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Question to you guys. Is the skin on the penis heating at the same rate than skin on other parts of the body. IE: if I test my IR wrist wrap solution on my arm and don’t get burned, does it mean that my tool would not get burned as well?


PhoenixNow Progress Reports

Started August 2018: BPEL/MEG 5.7" x 5" - Now: 6.1" x 5.25" BEG: 6.25"

Goal: BPEL/MEG 8.5" x 6.5"

Originally Posted by PhoenixNow
Question to you guys. Is the skin on the penis heating at the same rate than skin on other parts of the body. IE: if I test my IR wrist wrap solution on my arm and don’t get burned, does it mean that my tool would not get burned as well?

I would assume the skin of the phallic apparatus is more sensitive to temperature since its so thin. I would test it myself by rubbing a metal ring against fabric very fast until it gets hot, then quickly put it on the skin of your wrist to asses sensation. Then repeat the same this time putting it on your dick and see the difference. I believe the heating rate of the skin is the same, the question is if it feels more or not and that will be determined by the amount of nervous terminals.

Originally Posted by PhoenixNow
Question to you guys. Is the skin on the penis heating at the same rate than skin on other parts of the body. IE: if I test my IR wrist wrap solution on my arm and don’t get burned, does it mean that my tool would not get burned as well?

It’s not about the rate of heating of the epidermis itself which might be similar to the skin on the underside of your arm. There are other considerations.

While the glans is quite sensitive and vulnerable, the skin on your shaft and scrotum are actually not very sensitive at all. Go ahead. Reach down and pinch the skin nice and hard. Now do the same thing to the inside of your thigh just next to your scrotum. Consequently, the inside of your thigh is going to feel like it burns more easily than your scrotum. We are always most sensitive in life threatening areas, like those harboring vital arteries. Like your body is telling you to protect those areas.

My concerns in heating the penis are that because of the lower sensitivity, you are more likely to injure the skin on the penis or the underlying tissues simply because you won’t notice it happening as easily or as quickly.

Originally Posted by Tutt

Finally, even though the studies showed that if you heat the tissue up and go slow enough you can literally stretch the dead tissue like 20-30% beyond the normal limit, this isn’t even close to realistic for living tissue. Even if you go slow and hot enough to get through the typical elastic limit at around 2.5-4% beyond the previous BPFSL, you will have to start pulling REALLY hard and things will start to get REALLY painful because the nerves don’t adapt that quickly. So really, the physiological limits are going to be about 3-7mm of excess stretch before you give the body a chance to at least partially heal.

I have been reading with the thought these posts delivered lately and to this point not having the need for commenting anything.

But the nerve thing jumped in my eyes. Nerves can easily stretch up to 20-25% without any difficulties with slow stretch rates.
I suppose in our case it should the BPEL, for the dorsal nerves used as a baseline length before the nerve elongation.

I have not speculated this publicly earlier as I have not been experienced it since.

I have once felt my dorsal nerves aching from the tip of the glans deep down inside my body.
This was during the the end of P3, using ultrasound, temperature being at the peak already and the penis stretched out at the all time maximum of 24.1cm.
The sensation was like the dorsal nerves were resonating like a guitar string all the way through its length.
Removing the transducer the aching feeling subsided and though being in the excessive strain no pain was felt otherwise.
This was the time i thought that the dorsal nerves might be starting to limit the further elongation.

As we know the P4 did not bring further gains on BPFSL, yet it remains speculative if this has nothing to do with the plateau.
After all I did not feel any of these moments during the P4 though.

During the period 3 I had increased the BPFSL from 22.8 cm to my all time maximum of 24.1 pre-workout BPFSL in 10 weeks.

BPEL at the same time from 21.4cm to 22.2 cm. Calculating from the 21.4 cm BPEL the nerve strain was 12,5% at the time.
In total I had increased the BPFSL 34% in 14 months time frame.

In leg lengthening with the mechanical device they elongate nerves as well. What they have found is that if the limb lengthening strain rate exceeds 1mm/day, the nerve pain and nerve damages are significantly more likely to happen.


