Focus Your PE and Gain! TGC Theory

Hey,

I found your TGC theory very interesting. I might be able to provide some more evidence on your theory with an injury I have suffered..

Long story short..

I suffered a kegel enhanced jelqing injury about 2 years ago. I was a newbie and started off by doing Kegel enhanced jelqs. I had a lot of success and gained a lot in a short period of time. I took a couple of months off and then got back into Jelqing with the same routine that I left off on. Within three days of doing this routine I woke up to a dead penis. My penis was completely dead.. I had no feeling, no control and very little blood flow. My penis completely turtled up on me and got cold. It felt like a saggy deflated balloon. There was no pain or signs of bruising however, which was weird. I have seen numerous Urologists/specialists and have had different tests done with no avail. They all say that my penis is normal and is functioning properly. They have ruled out venous leakage, tunica breakages and nerve damage. I was sent to see a pelvic floors specialist after my Urologist gave up. My pelvic floor specialist seems to think she knows whats going on. She has basically said I have stressed out all my pelvic floor muscles and as a result my PC/BC muscles are not working properly.. And so on.. She has seen this before and has fixed problems like this for people in the past. She’s been giving me a bunch of different exercises and stretches to help RELAX my muscles and try to get my muscles to work properly again. This seems to be working pretty well, I have all the feeling back in my penis and my erections are getting a little bit better. I still have “squishy” erections and my flaccid penis feels like it has no body to it.

I have lost .5 an inch in length and .25 an inch of girth since my injury. It looks and feels like my penis is being pulled in side of my body. After reading your theory it raised a lot of questions.

Is it possible my smooth muscle could have atrophied over the past two years with no PE to the point where it can’t fill up my tunica?
- This could be an interesting discovery and could help me get my penis back

What exercises would you recommend me start doing if I wanted to get back into PEing? I would like to get back into a routine and see if it helps my penis health at all.

Please shed some light on this subject.

Thanks a lot

Hey, I was hoping maybe someone (Iguana?) could help me out here. My stats are as follows:

BPFSL: 7.6”
BPEL: 7.6”
EG: 5.4”
EQ: 9

Basically, the whole time I’ve done PE, I have only done girth orientated exercises (things like jelqing, clamping and O-bends a while back), yet have had length gains of 1.6”, and girth gains of 0.5”(from when I was at 5.5” girth, I have had a 3 month break and lost 0.1”). I only want to gain in girth right now, NOT length, and would hope to get it up to around 5.6”.

So I was thinking, obviously my tunica is limiting me here, but if I were to focus on stretching, would that not surely give me length gains in addition to girth? Perhaps I could try do some intensive stretching, immediately followed by some manual clamping?

I just really don’t want anymore length, I’m already at 7.2” NBPEL and would fear not being able to go balls deep with some girls if I were any longer.

Thanks in advance:) .

You might want to try some compression squeezes along with clamping. You want to focus on exercises that stress the tunica longitudinally.

Unfortunately, there is not that many. I think some stretching couldn’t hurt. If you start noticing length gains (which will undoubtedly be small at first) you can back off.

Thanks, I’ll give it a go!

Okay so why does my penus tighten when I do kegels shouldn’t my smooth muscles stop relaxing there by keeping my penus soft enough to train? And what happens to the tunica when I kegel? How can I make sure it’s soft, when I want 30% (erect) volume to be able to strecth it and jelq without getting erect quickly?

What exercises I have to do .. My EQ is 10/10 my bpfl is 1inch more than my bpel :?

1. Any more ideas on girth exercises that hit the tunica longitudinally? I am 7.25x4.5 with rock hard erections, but I would rather not do these length smooth muscle exercises.

2. I think for most of the routines from which I’ve gained, the exercises alternated between various stretches and jelqs. Do you think this could possibly be tied into the TGC theory? Say, weakening the “bicycle tire” first allows for better expansion of the “inner tube,” and vice versa.

I guess that meds like Viagra must cause an immediate increase in the smooth muscle of the penis - within 20 minutes or so.

