A few more thoughts:
Lil12big1, you mention the folks in “white coats”…well, they have spoken on this GvM model regarding PE (perhaps you haven’t heard) – they unanimously REJECT it.
So many of the members here are guilty of a common informal fallacy, a version of the “Faulty Dilemma.” You hear the medical community say such things as, “You can’t enlarge the penis by doing exercises…the penis is not a muscle…”etc., etc. Yet, we do PE and make gains; therefore, we conclude that the entire medical establishment is wrong – or lying (so that surgeons can make money on phalloplasty).
What these intrepid souls never seem to do is to consider that perhaps there’s a different mechanism involved with our (obvious) enlargement. A mechanism that is NOT a classic “building” model, a GvM model. That’s what EtP is – and it relies on far fewer assumptions than the other models posited here.
As for the so-called “science” supporting a GvM model (regarding PE), please. That’s bullshit. No such research exists. If you think you have it, then forward it to the NEMJ – or some other academic journal (you’ll be famous!).
* In hypertrophy, actual mass it increased; in PE, only area (albeit, during erection more blood is pooled in the penis).
* For an idea of PE “growth,” consider a balloon – not a biceps.
* In PE, enlargement occurs not from an aggregation, but from a stretching – and even a thinning – of structures (the reason that the penis can get “thicker” as the structures are thinning is that the walls of those structures are thinning, while the blood-holding spaces enlarge; and as the tunica thins, its area increases).
* As elasticity is used up, enlargement occurs [EtP]; as plasticity is replaced by (newer) elasticity, shrinkage occurs [PtE].
Full Circle
For me, I’ve recently experienced what I believe is the final “test” of my theory. Any in this forum who’ve read some of my earlier posts regarding my thoughts of being a profound “grower” knows of the despair I felt back in the school gym locker rooms of my youth.
Having been cursed with an F:E of more than 2:1, I seldom ever sported even a 3” flaccid. As I knew that I was 6 ½” EL, I assumed that the guys with 5-6” flaccid must’ve been around 11-12” EL; therefore, I had a “teeny weenie.”
Well into my PE, my flaccid went from < 50% of my erect to > 60% (near the endgame). That was, of course, no mere “proportional” growth, keeping in line with my erect gains. No, it was a disproportional increase – necessarily indicative of decreasing elasticity. Clearly so! Whereas my “natural” unit more than doubled in size (100+ %) with tumescence, it later expanded < 60% when becoming hard.
To put it simply, my PE’d penis ballooned less dramatically, “to boner,” than it did before; conversely, it shrank less when it became flaccid. Clearly a change in the elastic properties of my wang.
Recently, I took a depressing photo of my flaccid with a cheap, low-res camera. It’s jarring to me in that it dredges up my past dread. I’m seriously debating on whether or not to post it to illustrate my point.
What point?
I’m back to around a 2:1 FE….PtE.
That’s right, my flaccid losses are not proportional to my erect losses – they’re greater. Now, my weenie balloons up more during tumescence (i.e., “more elastic”).
That completes the cycle.