Using the ultrasound for therapeutic heat in PE
THE IMPORTANCE OF THE HEAT
Before stepping into the ultrasound therapy induced tissue heating we need to know the fundamentals of using therapeutical heat, heating tissue in general or just warming up.
Using constant heat has been an important catalyst for the gains in PE.
Anecdotal findings from the PE community show that the continuous heating has been providing gains for many, being the PE method which ever they have happened to chosen.
Studies indicate that raising collagenous tissue temperature by 3°C (moderate heating) from the baseline induces increased elasticity in the stretched tissues.
Going further up to 4- 8 °C (vigorous heating) above the resting temperature the magic starts to happen when the therapeutic heat starts to allow plastic deformation of collagen by thermal mechanisms.
After passing the threshold of 40°C the thermal transition is allowing permanent lengthening on tissues while stretched. Some percentage of the elongation reached during the heat never reverse back to the previous length.
Continuing to do this after few exercises we are getting longer penises. There is the magic.
Here are some excerpts and further interpretations from studies originally referred in the book: Science of flexibility by Michael Alter. It is the collection of highlights we should understand using any method of PE.
When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous when it is heated. (Warrent et al (1971,1976)
The mechanism behind this thermal transition may be destabilization inter-molecular bonding or enhanced viscous flow properties of the collagenous tissues, possibly allowing elongation to occur with less structural damage. (Rigby 1964)
Raising the temperature of tendon above 103 F(39,7 C) increases the amount of permanent elongation that results from the initial stretching. (Laban 1962, Lehmann et al . 1970)
Above 104F (40 C) occurs thermal transition of the microstructure of the collagen, significantly enhancing the viscous stress-relaxation of collagenous tissue and allowing greater plastic deformation during stretch. (Mason and Rigby 1964, Rigby et al 1959)
According to Sapega et al (1981), stretching connective tissue at elevated temperature the condition which under the tissue cools down affects the elongation that remains after tensile stress is removed. They based their hypothesis on Lehmann et al. (1970) , which found that after heated tissue is stretched , maintaining tensile force during cooling down significantly increasing the relative proportion of plastic deformation compared with unloading the tissue while its temperature still high.
They also speculated that cooling the tissue before releasing allows the collagenous micro-structure to re-stabilize more toward its new stretched length.
However (Hardy and Woodal 1998) have questioned benefits the aided cooling while tension. Applying cold while tension it diminished all the gains made in flexibility with every study group.
Therefor applying ice during cool down is not necessary and possibly even counterproductive.
With the above citations I am trying to say that operating at 40- 43 C , the tissues are able to go through plastic deformation by some degree at strains well below sub-failure proportions otherwise needed to cause plastic deformation at the same rate. Plastic deformation meaning permanent gains.
This permanent elongation is produced with lesser degree of structural or cellular damages with a lower stresses (load, time under load) needed with stretching the tissues below therapeutic heat temperatures.
Therapeutic heat is producing as a side product increased metabolic rate, enzymatic effects and enhanced healing rate. Increased enzymatic activity has been seen in tissues at 39-43C (102-109F).
Enzyme activity rates begin decreasing beyond 45C and to cease completely at 50C (122F).
Any increase in enzyme rate will increase the cellular biochemical processes and the uptake of oxygen resulting in accelerated healing.
Increase of tissue temperature shifts the oxygen-hemoglobin dissociation curve to the further which means releasing more oxygen for tissue repair.
Hemoglobin releases twice as much O2 when tissue temperature is 41C (106F), than at resting temperature.
START 18/13.15 cm Jul 24th 18 (7.09/5.18") NOW 22.5/15.2 cm Fer 12th 20 (8.86/5.98") GOAL 8.5"/ 6"
When connective tissue is stretched within therapeutic temperatures ranging 102 to 110 F (38.9- 43.3 C), the amount of structural weakening produced by a given amount of tissue elongation varies inversely with the temperature. This is apparently related to the progressive increase in the viscous flow properties of the collagenous tissue when it is heated. (Warren et al (1971,1976)