Originally Posted by Kyrpa
With ultrasound protocol we are we are concentrated completely in elongating BPFSL.
Which by the way is the primary target in all length training being it hanging or extending etc.
The BPEL comes as a seamless secondary co-product of this elongation.The difference being that with these novel methods we are able to achieve relatively significant increases in BPFSL, the BPEL being difficult to grow at the same rate.
BPFSL gains come more as a direct consequence of connective tissue elongation by the heat amplified stretching. BPEL growth is volumetric growth to fill this void as a simplification.
For inducing this growth almost every viable technique which involves expansion and stretching the cavernous tissue serves fine.
I have found BPEL growth with jelqs, any manual expansion exercise I have came up with, low erection bends, pumping, clamping , you name it.
There is a downside as well on adding these exercise in conjunction. As I have found out and stated it many times in my log, having excessive work load in conjunction with the BPFSL protocol the exhaustion is easily felt and EQ may drop significantly.
That’s why I dropped the jelqs and rest of the similar exercises aside at some stage.
And concentrated on BPFSL as long as it kept coming and then switched in to BPEL work for the rest of the period. It has worked just perfectly every time.
For what I have been really surprised that even that I dropped all additional workouts I still have managed to gain BPEL parallel to BPFSL.
Now seeing many already starting to master the BPFSL gaining, they may lack the BPEL gains.
This has made me think about it a lot and I can´t find any reasonable explanation other that that I have chosen to use the cyclical stretching as a cooldown method instead of static stretch /extension. I can´t prove it unless there more users going the same rout, but I do have very strong suspicion that being the crucial difference.
The cavernous tissue and tunica layers getting enough stimuli for inducing growth responses is the possible explanation.
As we can see the static method is working fine as it is also strongly backed with the literature.
Another thing is that is the protocol insufficient to trigger BPEL growth responses without additional exercise used along?