START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)

Originally Posted by Tutt

Having now looked at the data, I’m a bit disappointed that I couldn’t go further because the latter section of the curve had begun to linear and I now don’t know if that was real or just noise. In any case, at first glance the curve appears to be a typical exponential curve, but upon closer inspection and curve fitting, it is actually linear until almost exactly 2kg load. There is a noticeable point there where a log scale function transitions to a linear slope, indicating that this is the beginning of the transition from a linear curve to an exponential. The log scale function remains linear until just under 5kg load and then seems to indicate another transition into proportional strain. Because of the device malfunction, I don’t think that I can trust this, so until I can repeat with stronger wire, I will continue to assume that the load/strain relationship would’ve remained exponential. Given that there was no indication of device malfunction preceding the catastrophic failure, and the last 5 measurements indicated proportionality, I will remain suspicious until I can perform the test again. But it will have to wait until next Wednesday.

The suspiciousness is justifiable.This is the most crucial area of this research, the transition from the stiffening transitional slope to the proportional region.
Tutts main interest is to examine how much the different strain increase rates have influence on the subsiding stiffening.
The more there is additional strain available with as low as possible loading the more permanent elongation there should be gained.

I have already commented this privately and like Tutt said it remains to be un-confirmed that should the stiffening subside at that point despite the few datapoints showing initial proportionality.
There are few things to concern with this. Tutt has been able to smoothen the transitional region slope significantly because the vast number of datapoints due the precise load introducing and it is showing not so steep as it is with more harsh load increments. The curve might be extending exponentially and more test should be produced.

The setup may have some origins for the smoothening as well, the base ring with any extender user sinks relatively slowly in the dermal, adipose and muscular tissue before adjusting firmly against pubic bone.
Longer data logging cycles are needed using the extender setup for stabilized environment for the load - strain curves being 100% reliable.

As I mentioned earlier the equipment is dealed and further develoed separately and the main focus is in the behaviour of the tissue in this log at the moment.


START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)

Originally Posted by Tutt
Kyrpa and I have discussed further. I am becoming much more focused on strain rate and time under tension. As a quick summary, I tested a cyclic strain protocol today that was described by me the other day. It resulted in valuable insight but as a PE protocol was not successful. Nutshell… it produced less length at higher loads than the first 2 workouts. Notably, it wasn’t even successful in reaching equivalent BPFSL experienced on monday. In short, the original protocol was better; longer BPFSL at lower loads.

Strain percentage is the determiner. If the application, more precisely stress-relaxation application is directly driven by strain the better it gets.
Without the extender setup there is no such possiblity, the events need to be controlled via loading phases.


START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)

Originally Posted by Tutt
Yes…

The precise point I was trying to make is that my interim tests had abandoned a low strain rate in favor of limited duration and static load. Highly counterproductive as the fast strain rate resulted in lesser elongation at significantly higher loads. Such is the well established nature of viscoelastic tissue. The faster you pull, the harder you must pull. Ironically, in living tissue, the harder you pull the harder you must pull as well. Pull really hard and the ECM response will be to ensure that your tissue can handle that stress next time. Keep pulling harder and there is no question that the ECM response will render you unable to possibly pull hard enough without injury.

Every approach with sudden significant peaks in the load such as been used in some High pressure vacuum pumping protocols for instance, are counter-productive and wreck the potential of the exercise.
Any of the ballistic stretching manoeuvres are just plain insanity.


START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)


Last edited by Kyrpa : 06-06-2020 at .

Originally Posted by igigi
Second, it has some mystic-mythological origins. I remember back in the day when I first started researching about it in the late 90’s, it was advertised as an ancient African-middle eastern tradition to grow the penis.

Despite the tales of old time great gainers live in legendary proportions, we need to step out from the comfortable camp fire and put the light on.


START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)

Originally Posted by Tutt
There was some question about where on the load-strain curve we are referencing, but the modality of how to elongate the tissue is not in question.

Emphasis on the was. There should not be any controversies left with data enough.


START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)

Originally Posted by Kyrpa
Strain percentage is the determiner. If the application, more precisely stress-relaxation application is directly driven by strain the better it gets.
Without the extender setup there is no such possiblity, the events need to be controlled via loading phases.

Agreed. It’s quite apparent that the slow strain rate, strain duration, and relatively small incremental strains are predicated on use of an extender. Manual stretch protocols will not suffice.

Originally Posted by Kyrpa
Emphasis on the was. There should not be any controversies left with data enough.

Yes… that is a WAS.

Interestingly, digging through some nerve elongation studies, they discuss the stress-strain curve of a nerve which as we know is much the same as a tendon as the nerve is wrapped in layers of Type 1, 2, and 4 collagen. They casually reference a distinct difference between the elastic zone and the plastic zone that was quite a large portion of the apparent linear proportional zone well before the yield point and flow region.