Actually, this article states that it is the relaxing of the smooth muscle tissue within the CC sinuses that causes erection…

Erectile Function and Nitric Oxide. The penis is either flaccid or erect depending on the state of arousal. In the flaccid, or unerect, penis, the following normally occurs:

Small arteries leading to the cavernous sinuses contract, reducing the inflow of blood.
The smooth muscles regulating the many tiny blood vessels also stay contracted, limiting the amount of blood that can collect in the penis.
During arousal the following occurs:

The man’s central nervous system stimulates the release of a number of chemicals, including nitric oxide, which is now considered the main contributor for eliciting and maintaining erection.
Nitric oxide stimulates production of cyclic GMP, a chemical that relaxes the smooth muscles in the penis. This allows blood to flow into the tiny pool-like cavernous sinuses, flooding the penis.
This increased blood flow nearly doubles the diameter of the spongy chambers.
The veins surrounding the chambers are squeezed almost completely shut by this pressure.
The veins are unable to drain blood out of the penis and so the penis becomes rigid and erect.
After ejaculation or arousal, cyclic GMP is broken down by an enzyme called phosphodiesterase-5 (PDE5), and other compounds are released that cause the penis to become flaccid (unerect) again.

Well - The New York Times

A few inherent problems with any theory that “smooth muscle enlargement” might explain PE enlargement.

(1) All muscles of the body contract - biceps, pecs, triceps, etc….even the heart.

(2) Hypertrophy occurs when a contracting muscle is restricted/inhibited by resistance - i.e., “progressive resistance training.” This “strain” or “load” sets of a biological chain reaction in the body that, eventually, leads to hypertrophy.

(3) Muscle contraction is necessary for muscle growth; however, “The contraction of smooth muscle is generally not under voluntary control.”¹

(4) When we do PE, the penis “does” nothing; it does not “act,” it is acted upon. Therefore, the requisite “work” that triggers training-induced growth never occurs during PE.

(5) If hot wraps & jelqing can make our penis “grow,” why don’t bodybuilders wrap their biceps with towels soaked in hot water then “jelq” their upper arms?

The notion that jelqing & stretching causes penile tissues to “grow” is unsustainable. Furthermore, if jelqing did result in “growth,” why wasn’t continued jelqing necessary to maintain full growth for a long time after cessastion of PE (in my case, 3+ years)? Try that with gym training.

I have not doubt that the true paradigm is not that of a muscle growing, but that of a thick-walled balloon. PE “thins” those walls - both that of the tunica (thus increasing surface area), as well as those of the tribeculae (thus increasing volume).

Furthermore, the best recent, medical explanation of undoubtedly massive enlargement of the penis (referenced in my PDF) explains one man’s megalophalls (7 1/2” flaccid girth) as resulting from “…a sudden loss of elasticity…” of his tunica.

In other words, he achieved ridiculous girth enlargement - and instantly. Do you believe he suddenly grew billions of new cells in his penis? No, of course not.

The trauma of repeated & severe priapism produced sudden & extreme deformation of his tunica, depleting it of elasticity. The tunica became thinner - not thicker - as the surface area expanded accutely. This, of course, resulted in a much larger inner volume of the penis…taking a once normal penis to 7.5” flaccid girth.

¹ -

Regarding this theory would it be feasible that someone with less tunica could expect faster gains over the short term and less long term potential? While someone with more layers of tunica could expect the opposite?

Is this thread dead? Does anyone have any more girth exercises that hit the tunica longitudinally? As posted above…

I have only just read the opening posts of the thread.

How do you (remek or iguana) explain growth in EG from girth work, given the tire metaphor? Is the tunica expanding? I am not discussing harder erections, but actual significant EG growth.

Also, how does pumping play into this? Do you have any opinion, based on your theory?

WANTED: A MEDICAL SCIENCE OF PE

RESPONSE TO REMEK

“Like nearly everything else in PE, all the information in this post is no more than a theory.”

In my view this is the basic shortcoming of PE as a human activity.

Lay people – at best, individuals with backgrounds in another science – piece together information on an area of the biological sciences. These people are often intelligent, and they are certainly motivated. But there is little sense that the lack of research-driven, peer-reviewed scientific inquiry limits progress – in particular, understanding of what will work best, and what is safe.

The common view of PE that uses weight training and sports medicine as a model – stress and cell regeneration – is inadequate: Does a single member of this forum know how micro-ruptures of the tunica affect the collagen-elastin balance? (This information is, perhaps, discoverable, but, to my knowledge, it has never been investigated.) How many have thought of this question as relevant to what they do? How many grasp it’s importance, what it means if tunica ‘regeneration’ favors increased collagen?

Yes, of course, as remek says, the ‘practice’ of PE practitioners can be the science lab that tests theories such as his. But there are limits and hazards to anecdotalism, only one of which is that it does not rigorously evaluate long term results: I recently met with a cardiologist to discuss my cholesterol levels following a period of using the cholesterol-lowering drug Lipitor. We discussed raising the dosage, and he said: I don’t want to do that. We don’t know the long term effect of very low LDL (bad cholesterol) levels, and I don’t want you to be the guinea pig.

I am quite sure that we are all heavily aware of this. What is your point?