Yes. We agree completely on every aspect concerning the tissue stressing methods.Stress relaxation allows greater initial stress used, as you say it starts to subside in huge portion soon after loaded.
I also have 100% consensus with you about the timeline tissue starting to get stronger as response to the repeated exercise bouts.
What I have thought and more so after you talking about this strain rate relations, is that the permanent elongation needs fibril sliding level change.
After all it is a crucial thing on natural growth bursts on teenagers.
It has been demonstrated happening due decorin biglycan bond release and re-organisation.Same thing happening with the therapeutic heat to some extend.
In fact there is a study indicating total decorin denaturation at 45C, allowing fibril sliding at great extend. Once the temperature drops the re-organization starts and it quickly is normalized at resting temperature.
This will been seen as instant permanent elongation.
That is the exact wild card to my postulates. You’ve hit the nail right on the head. Is fibril sliding THE driver of permanent elongation? Is it just one of many or is it perhaps the primary driver of a multivariate function? While it is very possible that most or all permanence comes from sliding, I’m very skeptical. This would imply that the best method would be to tug hard and fast on the penis, which would provide the greatest amount of sliding. Guys have been doing that on these forums for decades with poor results. The baseline assumption, supported by literature, is that the body has a rapid mechanism to prevent further sliding. This manifests as more cross linking and increased fibril density.
I think that sufficiently high heat makes sliding achievable at lower stress. But I think that it is more optimal to achieve permanence through deformation of the fibrils themselves and widening of gaps.