I have already mentioned elsewhere that phenomenological modelling is the description for my contributions theory wise.
To establish direct relations between the external stimuli via stretching and the observed tissue response without trying to explain the mechanisms behind the observations.
But Tutt and Manko have somehow got me involved and I am kind of forced to open thinking behind the things I have chosen do with my dick and why.
Here is my take on studies referred in the book: Science of flexibility by Michael Alter. I had previously captured screenshots from this and took a while to remember the source again but here it is. There are few things which I rely for being crucial for the growth I have achieved with my concept.
Heat is the key, so many times mentioned here at Thunders, but I think it is not clearly understood how high temperatures are actually needed.
When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous when it is heated. (Warrent et al (1971,1976)
The mechanism behind this thermal transition may be destabilization inter-molecular bonding or enhanced viscous flow properties of the collagenous tissues, possibly allowing elongation to occur with less structural damage. (Rigby 1964)
Raising the temperature of tendon above 103 F(39,7 C) increases the amount of permanent elongation that results from the initial stretching. (Laban 1962, Lehmann et al . 1970)
Above 104F (40 C) occurs thermal transition of the microstructure of the collagen, significantly enhancing the viscous stress-relaxation of collagenous tissue and allowing greater plastic deformation during stretch. (Mason and Rigby 1964, Rigby et al 1959)
According to Sapega et al (1981), stretching connective tissue at elevated temperature the condition which under the tissue cools down affects the elongation that remains after tensile stress is removed.
They based their hypothesis on Lehmann et al. (1970) , which found that after heated tissue is stretched , maintaining tensile force during cooling down significantly increasing the relative proportion of plastic deformation compared with unloading the tissue while its temperature still high.
They also speculated that cooling the tissue before releasing allows the collagenous micro-structure to re-stabilize more toward its new stretched length.
However (Hardy and Woodlal 1998) have questioned benefits the cooling while tension. Applying cold while tension it diminished all the gains made in flexibility with every study group.
Therefor applying ice during cool down is not necessary.
IMO possibly even counterproductive. Perhaps, it is better to let the tissues cool down naturally , applying the tension via manual stretching or at extender with fixed length at maximal elongation achieved during heated exercises.
In my case all above mentioned should mean that operating at 41- 43 C the tissues are able to go through plastic deformation at some degree at strains well below proportional limit, not to mention plastic region which will remain untouchable in every circumstances while enlarging our penises.
Being able to elongate BPFSL at every cycle without reaching proportional limit at the stress strain curve.
Baseline BPFSL transitioning 1 mm at time forward and starting every cycle at new fresh starting point longer than before.
Stress strain curve staying the same and tangent modulus being intact, baseline just transitioning forward due elongation
I am hugely convinced that I am not breaking anything, no micro tears , no inflammation. Even if there is minor it is not the case of the growth here. This is something else.
Heat being the topic I would like to add few advantages with the temperatures I am using.
These were posted previously in here by guy named MX and are all valid:
“Increased Metabolic Rate - Metabolic effects - Enhances healing
Increased enzymatic activity has been seen in tissues at 39-43C (102-109F)
Enzyme activity rates begin decreasing beyond 43C (here was 45 but I corrected it) and cease completely at 50C (122F).
Any increase in enzyme rate will increase the cellular biochemical processes and the uptake of oxygen resulting in accelerated healing
Increase of tissue temperature shifts the oxygen-hemoglobin dissociation curve to the right which releases more oxygen for tissue repair.
Hemoglobin releases twice as much O2 when tissue temperature is 41C. (106F) than when it is at resting temperature. “
The last paragraphs comes very important to knowledge when starting to clamp with US.
Did some exercises too. Not much to mention except day 2 I skipped the manual stretching and went for the extender cool down.
Other wise basic routines. Keeping head down and staring my dick.
DAY 1 Measured pre BPFSL 23.6cm and post BPFSL 24.1cm. Strain of 2.6%. (avg)
DAY 2 Measured nothing.
DAY 3 Measured pre BPFSL 23.7cm and post BPFSL 24.1cm. Strain of 2.4%. (avg)