1971,1976)
The mechanism behind this thermal transition may be destabilization inter-molecular bonding or enhanced viscous flow properties of the collagenous tissues, possibly allowing elongation to occur with less structural damage. (Rigby 1964)

Raising the temperature of tendon above 103 F(39,7 C) increases the amount of permanent elongation that results from the initial stretching. (Laban 1962, Lehmann et al . 1970)
Above 104F (40 C) occurs thermal transition of the microstructure of the collagen, significantly enhancing the viscous stress-relaxation of collagenous tissue and allowing greater plastic deformation during stretch. (Mason and Rigby 1964, Rigby et al 1959)

According to Sapega et al (1981), stretching connective tissue at elevated temperature the condition which under the tissue cools down affects the elongation that remains after tensile stress is removed.
They based their hypothesis on Lehmann et al. (1970) , which found that after heated tissue is stretched , maintaining tensile force during cooling down significantly increasing the relative proportion of plastic deformation compared with unloading the tissue while its temperature still high.
They also speculated that cooling the tissue before releasing allows the collagenous micro-structure to re-stabilize more toward its new stretched length.

However (Hardy and Woodlal 1998) have questioned benefits the cooling while tension. Applying cold while tension it diminished all the gains made in flexibility with every study group.
Therefor applying ice during cool down is not necessary.
IMO possibly even counterproductive. Perhaps, it is better to let the tissues cool down naturally , applying the tension via manual stretching or at extender with fixed length at maximal elongation achieved during heated exercises.

In my case all above mentioned should mean that operating at 41- 43 C the tissues are able to go through plastic deformation at some degree at strains well below proportional limit, not to mention plastic region which will remain untouchable in every circumstances while enlarging our penises.
Being able to elongate BPFSL at every cycle without reaching proportional limit at the stress strain curve.
Baseline BPFSL transitioning 1 mm at time forward and starting every cycle at new fresh starting point longer than before.
Stress strain curve staying the same and tangent modulus being intact, baseline just transitioning forward due elongation

I am hugely convinced that I am not breaking anything, no micro tears , no inflammation. Even if there is minor it is not the case of the growth here. This is something else.

Heat being the topic I would like to add few advantages with the temperatures I am using.
These were posted previously in here by guy named MX and are all valid:

“Increased Metabolic Rate - Metabolic effects - Enhances healing
Increased enzymatic activity has been seen in tissues at 39-43C (102-109F)
Enzyme activity rates begin decreasing beyond 43C (here was 45 but I corrected it) and cease completely at 50C (122F).
Any increase in enzyme rate will increase the cellular biochemical processes and the uptake of oxygen resulting in accelerated healing
Increase of tissue temperature shifts the oxygen-hemoglobin dissociation curve to the right which releases more oxygen for tissue repair.
Hemoglobin releases twice as much O2 when tissue temperature is 41C. (106F) than when it is at resting temperature. “

The last paragraphs comes very important to knowledge when starting to clamp with US.

Did some exercises too. Not much to mention except day 2 I skipped the manual stretching and went for the extender cool down.
Other wise basic routines. Keeping head down and staring my dick.

DAY 1 Measured pre BPFSL 23.6cm and post BPFSL 24.1cm. Strain of 2.6%. (avg)

DAY 2 Measured nothing.

DAY 3 Measured pre BPFSL 23.7cm and post BPFSL 24.1cm. Strain of 2.4%. (avg)


START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)


Last edited by Kyrpa : 08-22-2019 at .

One thing slipped through my fingers when posting .

Importantly (Hardy and Woodall 1998) found out that heat and stretch have cumulative effect on collagen structural changes. They also stated that therapeutic heat during stretching is superior to any other forms of stretching.

Talking about cumulative effects I can concur with my experiences easily.


START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)

I am certainly enjoying heat while extending.


Once upon a time (2015): 6.40” x 4.50”

Today: 7.25” x 5.00”, Thunder Cocks Unite!

I think we can...Little Engine’s Climb

It seems like the 40° threshold cited above would be achievable without US. I’ve spent time in both hot tubs and saunas hotter than that. What are your thoughts?

I’m using an IR bulb above, with a rice sock under, my shaft. I get the bulb positioned (and the rice sock heated) to a level which is just before surface discomfort. Surface temperatures (according to an IR gun above, and temperature probe underneath) around 45°+/-. I’m following Kypra’s protocol with weights adjusted for my physiology (1.25 - 1.75Kg conditioning; 2.25 - 2.5Kg strain sets). I have 5 weeks worth of data in my spreadsheet, and so far it has been working consistently (according to BPFSL pre/post) week after week. My t0 BPFSL has increased by a little over 3/16” (conservatively). So, I would say that it can work without US. Prior to this protocol my length had stalled for close to 8 months. I have a cord - how tough it is compared to other folks, I don’t know.

I was talking about the importance of heat under a tensile load on another forum and in the same vein the importance of holding the tension as it cools down. I also stated I have done ice baths in past but feel letting it cool naturally would be best. Do you mind if I quote some of what you posted, referencing you of course, it was really well worded and referenced.

I also don’t feel this is really inflammation or micro tears, at least not the inflammation xeno imagined it as. My last campaign I was only mildly sore maybe 2 days but gained very well. What do you think could be going on? Could be a mixture of things going on I would assume. The high heat under a load and cooling under a load to aid in restructuring of collagen fibers and performing these cycles? IPR itself? Increased metabolic activity signaling need for growth? I’m very intrigued.

Also where do you get your books? I have anatomy books but little on biomechanics.


My MaxVac Setup Longerstretch's Golf Weight and HTW setup My Log

Starting Size: circa 2003: 5 BPEL x 5.0 MSEG August 2007: 6 2/3 BPEL x 5.5 MSEG 04/22/08: 7.5 BPEL x 5.6 MSEG... On and Off again for a while... 11/25/13: 7.75 BPEL x 5.75 MSEG 08/01/19 BPEL 8.03 x 5.6 10/01/19 BPEL 8.19 x 5.6

Originally Posted by da_dt
It seems like the 40° threshold cited above would be achievable without US. I’ve spent time in both hot tubs and saunas hotter than that. What are your thoughts?

Sauna probably won´t do it , according to skin temperature measurements :

Hot tub , I bet, won´t either. The water should be so hot you can´t tolerate it. Dipping your dick only would be on the verge of having skin burns in any minute.


START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)

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