Unlike structural tendons, nerves will commonly progress into the plastic region through normal range of motion.

Originally Posted by Kyrpa
The suspiciousness is justifiable.This is the most crucial area of this research, the transition from the stiffening transitional slope to the proportional region.
Tutts main interest is to examine how much the different strain increase rates have influence on the subsiding stiffening.
The more there is additional strain available with as low as possible loading the more permanent elongation there should be gained.

I have already commented this privately and like Tutt said it remains to be un-confirmed that should the stiffening subside at that point despite the few datapoints showing initial proportionality.
There are few things to concern with this. Tutt has been able to smoothen the transitional region slope significantly because the vast number of datapoints due the precise load introducing and it is showing not so steep as it is with more harsh load increments. The curve might be extending exponentially and more test should be produced.

The setup may have some origins for the smoothening as well, the base ring with any extender user sinks relatively slowly in the dermal, adipose and muscular tissue before adjusting firmly against pubic bone.
Longer data logging cycles are needed using the extender setup for stabilized environment for the load - strain curves being 100% reliable.

As I mentioned earlier the equipment is dealed and further develoed separately and the main focus is in the behaviour of the tissue in this log at the moment.

Yes, in the next test, I’ll need to progress more slowly with a stronger wire over a far longer duration. I’ll be performing this test once a week at different strain rates.

Also, the translation stage is independent of the fixed extender, so it is easy for me to place significant pressure against the pubic bone without significantly affecting the strain measurement. I propose to take a period of 5 minutes prior to beginning the test in which I apply roughly 6-8kg of pressure against the ring to preemptively collapse the soft tissue structure covering the bone. It isn’t very thick to begin with as I am a fairly fit individual.

Originally Posted by Kyrpa
I have been reading with the thought these posts delivered lately and to this point not having the need for commenting anything.

But the nerve thing jumped in my eyes. Nerves can easily stretch up to 20-25% without any difficulties with slow stretch rates.
I suppose in our case it should the BPEL, for the dorsal nerves used as a baseline length before the nerve elongation.

I have not speculated this publicly earlier as I have not been experienced it since.

I have once felt my dorsal nerves aching from the tip of the glans deep down inside my body.
This was during the the end of P3, using ultrasound, temperature being at the peak already and the penis stretched out at the all time maximum of 24.1cm.
The sensation was like the dorsal nerves were resonating like a guitar string all the way through its length.
Removing the transducer the aching feeling subsided and though being in the excessive strain no pain was felt otherwise.
This was the time i thought that the dorsal nerves might be starting to limit the further elongation.

As we know the P4 did not bring further gains on BPFSL, yet it remains speculative if this has nothing to do with the plateau.
After all I did not feel any of these moments during the P4 though.

During the period 3 I had increased the BPFSL from 22.8 cm to my all time maximum of 24.1 pre-workout BPFSL in 10 weeks.

BPEL at the same time from 21.4cm to 22.2 cm. Calculating from the 21.4 cm BPEL the nerve strain was 12,5% at the time.
In total I had increased the BPFSL 34% in 14 months time frame.

In leg lengthening with the mechanical device they elongate nerves as well. What they have found is that if the limb lengthening strain rate exceeds 1mm/day, the nerve pain and nerve damages are significantly more likely to happen.

I have experienced the beginnings of this sensation as well. I propose that it isn’t entirely a function of strain stress, but rather the resulting compressive stress that the strain is causing. We know that as nerves are strained they experience a dramatic increase in transverse fluid compression. This might be producing a pain sensation that is aggravated by high levels of heat and lack of blood flow to the nerve. We also know that even though axons have incredible ability to elongate, with as little as 6% strain, they begin to experience reduced blood flow and conductivity which increases dramatically as time under strain increases.

Originally Posted by Kyrpa
Emphasis on the was. There should not be any controversies left with data enough.

Hey Kyrpa, kindly look at your private messages :)

I am one week complete in my US routine.

I have a Sizegenetics with vacuum attachment, the delta Ultrasound 3mhz (same as Longstretch).

Originally Posted by DantheMan2020
Hey Kyrpa, kindly look at your private messages :)

I am one week complete in my US routine.

I have a Sizegenetics with vacuum attachment, the delta Ultrasound 3mhz (same as Longstretch).

Hi,

I gave you something to think.


START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"

When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)

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